Search Results - "SCHWANINGER, M"

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  1. 1

    NF-κB signaling in cerebral ischemia by Ridder, D.A, Schwaninger, M

    Published in Neuroscience (06-02-2009)
    “…Abstract The transcription factor NF-κB is a key regulator of hundreds of genes involved in cell survival and inflammation. There is ample evidence that NF-κB…”
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    Journal Article
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    Thrombomodulin--a new target for treating stroke at the crossroad of coagulation and inflammation by Wenzel, J, Assmann, J C, Schwaninger, M

    Published in Current medicinal chemistry (2014)
    “…Thrombomodulin (TM) is a membrane protein mainly expressed by endothelial cells. It is part of the anticoagulant protein C system but recently several effects…”
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    Journal Article
  3. 3

    Acute inhibition of TAK1 protects against neuronal death in cerebral ischemia by Neubert, M, Ridder, D A, Bargiotas, P, Akira, S, Schwaninger, M

    Published in Cell death and differentiation (01-09-2011)
    “…Neuronal apoptosis contributes to ischemic brain damage and neurodegenerative disorders. Key regulators of neuronal apoptosis are the transcription factor NF-…”
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  4. 4

    Neuroprotective effect of granulocyte colony-stimulating factor after focal cerebral ischemia by SCHÄBITZ, W.-R, KOLLMAR, R, SCHWANINGER, M, JUETTLER, E, BARDUTZKY, J, SCHÖLZKE, M. N, SOMMER, C, SCHWAB, S

    Published in Stroke (1970) (01-03-2003)
    “…The potential neuroprotective effect of the granulocyte colony-stimulating factor (G-CSF) after glutamate-induced excitotoxicity in cell culture and after…”
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    Journal Article
  5. 5

    The role of TWEAK/Fn14 signaling in the MPTP-model of Parkinson’s disease by Mustafa, S, Martin, H.L, Burkly, L, Costa, A, Martins, M.L, Schwaninger, M, Teismann, P

    Published in Neuroscience (05-04-2016)
    “…Highlights • We investigate the role of TWEAK and Fn14 in a model of Parkinson’s disease. • Ablation of TWEAK or Fn14 had no effect on acute MPTP toxicity. •…”
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    Journal Article
  6. 6

    Harmonizing and improving European education in prescribing: An overview of digital educational resources used in clinical pharmacology and therapeutics by Bakkum, Michiel J., Tichelaar, Jelle, Papaioannidou, Paraskevi, Likic, Robert, Sanz Alvarez, Emilio J., Christiaens, Thierry, Costa, João N., Mačiulaitis, Romaldas, Dima, Lorena, Coleman, Jamie, Richir, Milan C., Agtmael, Michiel A., Atanasova, Ivanka, Ganeva, Maria, Gatchev, Emil, Kostadinova, I. I., Mimica Matanovic, S., Vitezic, D, Greta, Wozniak, Kmonickova, E., Karel, Urbanek, Damkier, P., Huupponen, R. K., Auffret, Marine, Bejan‐Angoulvant, T., Laurent, Chouchana, Jean‐Luc, Cracowski, Drici, M. D., Faillie, J. L., Hélène, Geniaux, Molimard, M., Orlikowski, D., Palin, Karine, Pers, Y.‐M., Picard, Nicolas, Simon, N., Toussirot, E., Boger, R. H., Cascorbi, I., Mueller, S. C., Regenthal, R., Schwab, M., Schwaninger, M. S., Thuermann, P. A., Wojnowski, L., Kouvelas, D., Riba, P., Kerins, David M., Williams, David J., Cosentino, M., De Ponti, Fabrizio, Filippelli, Amelia, Leone, R., Locatelli, Vittorio, Jansone, Baiba, Gulbinovic, Romaldas, Mifsud, Janet, Braszko Jan, J., Kocic, I., Luiza, Breitenfeld, Castelo‐Branco, M., Simona, Conea, Ioan, Magyar, Bevc, S., Mojca, Krzan, Bernal, M. L., Capellà, D., Carcas, A., De Abajo, F. J., Lopez‐Rico, M., Lucena, M. I., Pontes, C., Sanz, E. J., Böttiger, Y., Le Grevès, Madeleine, Waard‐Siebinga, I., Janssen Ben, J. A., Wilma, Knol, Rahul, Pandit, Rosse, F., Dent, G., Albert, Ferro, Hitchings, A. W., Kapil, V., Linton, K. D., Loke, Y. K., Michael, Okorie, David, Plumb Richard, Pontefract, Sarah, Ranmuthu, S., Sampson, A. P., Thanacoody, H. K. R., Whitfield Jonathan, P., Wilson, Kurt

    Published in British journal of clinical pharmacology (01-03-2021)
    “…Aim Improvement and harmonization of European clinical pharmacology and therapeutics (CPT) education is urgently required. Because digital educational…”
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    Journal Article
  7. 7

    Granzyme-b is involved in mediating post-ischemic neuronal death during focal cerebral ischemia in rat model by Chaitanya, G.V, Schwaninger, M, Alexander, J.S, Babu, P. Prakash

    Published in Neuroscience (17-02-2010)
    “…Abstract Although peripheral immune cells infiltrate ischemic infarct tissue and elicit immune injury, the role of Cytotoxic T Lymphocytes (CTLs) and the…”
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    Evasion of Early Cellular Response Mechanisms following Low Level Radiation-induced DNA Damage by Collis, Spencer J., Schwaninger, Julie M., Ntambi, Alfred J., Keller, Thomas W., Nelson, William G., Dillehay, Larry E., DeWeese, Theodore L.

    Published in The Journal of biological chemistry (26-11-2004)
    “…DNA damage that is not repaired with high fidelity can lead to chromosomal aberrations or mitotic cell death. To date, it is unclear what factors control the…”
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    Combined toxicity of penicillin and aspirin therapy may elicit bilateral vestibulopathy by Hertel, S, Schwaninger, M, Helmchen, C

    Published in Clinical neurology and neurosurgery (01-07-2013)
    “…[...]aspirin has been found to modulate cochlear neurotoxicity of gentamicin, but an interaction between aspirin and penicillin has not been examined yet…”
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  12. 12

    A Transgenic Approach to Identify Thyroxine Transporter-Expressing Structures in Brain Development by Lang, M.-F., Salinin, S., Ridder, D. A., Kleesiek, J., Hroudova, J., Berger, S., Schütz, G., Schwaninger, M.

    Published in Journal of neuroendocrinology (01-12-2011)
    “…Transporters are essential in thyroid hormone metabolism. Thyroxine (T4) is transported by solute carrier organic anion transporter 1c1 (SLCO1C1, OATP14) into…”
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    Journal Article
  13. 13

    Intravenous brain-derived neurotrophic factor reduces infarct size and counterregulates Bax and Bcl-2 expression after temporary focal cerebral ischemia by Schäbitz, W R, Sommer, C, Zoder, W, Kiessling, M, Schwaninger, M, Schwab, S

    Published in Stroke (1970) (01-09-2000)
    “…Pretreatment with intraventricular brain-derived neurotrophic factor (BDNF) reduces ischemic damage after focal cerebral ischemia. In this experiment we…”
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  14. 14

    Expression of the kcnk3 potassium channel gene lessens the injury from cerebral ischemia, most likely by a general influence on blood pressure by Muhammad, S, Aller, M.I, Maser-Gluth, C, Schwaninger, M, Wisden, W

    Published in Neuroscience (19-05-2010)
    “…Abstract We examined the possible protective effect of TASK-1 (TWIK-related acid-sensitive potassium channel-1, kcnk3) and -3 potassium channels during stroke…”
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    Cerebrovascular manifestations of Takayasu arteritis in Europe by Ringleb, P. A., Strittmatter, E. I., Loewer, M., Hartmann, M., Fiebach, J. B., Lichy, C., Weber, R., Jacobi, C., Amendt, K., Schwaninger, M.

    Published in Rheumatology (Oxford, England) (01-08-2005)
    “…Objectives. Takayasu arteritis is well known as a cause of stroke in Asia but has rarely been described in the Western world. Here we report the clinical and…”
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    Journal Article
  17. 17

    The ICAM-1 E469K gene polymorphism is a risk factor for spontaneous cervical artery dissection by LONGONI, M, GROND-GINSBACH, C, GRAU, A. J, GENIUS, J, DEBETTE, S, SCHWANINGER, M, FERRARESE, C, LICHY, C

    Published in Neurology (25-04-2006)
    “…In a primary study on proinflammatory genetic profiles in stroke, the authors found the E469K polymorphism of the intercellular adhesion molecule 1 (ICAM-1)…”
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  18. 18

    Guidelines for the Diagnosis and Management of Food Allergy in the United States: Report of the NIAID-Sponsored Expert Panel by FAUCI, Antony S

    Published in Journal of allergy and clinical immunology (01-12-2010)
    “…Food allergy is an important public health problem that affects children and adults and may be increasing in prevalence. Despite the risk of severe allergic…”
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    Journal Article Conference Proceeding
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    Induction of Interleukin-6 by Depolarization of Neurons by Sallmann, Svea, Juttler, Eric, Prinz, Simone, Petersen, Nicole, Knopf, Udo, Weiser, Thomas, Schwaninger, Markus

    Published in The Journal of neuroscience (01-12-2000)
    “…Interleukin-6 (IL-6) has neuromodulatory and neuroprotective effects in vivo. It is expressed in glial cells and neurons both under physiological conditions…”
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    Journal Article