Search Results - "Pelker, Jeffrey W"

TH1-mediated airway hyperresponsiveness independent of neutrophilic inflammation by JUNQING CUI, PAZDZIORKO, Stephen, WILLIAMS, Cara M. M, MARUSIC, Suzana, MIYASHIRO, Joy S, THAKKER, Paresh, PELKER, Jeffrey W, DECLERCQ, Charlene, AIPING JIAO, GUNN, Jason, MASON, Lawrence, LEONARD, John P

“…T(H)2-mediated allergic asthma is characterized by eosinophilia, mucus overproduction, and airway hyperresponsiveness (AHR). Although it is clear that T(H)2…”
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Human bronchial epithelial cells express and secrete MMP-12 by Lavigne, Mark C., Thakker, Paresh, Gunn, Jason, Wong, Anthony, Miyashiro, Joy S., Wasserman, Aeona M., Wei, Shui-Qing, Pelker, Jeffrey W., Kobayashi, Michiko, Eppihimer, Michael J.

“…Matrix metalloproteinases (MMPs) degrade extracellular matrix proteins, which may be responsible for enlargement of alveoli in chronic obstructive pulmonary…”
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PD-1/PD-L1, but not PD-1/PD-L2, interactions regulate the severity of experimental autoimmune encephalomyelitis by Carter, Laura L, Leach, Michael W, Azoitei, Mihai L, Cui, Junqing, Pelker, Jeffrey W, Jussif, Jason, Benoit, Steve, Ireland, Gretchen, Luxenberg, Deborah, Askew, G. Roger, Milarski, Kim L, Groves, Christopher, Brown, Tom, Carito, Brenda A, Percival, Karen, Carreno, Beatriz M, Collins, Mary, Marusic, Suzana

“…Abstract Interactions between PD-1 and its two differentially expressed ligands, PD-L1 and PD-L2, attenuate T cell activation and effector function. To…”
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Cytosolic phospholipase A2 alpha-deficient mice are resistant to experimental autoimmune encephalomyelitis by Marusic, Suzana, Leach, Michael W, Pelker, Jeffrey W, Azoitei, Mihai L, Uozumi, Naonori, Cui, Junqing, Shen, Marina W H, DeClercq, Charlene M, Miyashiro, Joy S, Carito, Brenda A, Thakker, Paresh, Simmons, David L, Leonard, John P, Shimizu, Takao, Clark, James D

“…Experimental autoimmune encephalomyelitis (EAE), a Th1-mediated inflammatory disease of the central nervous system (CNS), is a model of human multiple…”
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Blockade of cytosolic phospholipase A2 α prevents experimental autoimmune encephalomyelitis and diminishes development of Th1 and Th17 responses by Marusic, Suzana, Thakker, Paresh, Pelker, Jeffrey W, Stedman, Nancy L, Lee, Katherine L, McKew, John C, Han, Lixin, Xu, Xin, Wolf, Stan F, Borey, Adam J, Cui, Junqing, Shen, Marina W.H, Donahue, Fran, Hassan-Zahraee, Mina, Leach, Michael W, Shimizu, Takao, Clark, James D

“…Abstract Cytosolic phospholipase A2 α (cPLA2 α) is the rate-limiting enzyme for release of arachidonic acid, which is converted primarily to prostaglandins via…”
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Local delivery of granulocyte macrophage colony-stimulating factor by retrovirally transduced antigen-specific T cells leads to severe, chronic experimental autoimmune encephalomyelitis in mice by Marusic, Suzana, Miyashiro, Joy S, Douhan, John, Konz, Richard F, Xuan, DeJun, Pelker, Jeffrey W, Ling, Vincent, Leonard, John P, Jacobs, Kenneth A

“…Experimental autoimmune encephalomyelitis (EAE) is an inflammatory disease of the central nervous system (CNS) that can be induced in susceptible mice by the…”
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Blockade of cytosolic phospholipase A 2α prevents experimental autoimmune encephalomyelitis and diminishes development of Th1 and Th17 responses by Marusic, Suzana, Thakker, Paresh, Pelker, Jeffrey W., Stedman, Nancy L., Lee, Katherine L., McKew, John C., Han, Lixin, Xu, Xin, Wolf, Stan F., Borey, Adam J., Cui, Junqing, Shen, Marina W.H., Donahue, Fran, Hassan-Zahraee, Mina, Leach, Michael W., Shimizu, Takao, Clark, James D.

“…Cytosolic phospholipase A 2α (cPLA 2α) is the rate-limiting enzyme for release of arachidonic acid, which is converted primarily to prostaglandins via the…”
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T H1-mediated airway hyperresponsiveness independent of neutrophilic inflammation by Cui, Junqing, Pazdziorko, Stephen, Miyashiro, Joy S., Thakker, Paresh, Pelker, Jeffrey W., DeClercq, Charlene, Jiao, Aiping, Gunn, Jason, Mason, Lawrence, Leonard, John P., Williams, Cara M.M., Marusic, Suzana

“…T H2-mediated allergic asthma is characterized by eosinophilia, mucus overproduction, and airway hyperresponsiveness (AHR). Although it is clear that T H2…”
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