Search Results - "Oswald, Kelli A."
-
1
Enhanced Tumor Cell Radiosensitivity and Abrogation of G2 and S Phase Arrest by the Hsp90 Inhibitor 17-(Dimethylaminoethylamino)-17-demethoxygeldanamycin
Published in Clinical cancer research (01-12-2004)“…Purpose: Because of the potential for affecting multiple signaling pathways, inhibition of Hsp90 may provide a strategy for enhancing tumor cell…”
Get full text
Journal Article -
2
Gleevec-mediated inhibition of Rad51 expression and enhancement of tumor cell radiosensitivity
Published in Cancer research (Chicago, Ill.) (01-11-2003)“…Rad51 is an essential component of the homologous DNA repair pathway and has been implicated as a determinant of cellular radiosensitivity. Gleevec is a…”
Get full text
Journal Article -
3
Enhanced cell killing induced by the combination of radiation and the heat shock protein 90 inhibitor 17-allylamino-17-demethoxygeldanamycin: A multitarget approach to radiosensitization
Published in Clinical cancer research (01-09-2003)“…Current strategies for tumor cell radiosensitization focus on a target-based approach. However, the radioresponse of a tumor cell is influenced by a wide…”
Get full text
Journal Article -
4
Enhanced Radiation-Induced Cell Killing and Prolongation of γH2AX Foci Expression by the Histone Deacetylase Inhibitor MS-275
Published in Cancer research (Chicago, Ill.) (01-01-2004)“…Histone deacetylase (HDAC) inhibitors are undergoing clinical evaluation for cancer therapy. Because HDAC modulates chromatin structure and gene expression,…”
Get full text
Journal Article -
5
Enhanced Cell Killing Induced by the Combination of Radiation and the Heat Shock Protein 90 Inhibitor 17-Allylamino-17- Demethoxygeldanamycin
Published in Clinical cancer research (01-09-2003)“…Purpose: Current strategies for tumor cell radiosensitization focus on a target-based approach. However, the radioresponse of a tumor cell is influenced by a…”
Get full text
Journal Article -
6
Enhanced radiation-induced cell killing and prolongation of gammaH2AX foci expression by the histone deacetylase inhibitor MS-275
Published in Cancer research (Chicago, Ill.) (01-01-2004)“…Histone deacetylase (HDAC) inhibitors are undergoing clinical evaluation for cancer therapy. Because HDAC modulates chromatin structure and gene expression,…”
Get full text
Journal Article -
7