Search Results - "Sensi, S.L"

Decreased numeric density of succinic dehydrogenase-positive mitochondria in CA1 pyramidal neurons of 3xTg-AD mice by Fattoretti, P, Balietti, M, Casoli, T, Giorgetti, B, Di Stefano, G, Bertoni-Freddari, C, Lattanzio, F, Sensi, S L

“…Alzheimer disease (AD) is associated with mitochondrial dysfunction. In this study, we investigated succinic dehydrogenase (SDH) activity in mitochondria of…”
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Calcium ionophores can induce either apoptosis or necrosis in cultured cortical neurons by Gwag, B.J., Canzoniero, L.M.T., Sensi, S.L., DeMaro, J.A., Koh, J.Y., Goldberg, M.P., Jacquin, M., Choi, D.W.

“…Cultured cortical neurons exposed for 24 h to low concentrations of the Ca 2+ ionophores, ionomycin (250 nM) or A-23187 (100 nM), underwent apoptosis,…”
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Measurement of intracellular free zinc in living neurons by Canzoniero, L M, Sensi, S L, Choi, D W

“…Excessive Zn2+ influx has been implicated in the pathogenesis of neuronal death after global ischemia or prolonged seizures, but little is presently known…”
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Characterization of MPP+-Induced Cell Death in a Dopaminergic Neuronal Cell Line: Role of Macromolecule Synthesis, Cytosolic Calcium, Caspase, and Bcl-2-Related Proteins by Choi, Won-Seok, Canzoniero, L.M.T., Sensi, S.L., O'Malley, Karen L., Gwag, Byung J., Sohn, Seonghyang, Kim, Ji-Eun, Oh, Tae H., Lee, Eunhee B., Oh, Young J.

“…To further characterize MPP+-induced cell death and to explore the role of Bcl-2-related proteins in this death paradigm, we utilized a mesencephalon-derived…”
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A case of Sars-Cov-2-related mania with prominent psychosis by Russo, M., Consoli, S., De Rosa, M.A., Calisi, D., Dono, F., Carrarini, C., Onofrj, M., De Angelis, M.V., Sensi, S.L.

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Alemtuzumab treatment of multiple sclerosis in real-world clinical practice: A report from a single Italian center by di Ioia, M., Di Stefano, V., Farina, D., Di Tommaso, V., Travaglini, D., Pietrolongo, E., Sensi, S.L., Onofrj, M., De Luca, G.

“…•Safety profile of alemtuzumab may differ in real-world populations compared to subject investigated in clinical trials.•The switch to alemtuzumab must take…”
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Measurement of Intracellular Free Zinc in Living Neurons by Canzoniero, L.M.T., Sensi, S.L., Choi, D.W.

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“Back to Braak”: Role of Nucleus Reuniens and Subcortical Pathways in Alzheimer’s Disease Progression by Censi, S., Sestieri, C., Punzi, M., Delli Pizzi, A., Ferretti, A., Gambi, F., Tomassini, V., Delli Pizzi, Stefano, Sensi, Stefano L.

“…Background Patients with Alzheimer’s Disease (AD) exhibit structural alterations of the thalamus that correlate with clinical symptoms. However, given the…”
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Glutamate receptor-mediated calcium entry in neurons derived from P19 embryonal carcinoma cells by Canzoniero, L.M.T., Sensi, S.L., Turetsky, D.M., Finley, M.F.A., Choi, D.W., Huettner, J.E.

“…We have examined the control of calcium elevation by glutamate in neurons derived from the mouse P19 embryonal carcinoma cell line. Following transient…”
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Measuring zinc in living cells.: A new generation of sensitive and selective fluorescent probes by Gee, K.R, Zhou, Z.-L, Ton-That, D, Sensi, S.L, Weiss, J.H

“…New fluorescent indicators with nanomolar to micromolar affinities for Zn 2+ have been synthesized in wavelengths from UV to the far red. The UV light-excited…”
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Measurement of Intracellular Free Zinc in Living Neurons: Volume4, Number 3-4 (1997), pages 275–279 by Canzoniero, L.M.T., Sensi, S.L., Choi, D.W.

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Measuring zinc in living cells by Gee, K.R, Zhou, Z.-L, Ton-That, D, Sensi, S.L, Weiss, J.H

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Nitric oxide reduces Ca2+ and Zn2+ influx through voltage-gated Ca2+ channels and reduces Zn2+ neurotoxicity by SNIDER, B. J, CHOI, J, TURETSKY, D. M, CANZONIERO, L. M. T, SENSI, S. L, SHELINE, C. T, WANG, X, YU, S. P, CHOI, D. W

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Nitric oxide reduces Ca 2+ and Zn 2+ influx through voltage-gated Ca 2+ channels and reduces Zn 2+ neurotoxicity by Snider, B.J, Choi, J, Turetsky, D.M, Canzoniero, L.M.T, Sensi, S.L, Sheline, C.T, Wang, X, Yu, S.P, Choi, D.W

“…The translocation of synaptic Zn 2+ from nerve terminals into selectively vulnerable neurons may contribute to the death of these neurons after global…”
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