Search Results - "Potapova, G I"

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    Genetic Defects as Tumor Markers by Lichtenstein, A V, Potapova, G I

    Published in Molecular biology (New York) (01-03-2003)
    “…Carcinogenesis is long-term multistep accumulation of defects of genes responsible for cell division, DNA repair, and apoptosis. The functions of these genes…”
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    Detection of mutant k-ras sequences in the urine of cancer patients by Serdyuk, O I, Botezatu, I V, Shelepov, V P, Potapova, G I, Alekhina, R P, Molyaka, Y K, Anan'ev, V S, Knysh, V I, Melkonyan, O S, Umanskii, S R, Lichtenshtein, A V

    “…DNA fragments from apoptotic cells crossing the renal barrier retain their matrix functions, which allows PCR identification of mutant sequences in excreted…”
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    Some biochemical mechanisms underlying the impairment of T and B cell immunity in C3HA mice during hepatoma growth by Potapova, G I, Khramtsova, S N, Dmitrieva, L V, Shapot, V S

    Published in Neoplasma (1987)
    “…In thymocytes of C3HA mice carrying the transplantable and ortoaminoazotoluene induced hepatomas at the time of their intense growth a drastic decrease in…”
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    Alterations of (CA)n DNA repeats and tumor suppressor genes in human gastric cancer by MIRONOV, N. M, AGUELON, M. A.-M, PATAPOVA, G. I, OMORI, Y, GORBUNOV, O. V, KLIMENKOV, A. A, YAMASAKI, H

    Published in Cancer research (Chicago, Ill.) (1994)
    “…We have examined whether alterations of simple (CA)n DNA repeats, as observed in human colon cancers, occur during human gastric carcinogenesis and whether…”
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    Genetic defects as markers of tumor development by Likhtenshteĭn, A V, Potapova, G I

    Published in Molekuliarnaia biologiia (01-03-2003)
    “…Carcinogenesis is long-term multistep accumulation of defects of genes responsible for cell division, DNA repair, and apoptosis. The functions of these genes…”
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    Combination of immunodepression and disorders in nucleic acid metabolism of lymphoid tissue as a manifestation of a paraneoplastic syndrome by Potapova, G I, Shapot, V S

    Published in Arkhiv patologii (1987)
    “…Several physiological, biochemical, and molecular biological approaches to the study of factors determining immunodepression in tumor-bearing animals are…”
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    Opposite effect of p53 on nucleotide metabolizing enzyme activity in Rat1 cells and their sublines, transformed by N-RAS or v-mos oncogenes by Khramtsova, S N, Osovskaia, V S, Semeniak, O Iu, Potapova, G I, Chumakov, P M, Kopnin, B P

    Published in Biokhimiia (Moscow, Russia) (01-11-1995)
    “…The effects of exogenous human p53 and its various mutants (Ala-141, His-175, His-194, Trp-248, His-273) on two key enzymes of purine uptake, adenosine…”
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    Biometric characteristics of renal corpuscles in reduced adrenal function and individually dosed physical loading by Potapova, I G

    Published in Arkhiv anatomii, gistologii i embriologii (01-04-1981)
    “…In 19 mongrel dogs (males) the effect of various doses of physical loading on the nephric body structure was studied at a decreased content of glucocorticoids…”
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    Change in activity of enzymes for purine metabolism and RNA and DNA biosynthesis in macrophages, reflecting impairment of their functions in neoplastic growth by Khramtsova, S N, Raĭnkhol'd, D, Sukhova, T I, Potapova, G I, Shapot, V S

    Published in Biokhimiia (Moscow, Russia) (01-10-1989)
    “…The changes in the biochemical parameters of peritoneal macrophages and their coupling to the secretory and phagocytic functions in CH3A mice during the growth…”
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    Changes in purine metabolism and in the immune response in the thymic and splenic lymphocytes of C3HA mice during chemical hepatocarcinogenesis by Khramtsova, S N, Potapova, G I, Shapot, V S

    “…The activities of adenosine deaminase, purine nucleoside phosphorylase, membrane 5'-nucleotidase and DNA content in thymus and spleen lymphocytes as well as…”
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    Methodological approaches to the study of tumor and body tissue metabolism in vitro and in vivo by Shapot, V S, Potapova, G I

    Published in Eksperimentalʹnaja onkologija (1986)
    “…The idea is put forward that in vitro measurements of the potential activity even of the key enzymes are not sufficient for correct evaluation of the rate of…”
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