Search Results - "Ranu, Hardeep K"
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The effect of Gi‐protein inactivation on basal, and β1‐ and β2AR‐stimulated contraction of myocytes from transgenic mice overexpressing the β2‐adrenoceptor
Published in British journal of pharmacology (01-10-2000)“…The atria and ventricles of transgenic mice (TGβ2) with cardiac overexpression of the human β2‐adrenoceptor (β2AR) were initially reported to show maximum…”
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2
SERCA2a Overexpression Decreases the Incidence of Aftercontractions in Adult Rabbit Ventricular Myocytes
Published in Journal of molecular and cellular cardiology (01-05-2001)“…K. Davia, E. Bernobich, H. K. Ranu, F. del Monte, C. M. N. Terracciano, K. T. MacLeod, D. L. Adamson, B. Chaudhri, R. J. Hajjar and S. E. Harding. SERCA2a…”
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3
Effects of Na(+)/Ca(2+)-exchanger overexpression on excitation-contraction coupling in adult rabbit ventricular myocytes
Published in Journal of molecular and cellular cardiology (01-04-2002)“…The Na(+)/Ca(2+)-exchanger (NCX) is the main mechanism by which Ca(2+) is transported out of the ventricular myocyte. NCX levels are raised in failing human…”
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4
Functional alterations in adult rat myocytes after overexpression of phospholamban with use of adenovirus
Published in Physiological genomics (31-08-1999)“…1 Cardiac Medicine, National Heart and Lung Institute, Imperial College School of Medicine, London SW3 6LY, United Kingdom 2 Cardiovascular Research Center,…”
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5
The effect of Gi‐protein inactivation on basal, and β 1 ‐ and β 2 AR‐stimulated contraction of myocytes from transgenic mice overexpressing the β 2 ‐adrenoceptor
Published in British journal of pharmacology (29-01-2009)“…The atria and ventricles of transgenic mice (TGβ 2 ) with cardiac overexpression of the human β 2 ‐adrenoceptor (β 2 AR) were initially reported to show…”
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6
Effects of Na+/Ca2+-exchanger Overexpression on Excitation–contraction Coupling in Adult Rabbit Ventricular Myocytes
Published in Journal of molecular and cellular cardiology (01-04-2002)“…The Na+/Ca2+-exchanger (NCX) is the main mechanism by which Ca2+ is transported out of the ventricular myocyte. NCX levels are raised in failing human heart,…”
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7
The effect of Gi-protein inactivation on basal, and [beta]1- and [beta]2AR-stimulated contraction of myocytes from transgenic mice overexpressing the [beta]2-adrenoceptor
Published in British journal of pharmacology (01-10-2000)“…The atria and ventricles of transgenic mice (TGbeta(2)) with cardiac overexpression of the human beta(2)-adrenoceptor (beta(2)AR) were initially reported to…”
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