Search Results - "Pieragostini, Andrea"

Characteristics and incidence of infections in patients with multiple myeloma treated by bispecific antibodies: a national retrospective study by Jourdes, Aurélie, Cellerin, Elise, Touzeau, Cyrille, Harel, Stéphanie, Denis, Blandine, Escure, Guillaume, Faure, Emmanuel, Jamard, Simon, Danion, Francois, Sonntag, Cécile, Ader, Florence, Karlin, Lionel, Soueges, Sarah, Cazelles, Clarisse, de La Porte des Vaux, Clémentine, Frenzel, Laurent, Lanternier, Fanny, Brousse, Xavier, Cazaubiel, Titouan, Berger, Pierre, Collignon, Aude, Blot, Mathieu, Pieragostini, Andrea, Charles, Morgane, Chaleteix, Carine, Redor, Alexis, Roland, Virginie, Cartau, Tom, Macro, Margaret, Chalopin, Thomas, Vallet, Nicolas, Perrot, Aurore, Martin-Blondel, Guillaume

“…Bispecific antibodies (BsAbs) are an effective treatment used in relapsed or refractory multiple myeloma. Despite a well-tolerated safety profile, infectious…”
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Cumulative Incidence and Characteristics of Infections Requiring Treatment, Delay in Treatment Administration or Hospitalisation in Patients with Relapsed or Refractory Multiple Myeloma Treated with Anti BCMA or Anti GPRC5D Bispecific Antibodies by Cellerin, Elise, Jourdes, Aurelie, Brousse, Xavier, Vallet, Nicolas, Cartau, Tom, Denis, Blandine, Harel, Stephanie, Jamard, Simon, Redor, Alexis, Cazaubiel, Titouan, Roland, Virginie, Caleteix, Carine, Charles, Morgane, Berger, Pierre, Escure, Guillaume, Collignon, Aude, Faure, Emmanuel, Cazelles, Clarisse, Lanternier, Fanny, de La Porte Des Vaux, Clementine, Frenzel, Laurent, Blot, Mathieu, Danion, Francois, Sonntag, Cécile, Touzeau, Cyrille, Pieragostini, Andrea, Ader, Florence, Karlin, Lionel, Macro, Margaret, martin-Blondel, Guillaume, Chalopin, Thomas, Perrot, Aurore

“…Background. Bispecific antibodies (BsAb) are a new effective treatment for relapsed/refractory multiple myeloma (RRMM). Although these immunotherapies are well…”
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Involvement of the JAK-STAT Pathway in the Molecular Landscape of Fusion-Free Myeloid Neoplasms with Eosinophilia by Groh, Matthieu, Fenwarth, Laurène, Boudry, Augustin, Fournier, Elise, Marceau-Renaut, Alice, Abraham, Julie, Barry, Marly, Blanche, Philippe, Bodard, Quentin, Braun, Thorsten, Chebrek, Safia, Daltro De Oliveira, Rafael, Decamp, Matthieu, Durel, Cécile Audrey, Forcade, Edouard, Gerfaud-Valentin, Mathieu, Golfier, Camille, Gourguechon, Clement, Grardel, Nathalie, Kosmider, Olivier, Labro, Mathilde, Melboucy Belkhir, Sarah, Merabet, Fatiha, Michon, Adrien, Martis, Nihal, Morice, Cécile, Moreau, Stephane, Néel, Antoine, Nicolini, Franck E., Pascal, Laurent, Pasquier, Florence, Pieragostini, Andrea, Roche-Lestienne, Catherine, Rousselot, Philippe, Thiebaut, Anne, Viallard, Jean-Francois, Wemeau, Mathieu, Preudhomme, Claude, Kahn, Jean-Emmanuel, Lefèvre, Guillaume, Duployez, Nicolas

“…Introduction. Gene fusions leading to the constitutive activation of tyrosine kinases (TK) such as PDGFRA, PDGFRB or FGFR1 have been the first recurrent…”
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Involvement of the JAK‐STAT pathway in the molecular landscape of tyrosine kinase fusion‐negative hypereosinophilic syndromes: A nationwide CEREO study by Groh, Matthieu, Fenwarth, Laurène, Labro, Mathilde, Boudry, Augustin, Fournier, Elise, Wemeau, Mathieu, Marceau‐Renaut, Alice, Daltro de Oliveira, Rafael, Abraham, Julie, Barry, Marly, Blanche, Philippe, Bodard, Quentin, Braun, Thorsten, Chebrek, Safia, Decamp, Matthieu, Durel, Cécile‐Audrey, Forcade, Edouard, Gerfaud‐Valentin, Mathieu, Golfier, Camille, Gourguechon, Clément, Grardel, Nathalie, Kosmider, Olivier, Martis, Nihal, Melboucy Belkhir, Sarah, Merabet, Fatiha, Michon, Adrien, Moreau, Stéphane, Morice, Cécile, Néel, Antoine, Nicolini, Franck E., Pascal, Laurent, Pasquier, Florence, Pieragostini, Andrea, Roche‐Lestienne, Catherine, Rousselot, Philippe, Terriou, Louis, Thiebaut‐Bertrand, Anne, Viallard, Jean‐François, Preudhomme, Claude, Kahn, Jean‐Emmanuel, Lefevre, Guillaume, Duployez, Nicolas

“…We investigated using a custom NGS panel of 149 genes the mutational landscape of 64 consecutive adult patients with tyrosine kinase fusion‐negative…”
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