Search Results - "Setiadi, Audi"

A sinister case of pseudothrombocytosis by Loyzer, Melissa N., Setiadi, Audi

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Monolobated megakaryocytes in Diamond–Blackfan anemia with RPL5 mutation at disease presentation mimicking myelodysplastic syndrome by Setiadi, Audi, Singh, Claudia, Li, Amanda, Au, Nicholas, Amid, Ali

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COVID-19, haemophagocytic lymphohistiocytosis, and infection-induced cytokine storm syndromes by Spaner, Caroline, Goubran, Mariam, Setiadi, Audi, Chen, Luke Y C

“…Recent research in COVID-19 cytokine storm syndrome and Castleman disease has expanded the concept of pathological immune activation and established important…”
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Circulating rhabdoid tumor cells in the peripheral blood of a neonate by Al‐Bakri, Maryam, Terry, Jefferson, Chipperfield, Kate, Morrison, Douglas, Setiadi, Audi

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Machine learning optimized multiparameter radar plots for B‐cell acute lymphoblastic leukemia minimal residual disease analysis by Shopsowitz, Kevin E., Liu, Lorraine, Setiadi, Audi, Al‐Bakri, Maryam, Vercauteren, Suzanne

“…Background Flow cytometry is widely used for B‐ALL minimal residual disease (MRD) analysis given its speed, availability, and sensitivity; however,…”
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Targeted treatment of immune thrombocytopenia in CTLA‐4 insufficiency: a case report by Lai, Chieh Min Benjamin, Setiadi, Audi, Barlas, Angeliki, Kanani, Amin, Pourshahnazari, Persia, Leitch, Heather A., Metzger, Daniel L., Merkeley, Hayley, Biggs, Catherine M.

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CRP and sCD25 help distinguish between adult‐onset Still's disease and HLH by Beckett, Madelaine, Spaner, Caroline, Goubran, Mariam, Wade, John, Avina‐Zubieta, Juan Antonio, Setiadi, Audi, Tucker, Lori, Shojania, Kam, Au, Sheila, Mattman, Andre, Lee, Agnes Y. Y., Fajgenbaum, David C., Chen, Luke Y. C.

“…Objective Adult‐onset Still's disease (AOSD) and secondary hemophagocytic lymphohistiocytosis (sHLH) are both hyperferritinemic cytokine storm syndromes that…”
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Notch Initiates the Endothelial-to-Mesenchymal Transition in the Atrioventricular Canal through Autocrine Activation of Soluble Guanylyl Cyclase by Chang, Alex C.Y., Fu, YangXin, Garside, Victoria C., Niessen, Kyle, Chang, Linda, Fuller, Megan, Setiadi, Audi, Smrz, Justin, Kyle, Alastair, Minchinton, Andrew, Marra, Marco, Hoodless, Pamela A., Karsan, Aly

“…The heart is the most common site of congenital defects, and valvuloseptal defects are the most common of the cardiac anomalies seen in the newborn. The…”
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Expanding the molecular and phenotypic spectrum of CTLA‐4 insufficiency by Duke, Sean, Maiarana, James, Yousefi, Pariya, Burks, Elijah, Gerrie, Samantha, Setiadi, Audi, Amid, Ali, Boerkel, Cornelius, Erdle, Stephanie, Guttman, Orlee, Kanani, Amin, Lawrence, Sally, Lee, Anna F., Pourshahnazari, Persia, Rayar, Meera, Rozmus, Jacob, Schrader, Dewi, Sharma, Mehul, Shopsowitz, Kevin E., Tan, Ryan, To, Fergus, Yang, Connie, Hildebrand, Kyla J., James, Elliot, Turvey, Stuart E., Markle, Janet G., Biggs, Catherine M.

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Clinical and laboratory features associated with myeloperoxidase expression in pediatric B‐lymphoblastic leukemia by McGinnis, Eric, Yang, David, Au, Nicholas, Morrison, Douglas, Chipperfield, Kate M., Setiadi, Audi F., Liu, Lorraine, Tsang, Angela, Vercauteren, Suzanne M.

“…Background B‐lymphoblastic leukemia (B‐ALL) is the most common childhood malignancy, and its diagnosis requires immunophenotypically demonstrating blast B cell…”
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T‐lymphoblastic transformation of chronic myeloid leukemia by Al Bakri, Maryam, Setiadi, Audi

“…Given the proportion of cells positive for BCR/ABL1 fusion, myeloid predominance, and smaller fraction of T lymphoblasts, diagnosis of T-lymphoblastic…”
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PB2039: INFLAMMATORY MARKERS IN HEMOPHAGOCYTIC LYMPHOHISTIOCYTOSIS ‐ A SINGLE CENTRE STUDY by Goubran, Mariam, Spaner, Caroline, LI, Amanda, Zoref‐Lorenz, Adi, Jordan, Michael, Chen, Luke Y C, Setiadi, Audi

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CD138-negative plasma cell myeloma: a diagnostic challenge and a unique entity by Setiadi, Audi Francesca, Sheikine, Yuri

“…Plasma cell neoplasms may exhibit variations in morphology and immunophenotype, which can mimic mature B-cell lymphoproliferative disorders and pose diagnostic…”
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A Longitudinal Single-Cell Atlas of Treatment Response in Pediatric AML by Lambo, Sander, Trinh, Diane L., Ries, Rhonda E, Jin, Dan, Setiadi, Audi, Ng, Michelle, Leblanc, Veronique G., Vercauteren, Suzanne, Meshinchi, Soheil, Marra, Marco

“…Introduction: Pediatric acute myeloid leukemia (pAML) is a heterogeneous disease in terms of driver alterations, treatment response and patient outcomes…”
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Malignancy-associated haemophagocytic lymphohistiocytosis by Setiadi, Audi, Zoref-Lorenz, Adi, Lee, Christina Y, Jordan, Michael B, Chen, Luke Y C

“…Haemophagocytic lymphohistiocytosis (HLH) is an inflammatory syndrome that can occur with cancer (malignancy-associated HLH) or with immune-activating…”
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EDTA-induced platelet hypogranulation: beware of dysplasia mimic by Al Moosawi, Muntadhar, Vawda, Ayesha, Setiadi, Audi

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Inflammatory Markers in Hemophagocytic Lymphohistiocytosis - a Single Centre Study by Spaner, Caroline, Goubran, Mariam, Stukas, Sophie, Shojania, Kamran, Li, Amanda M, Sekhon, Mypinder, Wellington, Cheryl, Zoref-Lorenz, Adi, Mattman, Andre, Peterson, Erica A., Jordan, Michael B., Chen, Luke Y. C., Setiadi, Audi

“…BACKGROUND Hemophagocytic lymphohistiocytosis (HLH) has significant clinical and biochemical overlap with other cytokine storm syndromes including Adult-Onset…”
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10 Optimizing implementation and continuous monitoring of a newborn screening program for severe combined immunodeficiency by Ngan, Abigail Netanya, Hildebrand, Kyla, Turvey, Stuart, James, Elliot, Cook, Victoria, Cameron, Scott, Setiadi, Audi, Vallance, Hilary, Rakic, Bojana, Sinclair, Graham, Biggs, Catherine M

“…Abstract Background Severe Combined Immunodeficiency (SCID) is an inborn error of immunity characterized by severely low T cell levels and function. Early…”
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Differential Regulation of Transforming Growth Factor β Signaling Pathways by Notch in Human Endothelial Cells by Fu, YangXin, Chang, Alex, Chang, Linda, Niessen, Kyle, Eapen, Shawn, Setiadi, Audi, Karsan, Aly

“…Notch and transforming growth factor β (TGFβ) play critical roles in endothelial-to-mesenchymal transition (EndMT), a process that is essential for heart…”
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Human complete NFAT1 deficiency causes a triad of joint contractures, osteochondromas, and B-cell malignancy by Sharma, Mehul, Fu, Maggie P., Lu, Henry Y., Sharma, Ashish A., Modi, Bhavi P., Michalski, Christina, Lin, Susan, Dalmann, Joshua, Salman, Areesha, Del Bel, Kate L., Waqas, Meriam, Terry, Jefferson, Setiadi, Audi, Lavoie, Pascal M., Wasserman, Wyeth W., Mwenifumbo, Jill, Kobor, Michael S., Lee, Anna F., Kuchenbauer, Florian, Lehman, Anna, Cheng, Sylvia, Cooper, Anthony, Patel, Millan S., Turvey, Stuart E.

“…•Human biallelic damaging variants in NFATC2 increase susceptibility to B-cell lymphoma and musculoskeletal defects.•Studying primary human cell types lacking…”
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