Search Results - "Rey, Amaury"

NLRP3 phosphorylation in its LRR domain critically regulates inflammasome assembly by Niu, Tingting, De Rosny, Charlotte, Chautard, Séverine, Rey, Amaury, Patoli, Danish, Groslambert, Marine, Cosson, Camille, Lagrange, Brice, Zhang, Zhirong, Visvikis, Orane, Hacot, Sabine, Hologne, Maggy, Walker, Olivier, Wong, Jeimin, Wang, Ping, Ricci, Roméo, Henry, Thomas, Boyer, Laurent, Petrilli, Virginie, Py, Bénédicte F.

“…NLRP3 controls the secretion of inflammatory cytokines IL-1β/18 and pyroptosis by assembling the inflammasome. Upon coordinated priming and activation stimuli,…”
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Escherichia coli Rho GTPase-activating toxin CNF1 mediates NLRP3 inflammasome activation via p21-activated kinases-1/2 during bacteraemia in mice by Dufies, Océane, Doye, Anne, Courjon, Johan, Torre, Cédric, Michel, Gregory, Loubatier, Celine, Jacquel, Arnaud, Chaintreuil, Paul, Majoor, Alissa, Guinamard, Rodolphe R., Gallerand, Alexandre, Saavedra, Pedro H. V., Verhoeyen, Els, Rey, Amaury, Marchetti, Sandrine, Ruimy, Raymond, Czerucka, Dorota, Lamkanfi, Mohamed, Py, Bénédicte F., Munro, Patrick, Visvikis, Orane, Boyer, Laurent

“…Inflammasomes are signalling platforms that are assembled in response to infection or sterile inflammation by cytosolic pattern recognition receptors. The…”
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Zeb1 represses TCR signaling, promotes the proliferation of T cell progenitors and is essential for NK1.1+ T cell development by Zhang, Jiang, Wencker, Mélanie, Marliac, Quentin, Berton, Aurore, Hasan, Uzma, Schneider, Raphaël, Laubreton, Daphné, Cherrier, Dylan E., Mathieu, Anne-Laure, Rey, Amaury, Jiang, Wenzheng, Caramel, Julie, Genestier, Laurent, Marçais, Antoine, Marvel, Jacqueline, Ghavi-Helm, Yad, Walzer, Thierry

“…T cell development proceeds under the influence of a network of transcription factors (TFs). The precise role of Zeb1, a member of this network, remains…”
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Functional diversity of NLRP3 gain-of-function mutants associated with CAPS autoinflammation by Cosson, Camille, Riou, Romane, Patoli, Danish, Niu, Tingting, Rey, Amaury, Groslambert, Marine, De Rosny, Charlotte, Chatre, Elodie, Allatif, Omran, Henry, Thomas, Venet, Fabienne, Milhavet, Florian, Boursier, Guilaine, Belot, Alexandre, Jamilloux, Yvan, Merlin, Etienne, Duquesne, Agnès, Grateau, Gilles, Savey, Léa, Jacques Maria, Alexandre Thibault, Pagnier, Anne, Poutrel, Solène, Lambotte, Olivier, Mallebranche, Coralie, Ardois, Samuel, Richer, Olivier, Lemelle, Irène, Rieux-Laucat, Frédéric, Bader-Meunier, Brigitte, Amoura, Zahir, Melki, Isabelle, Cuisset, Laurence, Touitou, Isabelle, Geyer, Matthias, Georgin-Lavialle, Sophie, Py, Bénédicte F

“…NLRP3-associated autoinflammatory disease is a heterogenous group of monogenic conditions caused by NLRP3 gain-of-function mutations. The poor functional…”
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mTOR Activation Underlies Enhanced B Cell Proliferation and Autoimmunity in PrkcdG510S/G510S Mice by Moreews, Marion, Mathieu, Anne-Laure, Pouxvielh, Kevin, Reuschlé, Quentin, Drouillard, Annabelle, Dessay, Pénélope, Meignien, Marie, Zhang, Jiang, Fallone, Lucie, Rousseaux, Noëmi, Ainouze, Michelle, Rey, Amaury, Omarjee, Ommar, Decembre, Elodie, Lenief, Vanina, Djebali, Sophia, Thaunat, Olivier, Dreux, Marlène, Genestier, Laurent, Defrance, Thierry, Soulas-Sprauel, Pauline, Marçais, Antoine, Walzer, Thierry, Belot, Alexandre

“…Autosomal recessive PRKCD deficiency has previously been associated with the development of systemic lupus erythematosus in human patients, but the mechanisms…”
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Escherichia coli RhoGTPase-activating toxin CNF1 mediates NLRP3 inflammasome activation via p21 activated kinases-1/2 during bacteremia in mice by Dufies, Océane, Doye, Anne, Courjon, Johan, Torre, Cédric, Michel, Gregory, Loubatier, Celine, Jacquel, Arnaud, Chaintreuil, Paul, Majoor, Alissa, Guinamard, Rodolphe R., Gallerand, Alexandre, Saavedra, Pedro H. V., Verhoeyen, Els, Rey, Amaury, Marchetti, Sandrine, Ruimy, Raymond, Czerucka, Dorota, Lamkanfi, Mohamed, Py, Bénédicte F., Munro, Patrick, Visvikis, Orane, Boyer, Laurent

“…Inflammasomes are signaling platforms that are assembled in response to infection or sterile inflammation by cytosolic pattern recognition receptors (PRRs)…”
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