Search Results - "Sadredini, Mani"

Myocardial oxidative stress is increased in early reperfusion, but systemic antioxidative therapy does not prevent ischemia-reperfusion arrhythmias in pigs by Haugsten Hansen, Marie, Sadredini, Mani, Hasic, Almira, Eriksen, Morten, Stokke, Mathis Korseberg

“…BackgroundArrhythmias in the early phase of reperfusion after myocardial infarction (MI) are common, and can lead to hemodynamic instability or even cardiac…”
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Arrhythmia initiation in catecholaminergic polymorphic ventricular tachycardia type 1 depends on both heart rate and sympathetic stimulation by Danielsen, Tore K, Manotheepan, Ravinea, Sadredini, Mani, Leren, Ida S, Edwards, Andrew G, Vincent, Kevin P, Lehnart, Stephan E, Sejersted, Ole M, Sjaastad, Ivar, Haugaa, Kristina H, Stokke, Mathis K

“…Catecholaminergic polymorphic ventricular tachycardia type 1 (CPVT1) predisposes to ventricular tachyarrhythmias (VTs) during high heart rates due to physical…”
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Beta-Adrenoceptor Stimulation Reveals Ca2+ Waves and Sarcoplasmic Reticulum Ca2+ Depletion in Left Ventricular Cardiomyocytes from Post-Infarction Rats with and without Heart Failure by Sadredini, Mani, Danielsen, Tore Kristian, Aronsen, Jan Magnus, Manotheepan, Ravinea, Hougen, Karina, Sjaastad, Ivar, Stokke, Mathis Korseberg

“…Abnormal cellular Ca2+ handling contributes to both contractile dysfunction and arrhythmias in heart failure. Reduced Ca2+ transient amplitude due to decreased…”
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The oxidation‐resistant CaMKII‐MM281/282VV mutation does not prevent arrhythmias in CPVT1 by Sadredini, Mani, Manotheepan, Ravinea, Lehnart, Stephan E., Anderson, Mark E., Sjaastad, Ivar, Stokke, Mathis K.

“…Catecholaminergic polymorphic ventricular tachycardia type 1 (CPVT1) is an inherited arrhythmogenic disorder caused by missense mutations in the cardiac…”
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Exercise Training Stabilizes RyR2-Dependent Ca2+ Release in Post-infarction Heart Failure by Danielsen, Tore Kristian, Sadredini, Mani, Manotheepan, Ravinea, Aronsen, Jan Magnus, Frisk, Michael, Hansen, Marie Haugsten, Andressen, Kjetil Wessel, Hougen, Karina, Levy, Finn Olav, Louch, William E., Sejersted, Ole Mathias, Sjaastad, Ivar, Stokke, Mathis Korseberg

“…Aim: Dysfunction of the cardiac ryanodine receptor (RyR2) is an almost ubiquitous finding in animal models of heart failure (HF) and results in abnormal Ca 2+…”
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Abstract 11558: The Role of Oxidized CaMKII in Early Ischemia-Reperfusion Triggered Ventricular Arrhythmias by Haugsten Hansen, Marie, Sadredini, Mani, Hasic, Almira, Anderson, Mark E, Sjaastad, Ivar, Stokke, Mathis

“…IntroductionArrhythmias can complicate revascularization procedures after transmural infarctions and induce hemodynamic instability, but most cases can be…”
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CaMKII and reactive oxygen species contribute to early reperfusion arrhythmias, but oxidation of CaMKIIδ at methionines 281/282 is not a determining factor by Hansen Haugsten, Marie, Sadredini, Mani, Hasic, Almira, Anderson, Mark E, Sjaastad, Ivar, Stokke, Mathis Korseberg

“…Background Available evidence suggest that Ca2+/calmodulin-dependent protein kinase type IIδ (CaMKIIδ) and reactive oxygen species (ROS) are important in early…”
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CaMKII and reactive oxygen species contribute to early reperfusion arrhythmias, but oxidation of CaMKIIδ at methionines 281/282 is not a determining factor by Haugsten Hansen, Marie, Sadredini, Mani, Hasic, Almira, Anderson, Mark E., Sjaastad, Ivar, Korseberg Stokke, Mathis

“…Available evidence suggest that Ca2+/calmodulin-dependent protein kinase type IIδ (CaMKIIδ) and reactive oxygen species (ROS) are important in early…”
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Beneficial effects of exercise initiated before development of hypertrophic cardiomyopathy in genotype-positive mice by Andreassen, Kristine, Rixon, Chloe, Hansen, Marie H, Hauge-Iversen, Ida Marie, Zhang, Lili, Sadredini, Mani, Erusappan, Pugazendhi M, Sjaastad, Ivar, Christensen, Geir, Haugaa, Kristina H, Edvardsen, Thor, Lunde, Ida G, Stokke, Mathis K

“…The effect of exercise on disease development in hypertrophic cardiomyopathy (HCM) genotype-positive individuals is unresolved. Our objective was to test the…”
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Exercise training prevents ventricular tachycardia in CPVT1 due to reduced CaMKII-dependent arrhythmogenic Ca2+ release by Manotheepan, Ravinea, Danielsen, Tore K, Sadredini, Mani, Anderson, Mark E, Carlson, Cathrine R, Lehnart, Stephan E, Sjaastad, Ivar, Stokke, Mathis K

“…Catecholaminergic polymorphic ventricular tachycardia type 1 (CPVT1) is caused by mutations in the cardiac ryanodine receptor (RyR2) that lead to disrupted…”
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Beta-Adrenoceptor Stimulation Reveals Ca.sup.2+ Waves and Sarcoplasmic Reticulum Ca.sup.2+ Depletion in Left Ventricular Cardiomyocytes from Post-Infarction Rats with and without Heart Failure by Sadredini, Mani, Danielsen, Tore Kristian, Aronsen, Jan Magnus, Manotheepan, Ravinea, Hougen, Karina, Sjaastad, Ivar, Stokke, Mathis Korseberg

“…Abnormal cellular Ca.sup.2+ handling contributes to both contractile dysfunction and arrhythmias in heart failure. Reduced Ca.sup.2+ transient amplitude due to…”
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Exercise training prevents ventricular tachycardia in CPVT1 due to reduced CaMKII-dependent arrhythmogenic Ca 2+ release by Manotheepan, Ravinea, Danielsen, Tore K., Sadredini, Mani, Anderson, Mark E., Carlson, Cathrine R., Lehnart, Stephan E., Sjaastad, Ivar, Stokke, Mathis K.

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Secretoneurin Is an Endogenous Calcium/Calmodulin-Dependent Protein Kinase II Inhibitor That Attenuates Ca2+-Dependent Arrhythmia by Ottesen, Anett H, Carlson, Cathrine R, Eken, Olav Søvik, Sadredini, Mani, Myhre, Peder L, Shen, Xin, Dalhus, Bjørn, Laver, Derek R, Lunde, Per Kristian, Kurola, Jouni, Lunde, Marianne, Hoff, Jon Erik, Godang, Kristin, Sjaastad, Ivar, Pettilä, Ville, Stridsberg, Mats, Lehnart, Stephan E, Edwards, Andrew G, Lunde, Ida G, Omland, Torbjørn, Stokke, Mathis K, Christensen, Geir, Røsjø, Helge, Louch, William E

“…BACKGROUND:Circulating SN (secretoneurin) concentrations are increased in patients with myocardial dysfunction and predict poor outcome. Because SN inhibits…”
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Ca2+ Wave Velocity in Cardiomyocytes is Regulated by Ryanodine Receptor Ca2+ Sensitivity and SR Ca2+ Content by Loose, Kristian O., Sadredini, Mani N., Sejersted, Ole M., Stokke, Mathis K., Louch, William E.

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Secretoneurin Is an Endogenous Calcium/Calmodulin-Dependent Protein Kinase II Inhibitor That Attenuates Ca 2+ -Dependent Arrhythmia by Ottesen, Anett H, Carlson, Cathrine R, Eken, Olav Søvik, Sadredini, Mani, Myhre, Peder L, Shen, Xin, Dalhus, Bjørn, Laver, Derek R, Lunde, Per Kristian, Kurola, Jouni, Lunde, Marianne, Hoff, Jon Erik, Godang, Kristin, Sjaastad, Ivar, Pettilä, Ville, Stridsberg, Mats, Lehnart, Stephan E, Edwards, Andrew G, Lunde, Ida G, Omland, Torbjørn, Stokke, Mathis K, Christensen, Geir, Røsjø, Helge, Louch, William E

“…Circulating SN (secretoneurin) concentrations are increased in patients with myocardial dysfunction and predict poor outcome. Because SN inhibits CaMKIIδ (Ca…”
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Exercise Training Stabilizes RyR2-Dependent Ca 2+ Release in Post-infarction Heart Failure by Danielsen, Tore Kristian, Sadredini, Mani, Manotheepan, Ravinea, Aronsen, Jan Magnus, Frisk, Michael, Hansen, Marie Haugsten, Andressen, Kjetil Wessel, Hougen, Karina, Levy, Finn Olav, Louch, William E, Sejersted, Ole Mathias, Sjaastad, Ivar, Stokke, Mathis Korseberg

“…Dysfunction of the cardiac ryanodine receptor (RyR2) is an almost ubiquitous finding in animal models of heart failure (HF) and results in abnormal Ca release…”
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