Search Results - "Pribis, John P"

A checklist is associated with increased quality of reporting preclinical biomedical research: A systematic review by Han, SeungHye, Olonisakin, Tolani F, Pribis, John P, Zupetic, Jill, Yoon, Joo Heung, Holleran, Kyle M, Jeong, Kwonho, Shaikh, Nader, Rubio, Doris M, Lee, Janet S

“…Irreproducibility of preclinical biomedical research has gained recent attention. It is suggested that requiring authors to complete a checklist at the time of…”
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MD-2 is required for disulfide HMGB1-dependent TLR4 signaling by Yang, Huan, Wang, Haichao, Ju, Zhongliang, Ragab, Ahmed A, Lundbäck, Peter, Long, Wei, Valdes-Ferrer, Sergio I, He, Mingzhu, Pribis, John P, Li, Jianhua, Lu, Ben, Gero, Domokos, Szabo, Csaba, Antoine, Daniel J, Harris, Helena E, Golenbock, Doug T, Meng, Jianmin, Roth, Jesse, Chavan, Sangeeta S, Andersson, Ulf, Billiar, Timothy R, Tracey, Kevin J, Al-Abed, Yousef

“…Innate immune receptors for pathogen- and damage-associated molecular patterns (PAMPs and DAMPs) orchestrate inflammatory responses to infection and injury…”
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Stress-Induced Mutagenesis, Gambler Cells, and Stealth Targeting Antibiotic-Induced Evolution by Pribis, John P, Zhai, Yin, Hastings, P J, Rosenberg, Susan M

“…Mechanisms of evolution and evolution of antibiotic resistance are both fundamental and world health problems. Stress-induced mutagenesis defines mechanisms of…”
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Signaling of high mobility group box 1 (HMGB1) through toll-like receptor 4 in macrophages requires CD14 by Kim, Sodam, Kim, Sun Young, Pribis, John P, Lotze, Michael, Mollen, Kevin P, Shapiro, Richard, Loughran, Patricia, Scott, Melanie J, Billiar, Timothy R

“…High mobility group box 1 (HMGB1) is a DNA-binding protein that possesses cytokinelike, proinflammatory properties when released extracellularly in the C23-C45…”
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Bacteria-to-Human Protein Networks Reveal Origins of Endogenous DNA Damage by Xia, Jun, Chiu, Li-Ya, Nehring, Ralf B., Bravo Núñez, María Angélica, Mei, Qian, Perez, Mercedes, Zhai, Yin, Fitzgerald, Devon M., Pribis, John P., Wang, Yumeng, Hu, Chenyue W., Powell, Reid T., LaBonte, Sandra A., Jalali, Ali, Matadamas Guzmán, Meztli L., Lentzsch, Alfred M., Szafran, Adam T., Joshi, Mohan C., Richters, Megan, Gibson, Janet L., Frisch, Ryan L., Hastings, P.J., Bates, David, Queitsch, Christine, Hilsenbeck, Susan G., Coarfa, Cristian, Hu, James C., Siegele, Deborah A., Scott, Kenneth L., Liang, Han, Mancini, Michael A., Herman, Christophe, Miller, Kyle M., Rosenberg, Susan M.

“…DNA damage provokes mutations and cancer and results from external carcinogens or endogenous cellular processes. However, the intrinsic instigators of…”
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HMGB1-Driven Inflammation and Intimal Hyperplasia After Arterial Injury Involves Cell-Specific Actions Mediated by TLR4 by Cai, Jingjing, Yuan, Hong, Wang, Qingde, Yang, Huan, Al-Abed, Yousef, Hua, Zhong, Wang, Jiemei, Chen, Dandan, Wu, Jinze, Lu, Ben, Pribis, John P, Jiang, Weihong, Yang, Kan, Hackam, David J, Tracey, Kevin J, Billiar, Timothy R, Chen, Alex F

“…OBJECTIVE—Endoluminal vascular interventions such as angioplasty initiate a sterile inflammatory response resulting from local tissue damage. This response…”
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Identification of pharmacological modulators of HMGB1-induced inflammatory response by cell-based screening by Gerö, Domokos, Szoleczky, Petra, Módis, Katalin, Pribis, John P, Al-Abed, Yousef, Yang, Huan, Chevan, Sangeeta, Billiar, Timothy R, Tracey, Kevin J, Szabo, Csaba

“…High mobility group box 1 (HMGB1), a highly conserved, ubiquitous protein, is released into the circulation during sterile inflammation (e.g. arthritis,…”
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Oral intake of hydrogen-rich water inhibits intimal hyperplasia in arterialized vein grafts in rats by QIANG SUN, KAWAMURA, Tomohiro, TOYODA, Yoshiya, NAKAO, Atsunori, MASUTANI, Kosuke, XIMEI PENG, QING SUN, STOLZ, Donna B, PRIBIS, John P, BILLIAR, Timothy R, XUEJUN SUN, BERMUDEZ, Christian A

“…Arterialized vein grafts often fail due to intimal hyperplasia. Hydrogen potently protects organs and cells from many insults via its anti-inflammatory and…”
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Two mechanisms of chromosome fragility at replication-termination sites in bacteria by Mei, Qian, Fitzgerald, Devon M, Liu, Jingjing, Xia, Jun, Pribis, John P, Zhai, Yin, Nehring, Ralf B, Paiano, Jacob, Li, Heyuan, Nussenzweig, Andre, Hastings, P J, Rosenberg, Susan M

“…Chromosomal fragile sites are implicated in promoting genome instability, which drives cancers and neurological diseases. Yet, the causes and mechanisms of…”
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Gamblers: An Antibiotic-Induced Evolvable Cell Subpopulation Differentiated by Reactive-Oxygen-Induced General Stress Response by Pribis, John P., García-Villada, Libertad, Zhai, Yin, Lewin-Epstein, Ohad, Wang, Anthony Z., Liu, Jingjing, Xia, Jun, Mei, Qian, Fitzgerald, Devon M., Bos, Julia, Austin, Robert H., Herman, Christophe, Bates, David, Hadany, Lilach, Hastings, P.J., Rosenberg, Susan M.

“…Antibiotics can induce mutations that cause antibiotic resistance. Yet, despite their importance, mechanisms of antibiotic-promoted mutagenesis remain elusive…”
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ppGpp and RNA-polymerase backtracking guide antibiotic-induced mutable gambler cells by Zhai, Yin, Minnick, P.J., Pribis, John P., Garcia-Villada, Libertad, Hastings, P.J., Herman, Christophe, Rosenberg, Susan M.

“…Antibiotic resistance is a global health threat and often results from new mutations. Antibiotics can induce mutations via mechanisms activated by stress…”
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The Central Role of Arginine Catabolism in T-Cell Dysfunction and Increased Susceptibility to Infection After Physical Injury by Zhu, Xinmei, Pribis, John P, Rodriguez, Paulo C, Morris, Sidney M, Vodovotz, Yoram, Billiar, Timothy R, Ochoa, Juan B

“…OBJECTIVE:To explore the hypothesis that decreased arginine availability by myeloid-derived suppressor cells (MDSCs) is a cause of T-cell dysfunction after…”
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Epsilon aminocaproic acid reduces blood transfusion and improves the coagulation test after pediatric open-heart surgery: a meta-analysis of 5 clinical trials by Lu, Jun, Meng, Haoyu, Meng, Zhaoyi, Sun, Ying, Pribis, John P, Zhu, Chunyan, Li, Quan

“…Excessive postoperative blood loss after cardiopulmonary bypass is a common problem, especially in patients suffering from congenital heart diseases. The…”
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Drugging evolution of antibiotic resistance at a regulatory network hub by Zhai, Yin, Pribis, John P, Dooling, Sean W, Garcia-Villada, Libertad, Minnick, P J, Xia, Jun, Liu, Jingjing, Mei, Qian, Fitzgerald, Devon M, Herman, Christophe, Hastings, P J, Costa-Mattioli, Mauro, Rosenberg, Susan M

“…Evolution of antibiotic resistance is a world health crisis, fueled by new mutations. Drugs to slow mutagenesis could, as cotherapies, prolong the shelf-life…”
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Systemic Arginine Depletion After a Murine Model of Surgery or Trauma by Pribis, John P., Zhu, Xinmei, Vodovotz, Yoram, Ochoa, Juan B.

“…Background: Arginine metabolism and availability after surgery or trauma (ST) is an important modulator of immune responses. Arginine levels are significantly…”
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The HIV Protease Inhibitor Saquinavir Inhibits HMGB1-Driven Inflammation by Targeting the Interaction of Cathepsin V with TLR4/MyD88 by Pribis, John P, Al-Abed, Yousef, Yang, Huan, Gero, Domokos, Xu, Hongbo, Montenegro, Marcelo F, Bauer, Eileen M, Kim, Sodam, Chavan, Sangeeta S, Cai, Changchun, Li, Tunliang, Szoleczky, Petra, Szabo, Csaba, Tracey, Kevin J, Billiar, Timothy R

“…Extracellular high-mobility group box 1 (HMGB1) (disulfide form), via activation of toll-like receptor 4 (TLR4)-dependent signaling, is a strong driver of…”
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Holliday junction trap shows how cells use recombination and a junction-guardian role of RecQ helicase by Xia, Jun, Chen, Li-Tzu, Mei, Qian, Ma, Chien-Hui, Halliday, Jennifer A, Lin, Hsin-Yu, Magnan, David, Pribis, John P, Fitzgerald, Devon M, Hamilton, Holly M, Richters, Megan, Nehring, Ralf B, Shen, Xi, Li, Lei, Bates, David, Hastings, P J, Herman, Christophe, Jayaram, Makkuni, Rosenberg, Susan M

“…DNA repair by homologous recombination (HR) underpins cell survival and fuels genome instability, cancer, and evolution. However, the main kinds and sources of…”
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The HIV Protease Inhibitor Saquinavir Inhibits HMGBl-Driven Inflammation by Targeting the Interaction of Cathepsin V with TLR4/MyD88 by Pribis, John P., Al-Abed, Yousef, Yang, Huan, Gero, Domokos, Xu, Hongbo, Montenegro, Marcelo F., Bauer, Eileen M., Kim, Sodam, Chavan, Sangeeta S., Cai, Changchun, Li, Tunliang, Szoleczky, Petra, Szabo, Csaba, Tracey, Kevin J., Billiar, Timothy R.

“…Extracellular high-mobility group box 1 (HMGB1) (disulfide form), via activation of toll-like receptor 4 (TLR4)-dependent signaling, is a strong driver of…”
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Nature of Myeloid Cells Expressing Arginase 1 in Peripheral Blood After Trauma by Bryk, Jodie A., Popovic, Petar J., Zenati, Mazen S., Munera, Veronica, Pribis, John P., Ochoa, Juan B.

“…BACKGROUND:Myeloid cells that express arginase 1 are upregulated by different stimuli, including trauma, and are capable of depleting arginine from the…”
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