Search Results - "PATZEL, Eva"

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  1. 1

    The myosin-interacting protein SMYD1 is essential for sarcomere organization by Just, Steffen, Meder, Benjamin, Berger, Ina M, Etard, Christelle, Trano, Nicole, Patzel, Eva, Hassel, David, Marquart, Sabine, Dahme, Tillman, Vogel, Britta, Fishman, Mark C, Katus, Hugo A, Strähle, Uwe, Rottbauer, Wolfgang

    Published in Journal of cell science (15-09-2011)
    “…Assembly, maintenance and renewal of sarcomeres require highly organized and balanced folding, transport, modification and degradation of sarcomeric proteins…”
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    Journal Article
  2. 2

    The Zebrafish Anillin-eGFP Reporter Marks Late Dividing Retinal Precursors and Stem Cells Entering Neuronal Lineages by Cepero Malo, Meret, Duchemin, Anne-Laure, Guglielmi, Luca, Patzel, Eva, Sel, Saadettin, Auffarth, Gerd U, Carl, Matthias, Poggi, Lucia

    Published in PloS one (20-01-2017)
    “…Monitoring cycling behaviours of stem and somatic cells in the living animal is a powerful tool to better understand tissue development and homeostasis. The…”
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    Temporal and spatial expression pattern of Nnat during mouse eye development by Sel, Saadettin, Patzel, Eva, Poggi, Lucia, Kaiser, Delia, Kalinski, Thomas, Schicht, Martin, Paulsen, Friedrich, Nass, Norbert

    Published in Gene Expression Patterns (01-01-2017)
    “…Neuronatin (Nnat) was initially identified as a highly expressed gene in neonatal mammalian brain. In this study, we analyze the spatial and temporal…”
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  5. 5

    Protein Kinase D2 Controls Cardiac Valve Formation in Zebrafish by Regulating Histone Deacetylase 5 Activity by JUST, Steffen, BERGER, Ina M, ROTTBAUER, Wolfgang, MEDER, Benjamin, BACKS, Johannes, KELLER, Andreas, MARQUART, Sabine, FRESE, Karen, PATZEL, Eva, RAUCH, Gerd-Jörg, KATUS, Hugo A

    Published in Circulation (New York, N.Y.) (19-07-2011)
    “…The molecular mechanisms that guide heart valve formation are not well understood. However, elucidation of the genetic basis of congenital heart disease is one…”
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  6. 6

    Reconstitution of defective protein trafficking rescues Long-QT syndrome in zebrafish by Meder, Benjamin, Scholz, Eberhard P., Hassel, David, Wolff, Christoph, Just, Steffen, Berger, Ina M., Patzel, Eva, Karle, Christoph, Katus, Hugo A., Rottbauer, Wolfgang

    “…► Embryonic and adult breakdance zebrafish mutants are a valuable model for human Long-QT syndrome. ► The breakdance mutation (zERG I59S) disrupts ERG protein…”
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    Journal Article
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