Search Results - "Petyunina, O V"

Т786С POLYMORPHISM IN THE ENDOTHELIAL NO SYNTHASE GENE, VASCULAR ENDOTHELIAL GROWTH FACTOR-А IN MYOCARDIAL INFARCTION WITH ST SEGMENT ELEVATION by Petyunina, O. V., Kopytsya, M. P., Pietienova, L. L.

“…Purpose. To investigate associations of polymorphism Т786С in the eNOS gene with clinical and anamnestic data in patients with ST-segment elevation myocardial…”
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Predicting adverse cardiac remodeling following ST-elevation myocardial infarction through the assessment of endothelin-1 levels by Vishnevskaya, I, Petyunina, O V, Kopytsya, M P

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P6604Role of macrophage migration inhibitory factor and early after infarction remodeling in patients with ST-segment elevation myocardial infarction by Storozhenko, T E, Kopytsya, M P, Vyshnevska, I R, Petyunina, O V

“…Abstract A heart failure formation in patients with ST-segment elevation myocardial infarction (STEMI) is the most often adverse outcome and determined by…”
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THE INFLUENCE OF ANXIETY AND DEPRESSIVE CONDITIONS ON AFTERINFARCTION REMODELING IN PATIENTS WITH STEMI by O. V. Petyunina

“…Depression after AMI increases the frequency of re-hospitalization because of acute coronary syndrome, heart failure, MI, and is a risk factor for cardiac…”
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P4608Soluble st2 for predicting acute kidney injury in patients with st-segment elevation myocardial infarction by Vyshnevska, I R, Hylova, Y V, Petyunina, O V, Kopytsya, M P

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ALLELE STATUS OF ALDOSTERONE SYNTHASE (CYP11B2) GENE POLYMORPHISM AND CARDIAC REMODELING AFTER ST SEGMENT ELEVATION MYOCARDIAL INFARCTION by Petyunina O. V., Kopytsya M. P., Vyshnevska I. R.

“…Aldosterone plays an important role in the development of reparative and reactive fibrosis and cardiac remodeling (CR) after myocardial infarction. The…”
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THE ROLE OF A NEW BIOMARKER GROWTH DIFFERENTIATION FACTOR 15 IN PROGNOSIS OF PATIENTS WITH ACUTE CORONARY SYNDROME AND TYPE 2 DIABETES MELLITUS by M. P. Kopytsya, O. V. Petyunina, I. R. Vyshnevska, N. V. Tytarenko, Y. V. Hilova

“…Numerous studies confirm worse results in diabetic patients with acute coronary syndrome (ACS) compared with non-diabetic patients. Different mechanisms…”
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