Search Results - "Prado, R Q"

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  1. 1
    Improvement of the resistance against early Mycobacterium tuberculosis-infection in the absence of PI3Kγ enzyme is associated with increase of CD4+IL-17+ cells and neutrophils

    Improvement of the resistance against early Mycobacterium tuberculosis-infection in the absence of PI3Kγ enzyme is associated with increase of CD4+IL-17+ cells and neutrophils by Cavalcanti-Neto, M.P., Prado, R.Q., Piñeros, A.R., Sérgio, C.A., Bertolini, T.B., Gembre, A.F., Ramos, S.G., Bonato, V.L.

    Published in Tuberculosis (Edinburgh, Scotland) (01-12-2018)
    “…Given the impossibility to study the lung immune response during Mycobacterium tuberculosis-latent infection, and consequently, the mechanisms that control the…”
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  2. 2
    M2 macrophages or IL-33 treatment attenuate ongoing Mycobacterium tuberculosis infection

    M2 macrophages or IL-33 treatment attenuate ongoing Mycobacterium tuberculosis infection by Piñeros, A. R., Campos, L. W., Fonseca, D. M., Bertolini, T. B., Gembre, A. F., Prado, R. Q., Alves-Filho, J. C., Ramos, S. G., Russo, M., Bonato, V. L. D.

    Published in Scientific reports (27-01-2017)
    “…The protective effects of mycobacterial infections on lung allergy are well documented. However, the inverse relationship between tuberculosis and type 2…”
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  3. 3
    Attenuation of experimental asthma by mycobacterial protein combined with CpG requires a TLR9-dependent IFN-[gamma]-CCR2 signalling circuit

    Attenuation of experimental asthma by mycobacterial protein combined with CpG requires a TLR9-dependent IFN-[gamma]-CCR2 signalling circuit by Prado, R Q, Bertolini, T B, Pineros, A R, Gembre, A F, Ramos, S G, Silva, C L, Borges, M C, Bonato, V L D

    Published in Clinical and experimental allergy (01-09-2015)
    “…Summary Background Allergic asthma is a chronic pulmonary disease characterized by a Th2 inflammatory response. The modulation of a Th2 immune response based…”
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  4. 4
    Attenuation of experimental asthma by mycobacterial protein combined with CpG requires a TLR9-dependent IFN-γ-CCR2 signalling circuit

    Attenuation of experimental asthma by mycobacterial protein combined with CpG requires a TLR9-dependent IFN-γ-CCR2 signalling circuit by Prado, R. Q., Bertolini, T. B., Piñeros, A. R., Gembre, A. F., Ramos, S. G., Silva, C. L., Borges, M. C., Bonato, V. L. D.

    Published in Clinical and experimental allergy (01-09-2015)
    “…Summary Background Allergic asthma is a chronic pulmonary disease characterized by a Th2 inflammatory response. The modulation of a Th2 immune response based…”
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  5. 5
    Attenuation of experimental asthma by mycobacterial protein combined with CpG requires a TLR 9‐dependent IFN ‐γ‐ CCR 2 signalling circuit

    Attenuation of experimental asthma by mycobacterial protein combined with CpG requires a TLR 9‐dependent IFN ‐γ‐ CCR 2 signalling circuit by Prado, R. Q., Bertolini, T. B., Piñeros, A. R., Gembre, A. F., Ramos, S. G., Silva, C. L., Borges, M. C., Bonato, V. L. D.

    Published in Clinical and experimental allergy (01-09-2015)
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