Search Results - "RIVARD, N"

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  1. 1

    Oncogenic KRAS signalling promotes the Wnt/β-catenin pathway through LRP6 in colorectal cancer by Lemieux, E, Cagnol, S, Beaudry, K, Carrier, J, Rivard, N

    Published in Oncogene (17-09-2015)
    “…Aberrant regulation of the Wnt/β-catenin signaling pathway is one of the major causes of colorectal cancer (CRC). Loss-of-function mutations in APC are…”
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    Journal Article
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    Oncogenic KRAS and BRAF activation of the MEK/ERK signaling pathway promotes expression of dual-specificity phosphatase 4 (DUSP4/MKP2) resulting in nuclear ERK1/2 inhibition by Cagnol, S, Rivard, N

    Published in Oncogene (31-01-2013)
    “…Gain-of-function mutations in KRAS and BRAF genes are found in up to 50% of colorectal cancers. These mutations result in the activation of the BRAF/MEK…”
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    Abstract # 1850 Early life stress predisposes to increased sensitivity to inflammatory stimuli through intestinal dysbiosis by Palma, G.De, Pigrau, M, Cocciolillo, S, Langlois, M.J, Lu, J, Deng, Y, Rivard, N, Verdu, E.F, Collins, S.M, Bercik, P

    Published in Brain, behavior, and immunity (01-10-2016)
    “…Stress, anxiety and depression are considered risk factors for Inflammatory Bowel Disease. We have previously shown in mice that early life stress induced by…”
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    A54 SHP2 FUNCTION IN NORMAL AND APC -MUTANT INTESTINAL EPITHELIAL CELLS by Côté-Biron, A, Gagné-Sansfaçon, J, Rivard, N

    “…Abstract Background Somatic SHP2 gain-of-function mutations were found in some colorectal tumours. However, its function in colorectal epithelium remains to be…”
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    A53 MOLECULAR MECHANISMS FOR A ROLE OF SHP-2 IN TLR4 SIGNALING IN INTESTINAL EPITHELIAL CELLS by Chabot, V, Lévesque, D, Boisvert, F, Rivard, N

    “…Abstract Background Shp-2 gene polymorphisms are associated with greater susceptibility to ulcerative colitis. How Shp-2 contributes to disease susceptibility…”
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    Oncogenic KRAS signalling promotes the Wnt/[beta]-catenin pathway through LRP6 in colorectal cancer by Lemieux, E, Cagnol, S, Beaudry, K, Carrier, J, Rivard, N

    Published in Oncogene (17-09-2015)
    “…Aberrant regulation of the Wnt/β-catenin signaling pathway is one of the major causes of colorectal cancer (CRC). Loss-of-function mutations in APC are…”
    Get full text
    Journal Article
  8. 8

    Pancreas growth, tyrosine kinase, PtdIns 3-kinase, and PLD involve high-affinity CCK-receptor occupation by Rivard, N, Rydzewska, G, Lods, J S, Martinez, J, Morisset, J

    Published in The American journal of physiology (01-01-1994)
    “…Cholecystokinin (CCK), CCK octapeptide (CCK-8), and caerulein (Cae) are the most potent trophic factors in the pancreas when given exogenously or released from…”
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    Epidermal growth factor and transforming growth factor α down-regulate human gastric lipase gene expression by Tremblay, Eric, Basque, Jean René, Rivard, Nathalie, Ménard, Daniel

    Published in Gastroenterology (New York, N.Y. 1943) (01-04-1999)
    “…Background & Aims: It was recently reported that human gastric lipase (HGL) activity is modulated by epidermal growth factor (EGF). The aims of this study were…”
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    Novel model of integration of signaling pathways in rat pancreatic acinar cells by Rivard, N, Rydzewska, G, Lods, J S, Morisset, J

    Published in The American journal of physiology (01-09-1995)
    “…Cholecystokinin (CCK) is the major pancreatic secretagogue and acinar cell mitogen. This study was performed to determine by which effector systems CCK…”
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    Dynamics of pancreatic tyrosine kinase and phospholipase D activities in the course of cerulein-induced acute pancreatitis and during regeneration by RYDZEWSKA, G, RIVARD, N, MORISSET, J

    Published in Pancreas (01-05-1995)
    “…Acute pancreatitis can be induced in the rat by high doses of cerulein, a cholecystokinin analogue. Regeneration of the pancreatic gland after this aggression…”
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  13. 13

    IncA/C Conjugative Plasmids Mobilize a New Family of Multidrug Resistance Islands in Clinical Vibrio cholerae Non-01/Non-0139 Isolates from Haiti by Carraro, N, Rivard, N, Ceccarelli, Daniela, Colwell, R.R, Burrus, V

    Published in mBio (2016)
    “…Mobile genetic elements play a pivotal role in the adaptation of bacterial populations, allowing them to rapidly cope with hostile conditions, including the…”
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    Regulation of pancreatic tyrosine kinase and phosphatase activities by cholecystokinin and somatostatin by Rivard, N, Lebel, D, Lainé, J, Morisset, J

    Published in The American journal of physiology (01-06-1994)
    “…Phosphorylation and dephosphorylation of proteins on tyrosyl residues are important reactions involved in cellular activities, namely, those associated with…”
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    A208 SHP-2 PHOSPHATASE PREVENTS SENESCENCE IN NORMAL AND TUMOR INTESTINAL EPITHELIAL CELLS by Vaillancourt-Lavigueur, V, Sansfaçon, J Gagné, Langlois, M, Rivard, N

    “…Abstract Background The protein tyrosine phosphatase SHP-2 is known to regulate many cellular functions including growth, differentiation and survival…”
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    A265 INVOLVEMENT OF LRP6 IN INTESTINAL HOMEOSTASIS AND INFLAMMATION by Raisch, J, Langlois, M, Lemieux, E, Rivard, N

    “…Abstract Background Self-renewal of the intestinal epithelium is tightly regulated by interacting intracellular signaling pathways, which control stem cell…”
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    Endogenous cholecystokinin release responsible for pancreatic growth observed after pancreatic juice diversion by Rivard, N, Guan, D, Maouyo, D, Grondin, G, Bérubé, F L, Morisset, J

    Published in Endocrinology (Philadelphia) (01-12-1991)
    “…This study was undertaken to determine whether intermittent pancreatic juice diversion (PJD) from the intestine can induce pancreatic and duodenal growth…”
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    Human Homolog of Disc-large Is Required for Adherens Junction Assembly and Differentiation of Human Intestinal Epithelial Cells by Laprise, Patrick, Viel, Alain, Rivard, Nathalie

    Published in The Journal of biological chemistry (12-03-2004)
    “…We and others have shown that phosphatidylinositol 3-kinase (PI3K) is recruited to and activated by E-cadherin engagement. This PI3K activation is essential…”
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