Search Results - "Schulthess, F. T"

Purinergic P2X₇ receptors regulate secretion of interleukin-1 receptor antagonist and beta cell function and survival by Glas, R, Sauter, N. S, Schulthess, F. T, Shu, L, Oberholzer, J, Maedler, K

“…Aims/hypothesis In obesity, beta cells activate compensatory mechanisms to adapt to the higher insulin demand. Interleukin-1 receptor antagonist (IL-1Ra)…”
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Identification of ALOX5 as a gene regulating adiposity and pancreatic function by Mehrabian, M, Schulthess, F. T, Nebohacova, M, Castellani, L. W, Zhou, Z, Hartiala, J, Oberholzer, J, Lusis, A. J, Maedler, K, Allayee, H

“…Aims/hypothesis We previously used an integrative genetics approach to demonstrate that 5-lipoxygenase (5-LO) deficiency in mice (Alox5 ⁻/⁻) protects against…”
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Erratum to: Purinergic P2X7 receptors regulate secretion of interleukin-1 receptor antagonist and beta cell function and survival by Glas, R., Sauter, N. S., Schulthess, F. T., Shu, L., Oberholzer, J., Maedler, K.

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TOSO promotes β-cell proliferation and protects from apoptosis by Dharmadhikari, G, Mühle, M, Schulthess, F.T, Laue, S, Oberholzer, J, Pattou, F, Kerr-Conte, J, Maedler, K

“…Abstract Decreased β-cell mass reflects a shift from quiescence/proliferation into apoptosis, it plays a crucial role in the pathophysiology of diabetes. A…”
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Expression of Concern: Deletion of the Mitochondrial Flavoprotein Apoptosis Inducing Factor (AIF) Induces β-Cell Apoptosis and Impairs β-Cell Mass

“…The corresponding author stated that the leftmost b-Actin panel in Fig 1A in [1], which served as loading control for the protein lysates of isolated islets…”
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Erratum to: Purinergic P2X^sub 7^ receptors regulate secretion of interleukin-1 receptor antagonist and beta cell function and survival by Glas, R, Sauter, N S, Schulthess, F T, Shu, L, Oberholzer, J, Maedler, K

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Purinergic P2X sub(7) receptors regulate secretion of interleukin-1 receptor antagonist and beta cell function and survival by Glas, R, Sauter, N S, Schulthess, F T, Shu, L, Oberholzer, J, Maedler, K

“…Aims/hypothesis: In obesity, beta cells activate compensatory mechanisms to adapt to the higher insulin demand. Interleukin-1 receptor antagonist (IL-1Ra)…”
Get full text