Search Results - "Oswald, Kelli A."

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    Gleevec-mediated inhibition of Rad51 expression and enhancement of tumor cell radiosensitivity by RUSSELL, Jeffery S, BRADY, Kristin, BURGAN, William E, CERRA, Michael A, OSWALD, Kelli A, CAMPHAUSEN, Kevin, TOFILON, Philip J

    Published in Cancer research (Chicago, Ill.) (01-11-2003)
    “…Rad51 is an essential component of the homologous DNA repair pathway and has been implicated as a determinant of cellular radiosensitivity. Gleevec is a…”
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    Journal Article
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    Enhanced cell killing induced by the combination of radiation and the heat shock protein 90 inhibitor 17-allylamino-17-demethoxygeldanamycin: A multitarget approach to radiosensitization by RUSSELL, Jeffery S, BURGAN, William, OSWALD, Kelli A, CAMPHAUSEN, Kevin, TOFILON, Philip J

    Published in Clinical cancer research (01-09-2003)
    “…Current strategies for tumor cell radiosensitization focus on a target-based approach. However, the radioresponse of a tumor cell is influenced by a wide…”
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    Journal Article
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    Enhanced Radiation-Induced Cell Killing and Prolongation of γH2AX Foci Expression by the Histone Deacetylase Inhibitor MS-275 by Camphausen, Kevin, Burgan, William, Cerra, Michael, Oswald, Kelli A., Trepel, Jane B., Lee, Min-Jung, Tofilon, Philip J.

    Published in Cancer research (Chicago, Ill.) (01-01-2004)
    “…Histone deacetylase (HDAC) inhibitors are undergoing clinical evaluation for cancer therapy. Because HDAC modulates chromatin structure and gene expression,…”
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    Journal Article
  5. 5

    Enhanced Cell Killing Induced by the Combination of Radiation and the Heat Shock Protein 90 Inhibitor 17-Allylamino-17- Demethoxygeldanamycin by Jeffery S. Russell, William Burgan, Kelli A. Oswald, Kevin Camphausen, Philip J. Tofilon

    Published in Clinical cancer research (01-09-2003)
    “…Purpose: Current strategies for tumor cell radiosensitization focus on a target-based approach. However, the radioresponse of a tumor cell is influenced by a…”
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    Journal Article
  6. 6

    Enhanced radiation-induced cell killing and prolongation of gammaH2AX foci expression by the histone deacetylase inhibitor MS-275 by Camphausen, Kevin, Burgan, William, Cerra, Michael, Oswald, Kelli A, Trepel, Jane B, Lee, Min-Jung, Tofilon, Philip J

    Published in Cancer research (Chicago, Ill.) (01-01-2004)
    “…Histone deacetylase (HDAC) inhibitors are undergoing clinical evaluation for cancer therapy. Because HDAC modulates chromatin structure and gene expression,…”
    Get full text
    Journal Article
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