Search Results - "Podestà, Francesca"

The relation between two polymorphisms in the glucocorticoid receptor gene and body mass index, blood pressure and cholesterol in obese patients by Di Blasio, Anna Maria, Van Rossum, Elisabeth F. C., Maestrini, Sabrina, Berselli, Maria Elisa, Tagliaferri, Mariantonella, Podestà, Francesca, Koper, Jan W., Liuzzi, Antonio, Lamberts, Steven W. J.

“…Summary objective  We have recently reported that, in healthy elderly Dutch individuals, a N363S polymorphism in the glucocorticoid receptor (GR) gene is…”
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Cyclic RGD‐Containing Functionalized Azabicycloalkane Peptides as Potent Integrin Antagonists for Tumor Targeting by Manzoni, Leonardo, Belvisi, Laura, Arosio, Daniela, Civera, Monica, Pilkington‐Miksa, Michael, Potenza, Donatella, Caprini, Andrea, Araldi, Elena M. V., Monferini, Eugenia, Mancino, Monica, Podestà, Francesca, Scolastico, Carlo

“…Vitronectin receptors αvβ3 and αvβ5 have emerged as potential therapeutic targets for the treatment of osteoporosis, restenosis, ocular disease, tumor‐induced…”
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IGF-I mRNA and Signaling in the Diabetic Retina by GERHARDINGER, Chiara, MCCLURE, Kimberly D, ROMEO, Giulio, PODESTA, Francesca, LORENZI, Mara

“…IGF-I mRNA and Signaling in the Diabetic Retina Chiara Gerhardinger , Kimberly D. McClure , Giulio Romeo , Francesca Podestà and Mara Lorenzi From the Schepens…”
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Bax Is Increased in the Retina of Diabetic Subjects and Is Associated with Pericyte Apoptosis in Vivo and in Vitro by Podestà, Francesca, Romeo, Giulio, Liu, Wei-Hua, Krajewski, Stanislaw, Reed, John C., Gerhardinger, Chiara, Lorenzi, Mara

“…Diabetes of even short duration accelerates the death of capillary cells and neurons in the inner retina by a process consistent with apoptosis. We examined…”
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Integrin Overexpression Induced by High Glucose and by Human Diabetes: Potential Pathway to Cell Dysfunction in Diabetic Microangiopathy by Roth, Timothy, Podesta, Francesca, Stepp, Mary Ann, Boeri, Daria, Lorenzi, Mara

“…The nature of the process leading to the acellular nonperfused capillaries of diabetic microangiopathy remains unknown. Because these capillaries manifest…”
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Low Ca2+ pump activity in diabetic nephropathy by Podestá, Francesca, Meregalli, Giancarla, Ghelardi, Renata, Del Giudice, Rosmina, Asnaghi, Veronica, Maestroni, Anna, Zerbini, Gianpaolo

“…Elevated cell Na+-H+ exchange (NHE) activity characterizes diabetic nephropathy (DN), but the mechanisms of this abnormality are unclear. Recent evidence…”
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Response of Capillary Cell Death to Aminoguanidine Predicts the Development of Retinopathy: Comparison of Diabetes and Galactosemia by Kern, Timothy S, Tang, Jie, Mizutani, Masakazu, Kowluru, Renu A, Nagaraj, Ram H, Romeo, Giulio, Podesta, Francesca, Lorenzi, Mara

“…To examine the relationship between early retinal capillary cell apoptosis and late histologic lesions of diabetic retinopathy and to compare the effects of…”
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Underestimate psychological impact of diet intervention by Zoppo, Adele, Asteria, Carmela, Podesta, Francesca, Baggi, Laura

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Retinal Levels of Occludin are Increased in Background Diabetic Retinopathy and are Associated with Overexpression of Glial Fibrillar Acidic Protein by GERHARDINGER, CHIARA, PODESTA, FRANCESCA, LORENZI, MARA

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Fibroblast Na+–Li+ countertransport rate is elevated in essential hypertension by Zerbini, Gianpaolo, Podesta, Francesca, Meregalli, Giancarla, Deferrari, Giacomo, Pontremoli, Roberto

“…OBJECTIVESElevated erythrocyte Na+–Li+ countertransport (SLC) rates are commonly found in essential hypertension. We have recently shown that human skin…”
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Activated Nuclear Factor-kB in Pericytes is Associated with Early Lesions of Human Diabetic Retinopathy by ROMEO, GIULIO, PODESTA, FRANCESCA, KERN, TIMOTHY S, LORENZI, MARA

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Increased expression of tissue plasminogen activator and its inhibitor and reduced fibrinolytic potential of human endothelial cells cultured in elevated glucose by Maiello, M, Boeri, D, Podesta, F, Cagliero, E, Vichi, M, Odetti, P, Adezati, L, Lorenzi, M

“…In diabetic patients, elevated plasma levels of t-PA and PAI-1 accompany impaired fibrinolysis. To identify mechanisms for these abnormalities, we examined…”
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