Search Results - "Rodrigues, Mariliza V."

Uncovering emergent phenotypes in endothelial cells by clustering of surrogates of cardiovascular risk factors by Pinheiro-de-Sousa, Iguaracy, Fonseca-Alaniz, Miriam H., Teixeira, Samantha K., Rodrigues, Mariliza V., Krieger, Jose E.

“…Endothelial dysfunction (ED) is a hallmark of atherosclerosis and is influenced by well-defined risk factors, including hypoxia, dyslipidemia, inflammation,…”
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A Novel Aldosterone Antagonist Limits Renal Injury in 5/6 Nephrectomy by Fujihara, Clarice K., Kowala, M. C., Breyer, M. D., Sena, Claudia R., Rodrigues, Mariliza V., Arias, Simone C. A., Fanelli, Camilla, Malheiros, Denise M., Jadhav, P. K., Montrose-Rafizadeh, Chahrzad, Krieger, Jose E., Zatz, Roberto

“…Aldosterone antagonists slow the progression of chronic kidney disease (CKD), but their use is limited by hyperkalemia, especially when associated with RAS…”
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Retinoic Acid and VEGF Delay Smooth Muscle Relative to Endothelial Differentiation to Coordinate Inner and Outer Coronary Vessel Wall Morphogenesis by Azambuja, Ana P, Portillo-Sánchez, Victor, Rodrigues, Mariliza V, Omae, Samantha V, Schechtman, Deborah, Strauss, Bryan E, Costanzi-Strauss, Eugenia, Krieger, José E, Perez-Pomares, José M, Xavier-Neto, José

“…RATIONALE:Major coronary vessels derive from the proepicardium, the cellular progenitor of the epicardium, coronary endothelium, and coronary smooth muscle…”
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Programmed hypertension in rats treated with a NF-κB inhibitor during nephrogenesis: renal mechanisms by Canale, Daniele, Rodrigues, Mariliza V, Ferreira, Daniele N, Machado, Flavia G, Veras, Mariana M, Malheiros, Denise M A C, Krieger, José E, Fujihara, Clarice K, Venturini, Gabriela, Zatz, Roberto

“…Suppression of the renin-angiotensin system (RAS) during murine lactation causes progressive renal injury, indicating a physiological action of angiotensin II…”
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Thioredoxin interacting protein genetic variation is associated with diabetes and hypertension in the Brazilian general population by Ferreira, Noely E, Omae, Samantha, Pereira, Abel, Rodrigues, Mariliza V, Miyakawa, Ayumi A, Campos, Luciene C.G, Santos, Paulo C.J.L, Dallan, Luiz A, Martinez, Tânia L, Santos, Raul D, Mill, José G, Krieger, José E, Pereira, Alexandre C

“…Abstract Objective To investigate the relationship between TXNIP polymorphisms, diabetes and hypertension phenotypes in the Brazilian general population…”
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Progression of microalbuminuria in SHR is associated with lower expression of critical components of the apical endocytic machinery in the renal proximal tubule by Inoue, Bruna H, Arruda-Junior, Daniel F, Campos, Luciene C G, Barreto, Ana Luiza T, Rodrigues, Mariliza V, Krieger, José Eduardo, Girardi, Adriana C C

“…Cumulative epidemiological evidence indicates that the presence of microalbuminuria predicts a higher frequency of cardiovascular events, peripheral disease,…”
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Association between glutathione S-transferase polymorphisms and triglycerides and HDL-cholesterol by Maciel, Simone S, Pereira, Alexandre da C, Silva, Gustavo J.J, Rodrigues, Mariliza V, Mill, José G, Krieger, Jose E

“…Abstract Objective Null genotypes of glutathione S-transferase (GSTs) exhibit absence of enzymatic activity and are hypothesized to modulate an increased risk…”
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Using gene-network landscape to dissect genotype effects of TCF7L2 genetic variant on diabetes and cardiovascular risk by Vaquero, Andre R, Ferreira, Noely E, Omae, Samantha V, Rodrigues, Mariliza V, Teixeira, Samantha K, Krieger, Jose E, Pereira, Alexandre C

“…The single nucleotide polymorphism (SNP) within the TCF7L2 gene, rs7903146, is, to date, the most significant genetic marker associated with Type 2 diabetes…”
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Congenic strains provide evidence that four mapped loci in chromosomes 2, 4, and 16 influence hypertension in the SHR by Aneas, Ivy, Rodrigues, Mariliza V, Pauletti, Bianca A, Silva, Gustavo J. J, Carmona, Renata, Cardoso, Leandro, Kwitek, Anne E, Jacob, Howard J, Soler, Julia M. P, Krieger, Jose E

“…1 Heart Institute (InCor), University of São Paulo Medical School, São Paulo, SP, Brazil 2 Department of Statistics, Institute of Mathematics and Statistics,…”
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