Search Results - "Récalde, Alice"
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Transient receptor potential channels in cardiac health and disease
Published in Nature reviews cardiology (01-06-2019)“…Transient receptor potential (TRP) channels are nonselective cationic channels that are generally Ca 2+ permeable and have a heterogeneous expression in the…”
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Microparticles From Ischemic Muscle Promotes Postnatal Vasculogenesis
Published in Circulation (New York, N.Y.) (02-06-2009)“…We hypothesized that microparticles (MPs) released after ischemia are endogenous signals leading to postischemic vasculogenesis. MPs from mice ischemic…”
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Découverte du rôle des facteurs mécaniques dans l'athérogenèse
Published in Sang, thrombose, vaisseaux : STV (01-04-2010)Get full text
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The Chemokine Decoy Receptor D6 Prevents Excessive Inflammation and Adverse Ventricular Remodeling After Myocardial Infarction
Published in Arteriosclerosis, thrombosis, and vascular biology (01-09-2012)“…OBJECTIVE—Leukocyte infiltration in ischemic areas is a hallmark of myocardial infarction, and overwhelming infiltration of innate immune cells has been shown…”
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Homeostatic and Tissue Reparation Defaults in Mice Carrying Selective Genetic Invalidation of CXCL12/Proteoglycan Interactions
Published in Circulation (New York, N.Y.) (09-10-2012)“…Interaction with heparan sulfate proteoglycans is supposed to provide chemokines with the capacity to immobilize on cell surface and extracellular matrix for…”
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Regional Differences in Ca2+ Signaling and Transverse-Tubules across Left Atrium from Adult Sheep
Published in International journal of molecular sciences (25-01-2023)“…Cardiac excitation-contraction coupling can be different between regions of the heart. Little is known at the atria level, specifically in different regions of…”
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Regulatory T Cells Modulate Postischemic Neovascularization
Published in Circulation (New York, N.Y.) (06-10-2009)“…CD4+ and CD8+ T lymphocytes are key regulators of postischemic neovascularization. T-cell activation is promoted by 2 major costimulatory signalings, the…”
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Inhibition of EPAC1 signaling pathway alters atrial electrophysiology and prevents atrial fibrillation
Published in Frontiers in physiology (22-02-2023)“…Atrial fibrillation (AF) is the most common sustained cardiac arrhythmia and is associated with increased mortality and morbidity. The Exchange Protein…”
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PO-01-171 ROLE OF THE EXCHANGE PROTEIN ACTIVATED BY CAMP (EPAC) IN ATRIAL ELECTROPHYSIOLOGY AND ATRIAL FIBRILLATION MECHANISMS
Published in Heart rhythm (01-05-2023)Get full text
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Functional Epicardial Conduction Disturbances Due to a SCN5A Variant Associated With Brugada Syndrome
Published in JACC. Clinical electrophysiology (01-08-2023)“…Brugada syndrome is a significant cause of sudden cardiac death (SCD), but the underlying mechanisms remain hypothetical. This study aimed to elucidate this…”
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C/EBP Homologous Protein-10 (CHOP-10) Limits Postnatal Neovascularization Through Control of Endothelial Nitric Oxide Synthase Gene Expression
Published in Circulation (New York, N.Y.) (28-02-2012)“…C/EBP homologous protein-10 (CHOP-10) is a novel developmentally regulated nuclear protein that emerges as a critical transcriptional integrator among pathways…”
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Sympathetic Nervous System Regulates Bone Marrow–Derived Cell Egress Through Endothelial Nitric Oxide Synthase Activation: Role in Postischemic Tissue Remodeling
Published in Arteriosclerosis, thrombosis, and vascular biology (01-03-2012)“…OBJECTIVE—Catecholamines have been shown to control bone marrow (BM)–derived cell egress, yet the cellular and molecular mechanisms involved in this effect and…”
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Regulation of monocyte subset systemic levels by distinct chemokine receptors controls post-ischaemic neovascularization
Published in Cardiovascular research (01-10-2010)“…Aims Monocyte systemic levels are known to be a major determinant of ischaemic tissue revascularization, but the mechanisms mediating mobilization of different…”
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Inhibition of Prolyl Hydroxylase Domain Proteins Promotes Therapeutic Revascularization
Published in Circulation (New York, N.Y.) (07-07-2009)“…The hypoxia-inducible transcription factor (HIF) subunits are destabilized via the O(2)-dependent prolyl hydroxylase domain proteins (PHD1, PHD2, and PHD3). We…”
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Endothelial Nitric Oxide Synthase Overexpression Restores the Efficiency of Bone Marrow Mononuclear Cell-Based Therapy
Published in The American journal of pathology (2011)“…Bone marrow-derived mononuclear cells (BMMNCs) enhance postischemic neovascularization, and their therapeutic use is currently under clinical investigation…”
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Tetrahydrobiopterin Protects Against Hypertrophic Heart Disease Independent of Myocardial Nitric Oxide Synthase Coupling
Published in Journal of the American Heart Association (21-03-2016)“…Background Nitric oxide synthase uncoupling occurs under conditions of oxidative stress modifying the enzyme's function so it generates superoxide rather than…”
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Abstract 536: Prolyl-hydroxylase Inhibition as a Strategy of Therapeutic Angiogenesis
Published in Arteriosclerosis, thrombosis, and vascular biology (01-05-2012)“…Abstract only Therapeutic neovascularization represents an alternative treatment modality for patients with advanced ischemic coronary disease or critical limb…”
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Mutual Regulation of Epicardial Adipose Tissue and Myocardial Redox State by PPAR-γ/Adiponectin Signalling
Published in Circulation research (04-03-2016)“…RATIONALE:Adiponectin has anti-inflammatory effects in experimental models, but its role in the regulation of myocardial redox state in humans is unknown…”
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Inducibility, but not stability, of atrial fibrillation is increased by NOX2 overexpression in mice
Published in Cardiovascular research (28-09-2021)“…Abstract Aims Gp91-containing NADPH oxidases (NOX2) are a significant source of myocardial superoxide production. An increase in NOX2 activity accompanies…”
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