Search Results - "STAHL, R. A. K"

Rituximab in adult patients with immunosuppressive-dependent minimal change disease by Hoxha, E, Stahl, R A K, Harendza, S

“…Minimal change disease (MCD) is one of the leading causes of nephrotic syndrome. Steroid therapy is effective in achieving remission, but relapses, steroid…”
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Antihypertensive therapy upregulates renin and (pro)renin receptor in the clipped kidney of Goldblatt hypertensive rats by Krebs, C., Hamming, I., Sadaghiani, S., Steinmetz, O.M., Meyer-Schwesinger, C., Fehr, S., Stahl, R.A.K., Garrelds, I.M., Danser, A.H.J., van Goor, H., Contrepas, A., Nguyen, G., Wenzel, U.

“…Recently, a (pro)renin receptor has been identified which mediates profibrotic effects independent of angiotensin II. Because antihypertensive therapy induces…”
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A new mouse model of immune-mediated podocyte injury by Meyer, T.N., Schwesinger, C., Wahlefeld, J., Dehde, S., Kerjaschki, D., Becker, J.U., Stahl, R.A.K., Thaiss, F.

“…Podocytes play a major role in the initiation and progression of glomerular diseases and are a target of both immune-mediated and non-immune-mediated injury…”
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Antihypertensive therapy induces compartment-specific chemokine expression and a Th1 immune response in the clipped kidney of Goldblatt hypertensive rats by Steinmetz, O M, Sadaghiani, S, Panzer, U, Krebs, C, Meyer-Schwesinger, C, Streichert, T, Fehr, S, Hamming, I, van Goor, H, Stahl, R A K, Wenzel, U

“…The present study examined the pathogenesis of interstitial inflammation and fibrosis in antihypertensively treated rats with two-kidney, one-clip…”
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A pitfall of glomerular sieving: profibrotic and matrix proteins derive from the Bowman's capsule and not the glomerular tuft in rats with renovascular hypertension by Steinmetz, O. M., Panzer, U., Fehr, S., Meyer-Schwesinger, C., Stahl, R. A. K., Wenzel, U. O.

“…Background. The glomeruli in the non-clipped kidney of rats with 2-kidney, 1-clip hypertension are a classical model for studying the mechanisms of glomerular…”
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Statin therapy in patients with chronic kidney disease: to use or not to use by Steinmetz, O. M., Panzer, U., Stahl, R. A. K., Wenzel, U. O.

“…Dyslipdemia is a common complication of chronic kidney disease (CKD) and contributes to high cardiovascular morbidity and mortality of CKD patients…”
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new role for the neuronal ubiquitin C-terminal hydrolase-L1 (UCH-L1) in podocyte process formation and podocyte injury in human glomerulopathies by Meyer-Schwesinger, C, Meyer, TN, Münster, S, Klug, P, Saleem, M, Helmchen, U, Stahl, RAK

“…Glomerular epithelial cell (podocyte) injury is characterized by foot process retraction, slit diaphragm reorganization, and degradation of podocyte-specific…”
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RORγt expression in Tregs promotes systemic lupus erythematosus via IL‐17 secretion, alteration of Treg phenotype and suppression of Th2 responses by Kluger, M. A., Nosko, A., Ramcke, T., Goerke, B., Meyer, M. C., Wegscheid, C., Luig, M., Tiegs, G., Stahl, R. A. K., Steinmetz, O. M.

“…Summary Systemic lupus erythematosus (SLE) is a common autoimmune disorder with a complex and poorly understood immunopathogenesis. However, a pathogenic role…”
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PDGF-B gene single-nucleotide polymorphisms are not predictive for disease onset or progression of IgA nephropathy by Bicanski, B, Wenderdel, M, Mertens, Peter R, Senderek, J, Panzer, U, Steinmetz, O, Stahl, R A K, Cerullo, G, Diletta Torres, D, Schena, F P, Zerres, K, Floege, J

“…Few genetic factors have been identified that determine susceptibility to and progression of IgA-nephropathy (IgAN). Given that IgAN is usually characterized…”
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UCH-L1 induces podocyte hypertrophy in membranous nephropathy by protein accumulation by Lohmann, Frithjof, Sachs, Marlies, Meyer, Tobias N., Sievert, Henning, Lindenmeyer, Maja T., Wiech, Thorsten, Cohen, Clemens D., Balabanov, Stefan, Stahl, R.A.K., Meyer-Schwesinger, Catherine

“…Podocytes are terminally differentiated cells of the glomerular filtration barrier that react with hypertrophy in the course of injury such as in membranous…”
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Rapid development of severe end-organ damage in C57BL/6 mice by combining DOCA salt and angiotensin II by Kirchhoff, F., Krebs, C., Abdulhag, U.N., Meyer-Schwesinger, C., Maas, R., Helmchen, U., Hilgers, K.F., Wolf, G., Stahl, R.A.K., Wenzel, U.

“…The C57BL/6 mouse strain serves as the genetic background of many transgenic and gene knockout models; however, this strain appears to be resistant to…”
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Membranous nephropathy--crucial developments for diagnostic and treatment by Stahl, R A K, Harendza, S, Helmchen, U, Hoxha, E

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Immunosuppression in membranous glomerulonephritis - pro by Stahl, R A K

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Erk 1,2 phosphorylates p27Kip1: Functional evidence for a role in high glucose-induced hypertrophy of mesangial cells by WOLF, G, REINKING, R, ZAHNER, G, STAHL, R. A. K, SHANKLAND, S. J

“…Mesangial cell hypertrophy is one of the earliest morphological abnormalities of diabetic nephropathy. We have previously shown that high glucose induces…”
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Erk 1,2 phosphorylates p27 Kip1 : Functional evidence for a role in high glucose-induced hypertrophy of mesangial cells by Wolf, G., Reinking, R., Zahner, G., Stahl, R. A. K., Shankland, S. J.

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Cyclooxygenase-2 (COX-2) and the kidney: current status and potential perspectives by Schneider, André, Stahl, Rolf A. K.

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An immunofluorescence test for phospholipase-A2-receptor antibodies and its clinical usefulness in patients with membranous glomerulonephritis by Hoxha, Elion, Harendza, Sigrid, Zahner, Gunter, Panzer, Ulf, Steinmetz, Oliver, Fechner, Kai, Helmchen, Udo, Stahl, Rolf A.K.

“…Background. The recent finding that phospholipase-A2-receptor antibodies (PLA2R-AB) may play a role in the development of primary membranous glomerulonephritis…”
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Angiotensinase A gene expression and enzyme activity in isolated glomeruli of diabetic rats by THAISS, F, WOLF, G, ASSAD, N, ZAHNER, G, STAHL, R. A. K

“…One of the characteristics of early diabetic nephropathy is glomerular hyperfiltration and hyperperfusion. Many factors have been suggested to induce…”
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Isoproterenol stimulates tubular DNA replication in mice by Wolf, G., Helmchen, U., Stahl, R. A. K.

“…The β3-adrenergic stimulant isoproterenol is a mitogen for cultured murine proximal tubular cells. The present study was undertaken to test whether…”
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Angiotensin converting-enzyme inhibitor treatment reduces glomerular p16 INK4 and p27 Kip1 expression in diabetic BBdp rats by Wolf, G., Wenzel, U., Ziyadeh, F. N., Stahl, R. A. K.

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