Search Results - "Page, Patrick G.T."

A conserved annexin A6–mediated membrane repair mechanism in muscle, heart, and nerve by Demonbreun, Alexis R., Bogdanovic, Elena, Vaught, Lauren A., Reiser, Nina L., Fallon, Katherine S., Long, Ashlee M., Oosterbaan, Claire C., Hadhazy, Michele, Page, Patrick G.T., Joseph, Prem Raj B., Cowen, Gabrielle, Telenson, Alexander M., Khatri, Ammaarah, Sadleir, Katherine R., Vassar, Robert, McNally, Elizabeth M.

“…Membrane instability and disruption underlie myriad acute and chronic disorders. Anxa6 encodes the membrane-associated protein annexin A6 and was identified as…”
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The extracellular matrix differentially directs myoblast motility and differentiation in distinct forms of muscular dystrophy by Long, Ashlee M., Kwon, Jason M., Lee, GaHyun, Reiser, Nina L., Vaught, Lauren A., O'Brien, Joseph G., Page, Patrick G.T., Hadhazy, Michele, Reynolds, Joseph C., Crosbie, Rachelle H., Demonbreun, Alexis R., McNally, Elizabeth M.

“…•Spatial architecture and composition of the ECM differ across genetically distinct forms of muscular dystrophy, especially with respect to Annexin A6 protein…”
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Anti-latent TGFβ binding protein 4 antibody improves muscle function and reduces muscle fibrosis in muscular dystrophy by Demonbreun, Alexis R, Fallon, Katherine S, Oosterbaan, Claire C, Vaught, Lauren A, Reiser, Nina L, Bogdanovic, Elena, Velez, Matthew P, Salamone, Isabella M, Page, Patrick G T, Hadhazy, Michele, Quattrocelli, Mattia, Barefield, David Y, Wood, Lauren D, Gonzalez, J Patrick, Morris, Carl, McNally, Elizabeth M

“…Duchenne muscular dystrophy, like other muscular dystrophies, is a progressive disorder hallmarked by muscle degeneration, inflammation, and fibrosis. Latent…”
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The extracellular matrix differentially directs myoblast motility and differentiation in distinct forms of muscular dystrophy: Dystrophic matrices alter myoblast motility by Long, Ashlee M, Kwon, Jason M, Lee, GaHyun, Reiser, Nina L, Vaught, Lauren A, O'Brien, Joseph G, Page, Patrick G T, Hadhazy, Michele, Reynolds, Joseph C, Crosbie, Rachelle H, Demonbreun, Alexis R, McNally, Elizabeth M

“…Extracellular matrix (ECM) pathologic remodeling underlies many disorders, including muscular dystrophy. Tissue decellularization removes cellular components…”
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Altered Enhancer and Promoter Usage Leads to Differential Gene Expression in the Normal and Failed Human Heart by Gacita, Anthony M., Dellefave-Castillo, Lisa, Page, Patrick G.T., Barefield, David Y., Wasserstrom, J. Andrew, Puckelwartz, Megan J., Nobrega, Marcelo A., McNally, Elizabeth M.

“…The failing heart is characterized by changes in gene expression. However, the regulatory regions of the genome that drive these gene expression changes have…”
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Dusp6 is a genetic modifier of growth through enhanced ERK activity by Vo, Andy H, Swaggart, Kayleigh A, Woo, Anna, Gao, Quan Q, Demonbreun, Alexis R, Fallon, Katherine S, Quattrocelli, Mattia, Hadhazy, Michele, Page, Patrick G T, Chen, Zugen, Eskin, Ascia, Squire, Kevin, Nelson, Stanley F, McNally, Elizabeth M

“…Abstract Like other single-gene disorders, muscular dystrophy displays a range of phenotypic heterogeneity even with the same primary mutation. Identifying…”
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The super-healing MRL strain promotes muscle growth in muscular dystrophy through a regenerative extracellular matrix by O'Brien, Joseph G, Willis, Alexander B, Long, Ashlee M, Kwon, Jason M, Lee, GaHyun, Li, Frank W, Page, Patrick Gt, Vo, Andy H, Hadhazy, Michele, Spencer, Melissa J, Crosbie, Rachelle H, Demonbreun, Alexis R, McNally, Elizabeth M

“…The Murphy Roths Large (MRL) mouse strain has "super-healing" properties that enhance recovery from injury. In mice, the DBA/2J strain intensifies many aspects…”
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