Search Results - "Reedich, Emily J."

Spinal motoneurons respond aberrantly to serotonin in a rabbit model of cerebral palsy by Reedich, Emily J, Genry, Landon T, Steele, Preston R, Mena Avila, Elvia, Dowaliby, Lisa, Drobyshevsky, Alexander, Manuel, Marin, Quinlan, Katharina A

“…Cerebral palsy (CP) is caused by a variety of factors that damage the developing central nervous system. Impaired motor control, including muscle stiffness and…”
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In Search of a Cure: The Development of Therapeutics to Alter the Progression of Spinal Muscular Atrophy by Ojala, Kristine S, Reedich, Emily J, DiDonato, Christine J, Meriney, Stephen D

“…Until the recent development of disease-modifying therapeutics, spinal muscular atrophy (SMA) was considered a devastating neuromuscular disease with a poor…”
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In vitro and in vivo effects of 2,4 diaminoquinazoline inhibitors of the decapping scavenger enzyme DcpS: Context-specific modulation of SMN transcript levels by Cherry, Jonathan J, DiDonato, Christine J, Androphy, Elliot J, Calo, Alessandro, Potter, Kyle, Custer, Sara K, Du, Sarah, Foley, Timothy L, Gopalsamy, Ariamala, Reedich, Emily J, Gordo, Susana M, Gordon, William, Hosea, Natalie, Jones, Lyn H, Krizay, Daniel K, LaRosa, Gregory, Li, Hongxia, Mathur, Sachin, Menard, Carol A, Patel, Paraj, Ramos-Zayas, Rebeca, Rietz, Anne, Rong, Haojing, Zhang, Baohong, Tones, Michael A

“…C5-substituted 2,4-diaminoquinazoline inhibitors of the decapping scavenger enzyme DcpS (DAQ-DcpSi) have been developed for the treatment of spinal muscular…”
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Inhibitory interneurons show early dysfunction in a SOD1 mouse model of amyotrophic lateral sclerosis by Cavarsan, Clarissa F., Steele, Preston R., Genry, Landon T., Reedich, Emily J., McCane, Lynn M., LaPre, Kay J., Puritz, Alyssa C., Manuel, Marin, Katenka, Natallia, Quinlan, Katharina A.

“…Few studies in amyotrophic lateral sclerosis (ALS) measure effects of the disease on inhibitory interneurons synapsing onto motoneurons (MNs). However,…”
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Spinal motor neuron loss occurs through a p53-and-p21-independent mechanism in the Smn2B/− mouse model of spinal muscular atrophy by Reedich, Emily J., Kalski, Martin, Armijo, Nicholas, Cox, Gregory A., DiDonato, Christine J.

“…Spinal muscular atrophy (SMA) is a pediatric neuromuscular disease caused by genetic deficiency of the survival motor neuron (SMN) protein. Pathological…”
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Enhanced nociceptive behavior and expansion of associated primary afferents in a rabbit model of cerebral palsy by Reedich, Emily J., Genry, Landon T., Singer, Meredith A., Cavarsan, Clarissa Fantin, Mena Avila, Elvia, Boudreau, Daphne M., Brennan, Michael C., Garrett, Alyssa M., Dowaliby, Lisa, Detloff, Megan R., Quinlan, Katharina A.

“…Spastic cerebral palsy (CP) is a movement disorder marked by hypertonia and hyperreflexia; the most prevalent comorbidity is pain. Since spinal nociceptive…”
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Spinal motor neuron loss occurs through a p53-and-p21-independent mechanism in the Smn mouse model of spinal muscular atrophy by Reedich, Emily J., Kalski, Martin, Armijo, Nicholas, Cox, Gregory A., DiDonato, Christine J.

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Spinal motor neuron loss occurs through a p53-and-p21-independent mechanism in the Smn 2B/- mouse model of spinal muscular atrophy by Reedich, Emily J, Kalski, Martin, Armijo, Nicholas, Cox, Gregory A, DiDonato, Christine J

“…Spinal muscular atrophy (SMA) is a pediatric neuromuscular disease caused by genetic deficiency of the survival motor neuron (SMN) protein. Pathological…”
Get full text