Search Results - "Schneider, Dina A"

Saturated fatty acids activate TLR-mediated proinflammatory signaling pathways[S] by Huang, Shurong, Rutkowsky, Jennifer M., Snodgrass, Ryan G., Ono-Moore, Kikumi D., Schneider, Dina A., Newman, John W., Adams, Sean H., Hwang, Daniel H.

“…Toll-like receptor 4 (TLR4) and TLR2 were shown to be activated by saturated fatty acids (SFAs) but inhibited by docosahexaenoic acid (DHA). However, one…”
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Inhibition of Neuroinflammation by AIBP: Spinal Effects upon Facilitated Pain States by Woller, Sarah A., Choi, Soo-Ho, An, Eun Jung, Low, Hann, Schneider, Dina A., Ramachandran, Roshni, Kim, Jungsu, Bae, Yun Soo, Sviridov, Dmitri, Corr, Maripat, Yaksh, Tony L., Miller, Yury I.

“…Apolipoprotein A-I binding protein (AIBP) reduces lipid raft abundance by augmenting the removal of excess cholesterol from the plasma membrane. Here, we…”
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AIBP protects against metabolic abnormalities and atherosclerosis[S] by Schneider, Dina A., Choi, Soo-Ho, Agatisa-Boyle, Colin, Zhu, Laurence, Kim, Jungsu, Pattison, Jennifer, Sears, Dorothy D., Gordts, PhilipL.S.M., Fang, Longhou, Miller, Yury I.

“…Apolipoprotein A-I binding protein (AIBP) has been shown to augment cholesterol efflux from endothelial cells and macrophages. In zebrafish and mice,…”
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Apoc2 loss-of-function zebrafish mutant as a genetic model of hyperlipidemia by Liu, Chao, Gates, Keith P, Fang, Longhou, Amar, Marcelo J, Schneider, Dina A, Geng, Honglian, Huang, Wei, Kim, Jungsu, Pattison, Jennifer, Zhang, Jian, Witztum, Joseph L, Remaley, Alan T, Dong, P Duc, Miller, Yury I

“…Apolipoprotein C-II (APOC2) is an obligatory activator of lipoprotein lipase. Human patients with APOC2 deficiency display severe hypertriglyceridemia while…”
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AIBP augments cholesterol efflux from alveolar macrophages to surfactant and reduces acute lung inflammation by Choi, Soo-Ho, Wallace, Aaron M., Schneider, Dina A., Burg, Elianne, Kim, Jungsu, Alekseeva, Elena, Ubags, Niki D.J., Cool, Carlyne D., Fang, Longhou, Suratt, Benjamin T., Miller, Yury I.

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AIBP augments cholesterol efflux from alveolar macrophages to surfactant and reduces acute lung inflammation by Choi, Soo-Ho, Wallace, Aaron M, Schneider, Dina A, Burg, Elianne, Kim, Jungsu, Alekseeva, Elena, Ubags, Niki Dj, Cool, Carlyne D, Fang, Longhou, Suratt, Benjamin T, Miller, Yury I

“…Acute respiratory distress syndrome (ARDS) is characterized by an excessive pulmonary inflammatory response. Removal of excess cholesterol from the plasma…”
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Transgenic mice with ectopic expression of constitutively active TLR4 in adipose tissues do not show impaired insulin sensitivity by Ono‐Moore, Kikumi D., Zhao, Ling, Huang, Shurong, Kim, Jeonga, Rutkowsky, Jennifer M., Snodgrass, Ryan G., Schneider, Dina A., Quon, Michael J., Graham, James L., Havel, Peter J., Hwang, Daniel H.

“…Introduction Chronic low‐grade inflammation is associated with obesity and diabetes. However, what causes and mediates chronic inflammation in metabolic…”
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Abstract 260: Apolipoprotein A-I Binding Protein Regulates Macrophage Polarization in Atherosclerosis by Schneider, Dina A, Fang, Longhou, Miller, Yury I

“…Abstract only Our laboratory recently demonstrated that Apolipoprotein A-I Binding Protein (AIBP), an evolutionarily conserved intracellular and secreted…”
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Improved insulin sensitivity and reduced adiposity with aP2 driven TLR4 overexpression in transgenic mice by Ono‐Moore, Kikumi D, Zhao, Ling, Huang, Shurong, Kim, Jeong‐a, Rutkowsky, Jennifer M, Snodgrass, Ryan G, Schneider, Dina A, Quon, Michael J, Hwang, Daniel H

“…Abstract only Chronic low grade inflammation is associated with obesity and diabetes. The underlying causes and mechanisms mediating chronic low grade…”
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Saturated fatty acids activate TLR‐mediated proinflammatory signaling pathways by Hwang, Daniel, Huang, Shurong, Rutkowsky, Jennifer M., Snodgrass, Ryan G., Ono-Moore, Kikumi D., Schneider, Dina A., Newman, John W., Adams, Sean H.

“…Abstract only Toll‐like receptor 4 (TLR4) and TLR2 were shown to be activated by saturated fatty acids (SFAs) but inhibited by docosahexaenoic acid (DHA)…”
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