Search Results - "Sanelli, Leo"
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Recovery of motoneuron and locomotor function after spinal cord injury depends on constitutive activity in 5-HT2C receptors
Published in Nature medicine (01-06-2010)“…Spinal cord injury leads to flaccid paralysis resulting from the loss of descending serotonergic modulation. Murray et al . demonstrate that spontaneous…”
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Pericytes impair capillary blood flow and motor function after chronic spinal cord injury
Published in Nature medicine (01-06-2017)“…In rat models of spinal cord injury, the region of the spinal cord below the site of injury becomes hypoxic owing to inadequate blood flow. This effect on…”
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Decrease of mRNA Editing after Spinal Cord Injury is Caused by Down-regulation of ADAR2 that is Triggered by Inflammatory Response
Published in Scientific reports (30-07-2015)“…We recently showed that spinal cord injury (SCI) leads to a decrease in mRNA editing of serotonin receptor 2C (5-HT 2C R) contributing to post-SCI spasticity…”
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Tail Muscles Become Slow but Fatigable in Chronic Sacral Spinal Rats With Spasticity
Published in Journal of neurophysiology (01-02-2006)“…Centre for Neuroscience, University of Alberta, Edmonton, Canada Submitted 3 May 2005; accepted in final form 3 November 2005 Paralyzed skeletal muscle…”
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Spastic Tail Muscles Recover From Myofiber Atrophy and Myosin Heavy Chain Transformations in Chronic Spinal Rats
Published in Journal of neurophysiology (01-02-2007)“…Centre for Neuroscience, University of Alberta, Edmonton, Canada Submitted 14 June 2006; accepted in final form 7 November 2006 Without intervention after…”
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Recovery of motoneuron and locomotor function after spinal cord injury depends on constitutive activity in 5-HT 2C receptors
Published in Nature medicine (01-06-2010)Get full text
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Recovery of motoneuron and locomotor function after spinal cord injury depends on constitutive activity in 5-HT sub(2C) receptors
Published in Nature medicine (01-06-2010)“…Muscle paralysis after spinal cord injury is partly caused by a loss of brainstem-derived serotonin (5-HT), which normally maintains motoneuron excitability by…”
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Recovery of motoneuron and locomotor function after spinal cord injury depends on constitutive activity in [5-HT.sub.2C] receptors
Published in Nature medicine (01-06-2010)“…Muscle paralysis after spinal cord injury is partly caused by a loss of brainstem-derived serotonin (5-HT), which normally maintains motoneuron excitability by…”
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