Search Results - "SCANGA, C. B"

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  1. 1

    Allergic airway inflammation is exacerbated during acute influenza infection and correlates with increased allergen presentation and recruitment of allergen-specific T-helper type 2 cells by Marsland, B. J., Scanga, C. B., Kopf, M., Le Gros, G.

    Published in Clinical and experimental allergy (01-08-2004)
    “…Summary Background Respiratory viral infections are a leading cause of the hospitalization of asthmatics, however, the cellular immunological interactions…”
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    Journal Article
  2. 2

    Development of an asthma vaccine: Research into BCG by SCANGA, C. B, LE GROS, G

    Published in Drugs (New York, N.Y.) (01-06-2000)
    “…Asthma is an atopic disorder characterised by the activation and recruitment of eosinophils to the lung resulting in chronic swelling and inflammation of the…”
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    Journal Article
  3. 3

    Effects of weight loss and exercise training on natural killer cell activity in obese women by SCANGA, C. B, VERDE, T. J, PAOLONE, A. M, ANDERSEN, R. E, WADDEN, T. A

    “…The purposes of this study were two-fold: (1) to evaluate the effects of an 8-wk weight loss program on natural killer (NK) cell activity in obese women and 2)…”
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    Journal Article
  4. 4

    Dendritic cell elimination as an assay of cytotoxic T lymphocyte activity in vivo by Ritchie, David S, Hermans, Ian F, Lumsden, Joanne M, Scanga, Connie B, Roberts, Joanna M, Yang, Jiangping, Kemp, Roslyn A, Ronchese, Franca

    Published in Journal of immunological methods (01-12-2000)
    “…We show in this paper that the survival of antigen-loaded dendritic cells in vivo may be used as a sensitive readout of CTL activity. We have previously shown…”
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    Journal Article
  5. 5

    Suppression of allergic airway disease using mycobacterial lipoglycans by Sayers, Ian, Severn, Wayne, Scanga, Connie B, Hudson, Jenny, Le Gros, Graham, Harper, Jacquie L

    Published in Journal of allergy and clinical immunology (01-08-2004)
    “…Administration of heat-killed mycobacteria can suppress allergic disease in mice and humans. The active components of mycobacteria mediating these effects…”
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    Journal Article Conference Proceeding