Search Results - "Ohle, Sarah J."

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  1. 1

    Lentiviral Delivery of RNAi for In Vivo Lineage-Specific Modulation of Gene Expression in Mouse Lung Macrophages by Wilson, Andrew A, Kwok, Letty W, Porter, Emily L, Payne, Julie G, McElroy, Gregory S, Ohle, Sarah J, Greenhill, Sara R, Blahna, Matthew T, Yamamoto, Kazuko, Jean, Jyh C, Mizgerd, Joseph P, Kotton, Darrell N

    Published in Molecular therapy (01-04-2013)
    “…Although RNA interference (RNAi) has become a ubiquitous laboratory tool since its discovery 12 years ago, in vivo delivery to selected cell types remains a…”
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    Journal Article
  2. 2

    Maintenance and repair of the lung endothelium does not involve contributions from marrow-derived endothelial precursor cells by Ohle, Sarah J, Anandaiah, Asha, Fabian, Attila J, Fine, Alan, Kotton, Darrell N

    “…Lung endothelium is believed to be a quiescent tissue with the potential to exhibit rapid and effective repair after injury. Endothelial progenitor cells…”
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    Journal Article
  3. 3

    Sustained Expression of α1-Antitrypsin after Transplantation of Manipulated Hematopoietic Stem Cells by Wilson, Andrew A., Kwok, Letty W., Hovav, Avi-Hai, Ohle, Sarah J., Little, Frederic F., Fine, Alan, Kotton, Darrell N.

    “…Inherited mutations in the human α 1 -antitrypsin (AAT) gene lead to deficient circulating levels of AAT protein and a predisposition to developing emphysema…”
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  4. 4

    Sustained Expression of [alpha]^sub 1^-Antitrypsin after Transplantation of Manipulated Hematopoietic Stem Cells by Wilson, Andrew A, Kwok, Letty W, Hovav, Avi-Hai, Ohle, Sarah J, Little, Frederic F, Fine, Alan, Kotton, Darrell N

    “…Inherited mutations in the human alpha(1)-antitrypsin (AAT) gene lead to deficient circulating levels of AAT protein and a predisposition to developing…”
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    Journal Article
  5. 5
  6. 6

    Sustained expression of alpha1-antitrypsin after transplantation of manipulated hematopoietic stem cells by Wilson, Andrew A, Kwok, Letty W, Hovav, Avi-Hai, Ohle, Sarah J, Little, Frederic F, Fine, Alan, Kotton, Darrell N

    “…Inherited mutations in the human alpha(1)-antitrypsin (AAT) gene lead to deficient circulating levels of AAT protein and a predisposition to developing…”
    Get full text
    Journal Article
  7. 7