Search Results - "Rosas, Odrick"

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  1. 1

    A new mouse model of mild ornithine transcarbamylase deficiency (spf-j) displays cerebral amino acid perturbations at baseline and upon systemic immune activation by Tarasenko, Tatyana N, Rosas, Odrick R, Singh, Larry N, Kristaponis, Kara, Vernon, Hilary, McGuire, Peter J

    Published in PloS one (03-02-2015)
    “…Ornithine transcarbamylase deficiency (OTCD, OMIM# 311250) is an inherited X-linked urea cycle disorder that is characterized by hyperammonemia and orotic…”
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    Journal Article
  2. 2

    Expression and activation of ephexin is altered after spinal cord injury by Rosas, Odrick R., Figueroa, Johnny D., Torrado, Aranza I., Rivera, Mónica, Santiago, José M., Konig‐Toro, Franchesca, Miranda, Jorge D.

    Published in Developmental neurobiology (Hoboken, N.J.) (01-07-2011)
    “…Failure of axon regeneration after traumatic spinal cord injury (SCI) is attributable in part to the presence of inhibitory molecular interactions. Recent…”
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    Journal Article
  3. 3

    Long-term treatment with PP2 after spinal cord injury resulted in functional locomotor recovery and increased spared tissue by Rosas, Odrick R, Torrado, Aranza I, Santiago, Jose M, Rodriguez, Ana E, Salgado, Iris K, Miranda, Jorge D

    Published in Neural regeneration research (15-12-2014)
    “…The spinal cord has the ability to regenerate but the microenvironment generated after trauma reduces that capacity. An increase in Src family kinase (SFK)…”
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    Journal Article
  4. 4

    Blockade of P2 Nucleotide Receptors After Spinal Cord Injury Reduced the Gliotic Response and Spared Tissue by Rodríguez-Zayas, Ana E., Torrado, Aranza I., Rosas, Odrick R., Santiago, José M., Figueroa, Johnny D., Miranda, Jorge D.

    Published in Journal of molecular neuroscience (01-01-2012)
    “…Spinal cord injury (SCI) triggers a sequel of events commonly associated with cell death and dysfunction of glias and neurons surrounding the lesion. Although…”
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    Journal Article
  5. 5

    Expression Profile of Flotillin-2 and Its Pathophysiological Role After Spinal Cord Injury by Santiago, José M., Torrado, Aranza I., Arocho, Luz C., Rosas, Odrick R., Rodríguez, Ana E., Toro, Franchesca König, Salgado, Iris K., Torres, Yaría Arroyo, Silva, Walter I., Miranda, Jorge D.

    Published in Journal of molecular neuroscience (01-02-2013)
    “…Some receptors that block axonal regeneration or promote cell death after spinal cord injury (SCI) are localized in membrane rafts. Flotillin-2 (Flot-2) is an…”
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    Journal Article
  6. 6

    Molecular, anatomical, physiological, and behavioral studies of rats treated with buprenorphine after spinal cord injury by Santiago, José M, Rosas, Odrick, Torrado, Aranza I, González, María M, Kalyan-Masih, Priya O, Miranda, Jorge D

    Published in Journal of neurotrauma (01-10-2009)
    “…Acute pain is a common symptom experienced after spinal cord injury (SCI). The presence of this pain calls for treatment with analgesics, such as…”
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    Journal Article
  7. 7

    A New Mouse Model of Mild Ornithine Transcarbamylase Deficiency (spf-j) Displays Cerebral Amino Acid Perturbations at Baseline and upon Systemic Immune Activation: e0116594 by Tarasenko, Tatyana N, Rosas, Odrick R, Singh, Larry N, Kristaponis, Kara, Vernon, Hilary, McGuire, Peter J

    Published in PloS one (01-02-2015)
    “…Ornithine transcarbamylase deficiency (OTCD, OMIM# 311250) is an inherited X-linked urea cycle disorder that is characterized by hyperammonemia and orotic…”
    Get full text
    Journal Article
  8. 8
  9. 9

    Long-term treatment with PP2 after spinal cord injur y resulted in functional locomotor recover y and increased spared tissue by Odrick R Rosas, Aranza I Torrado, Jose M Santiago, Ana E Rodriguez, Iris K Salgado, Jorge D Miranda

    “…The spinal cord has the ability to regenerate but the microenvironment generated after trauma reduces that capacity. An increase in Src family kinase (SFK)…”
    Get full text
    Journal Article