Search Results - "Olijslagers, J. E."
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Rapid changes in hippocampal CA1 pyramidal cell function via pre- as well as postsynaptic membrane mineralocorticoid receptors
Published in The European journal of neuroscience (01-05-2008)“…Corticosterone (100 nm) rapidly increases the frequency of miniature excitatory postsynaptic currents in mouse CA1 pyramidal neurons via membrane‐located…”
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5-HT2 receptors differentially modulate dopamine-mediated auto-inhibition in A9 and A10 midbrain areas of the rat
Published in Neuropharmacology (01-03-2004)“…5-HT (20 microM) enhanced dopamine (DA) D2-like receptor mediated reduction of the firing rate of DA neurons in the substantia nigra pars compacta (A9) and…”
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Modulation of Midbrain Dopamine Neurotransmission by Serotonin, a Versatile Interaction Between Neurotransmitters and Significance for Antipsychotic Drug Action
Published in Current neuropharmacology (01-01-2006)“…Schizophrenia has been associated with a dysfunction of brain dopamine (DA). This, so called, DA hypothesis has been refined as new insights into the…”
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The role of 5-HT2A receptor antagonism in amphetamine-induced inhibition of A10 dopamine neurons in vitro
Published in European journal of pharmacology (27-09-2005)“…The role of the 5-HT2A receptor in modulating amphetamine-induced inhibition of dopamine neuronal firing in A9 and A10 was investigated in rat midbrain slices…”
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5-HT sub(2) receptors differentially modulate dopamine-mediated auto-inhibition in A9 and A10 midbrain areas of the rat
Published in Neuropharmacology (01-03-2004)“…5-HT (20 mu M) enhanced dopamine (DA) D sub(2)-like receptor mediated reduction of the firing rate of da neurons in the substantia nigra pars compacta (A9) and…”
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Quetiapine increases the firing rate of rat substantia nigra and ventral tegmental area dopamine neurons in vitro
Published in European journal of pharmacology (03-12-2004)“…The antipsychotic drug quetiapine increases the firing rate of dopamine neurons in the substantia nigra and the ventral tegmental area of the rat. In the…”
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