Search Results - "Rauch, Katharina S"

Eomesodermin Expression in CD4+ T Cells Restricts Peripheral Foxp3 Induction by Lupar, Ekaterina, Brack, Maria, Garnier, Laure, Laffont, Sophie, Rauch, Katharina S, Schachtrup, Kristina, Arnold, Sebastian J, Guéry, Jean-Charles, Izcue, Ana

“…CD4(+) T cells polarize into effector Th subsets characterized by signature transcription factors and cytokines. Although T-bet drives Th1 responses and…”
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Id3 Controls Cell Death of 2B4+ Virus-Specific CD8+ T Cells in Chronic Viral Infection by Menner, Alexandra J, Rauch, Katharina S, Aichele, Peter, Pircher, Hanspeter, Schachtrup, Christian, Schachtrup, Kristina

“…Sustained Ag persistence in chronic infection results in a deregulated CD8(+) T cell response that is characterized by T cell exhaustion and cell death of…”
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The balance of Id3 and E47 determines neural stem/precursor cell differentiation into astrocytes by Bohrer, Christian, Pfurr, Sabrina, Mammadzada, Könül, Schildge, Sebastian, Plappert, Leandra, Hils, Miriam, Pous, Lauriane, Rauch, Katharina S, Dumit, Verónica I, Pfeifer, Dietmar, Dengjel, Jörn, Kirsch, Matthias, Schachtrup, Kristina, Schachtrup, Christian

“…Adult neural stem/precursor cells (NSPCs) of the subventricular zone (SVZ) are an endogenous source for neuronal replacement in CNS disease. However, adult…”
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Caveolin-1 regulates TCR signal strength and regulatory T-cell differentiation into alloreactive T cells by Schönle, Anne, Hartl, Frederike A., Mentzel, Jan, Nöltner, Theresa, Rauch, Katharina S., Prestipino, Alessandro, Wohlfeil, Sebastian A., Apostolova, Petya, Hechinger, Anne-Kathrin, Melchinger, Wolfgang, Fehrenbach, Kerstin, Guadamillas, Marta C., Follo, Marie, Prinz, Gabriele, Ruess, Ann-Katrin, Pfeifer, Dietmar, Angel del Pozo, Miguel, Schmitt-Graeff, Annette, Duyster, Justus, Hippen, Keli I., Blazar, Bruce R., Schachtrup, Kristina, Minguet, Susana, Zeiser, Robert

“…Caveolin-1 (Cav-1) is a key organizer of membrane specializations and a scaffold protein that regulates signaling in multiple cell types. We found increased…”
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Id3 Maintains Foxp3 Expression in Regulatory T Cells by Controlling a Transcriptional Network of E47, Spi-B, and SOCS3 by Rauch, Katharina S., Hils, Miriam, Lupar, Ekaterina, Minguet, Susana, Sigvardsson, Mikael, Rottenberg, Martin E., Izcue, Ana, Schachtrup, Christian, Schachtrup, Kristina

“…The transcription factor Foxp3 dominantly controls regulatory T (Treg) cell function, and only its continuous expression guarantees the maintenance of full…”
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Regulatory T cells characterized by low Id3 expression are highly suppressive and accumulate during chronic infection by Rauch, Katharina S, Hils, Miriam, Menner, Alexandra J, Sigvardsson, Mikael, Minguet, Susana, Aichele, Peter, Schachtrup, Christian, Schachtrup, Kristina

“…Foxp3 regulatory T (Treg) cells are broadly divided into naive-like and activated Treg cells, however recent studies suggest further Treg cell heterogeneity…”
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NO is a macrophage autonomous modifier of the cytokine response to streptococcal single-stranded RNA by Deshmukh, Sachin D, Müller, Sabrina, Hese, Katrin, Rauch, Katharina S, Wennekamp, Julia, Takeuchi, Osamu, Akira, Shizuo, Golenbock, Douglas T, Henneke, Philipp

“…Group B streptococci, a major cause of sepsis, induce inflammatory cytokines in strict dependence on bacterial ssRNA and the host molecules MyD88 and UNC-93B…”
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Id3 Maintains Foxp3 Expression in Regulatory T Cells by Controlling a TranscriptionalNetwork of E47, Spi-B, and SOCS3 by Rauch, Katharina S., Hils, Miriam, Lupar, Ekaterina, Minguet, Susana, Sigvardsson, Mikael, Rottenberg, Martin E., Izcue, Ana, Schachtrup, Christian, Schachtrup, Kristina

“…The transcription factor Foxp3 dominantly controls regulatory T (Treg) cell function, and only its continuous expression guarantees the maintenance of full…”
Get full text

NO is a macrophage autonomous modifier of the cytokine response to streptococcal ssRNA by Deshmukh, Sachin D., Müller, Sabrina, Hese, Katrin, Rauch, Katharina S., Wennekamp, Julia, Takeuchi, Osamu, Akira, Shizuo, Golenbock, Douglas T., Henneke, Philipp

“…Group B streptococcus (GBS), a major cause of sepsis, induces inflammatory cytokines in strict dependence of bacterial ssRNA and the host molecules MyD88 und…”
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