Search Results - "Navasa, N."

MCJ/DnaJC15, the Mitochondrial Foe in Liver Injury by Torres, L.B, Iruzubieta, P, Taibo, D, Cardoso, T, Navasa, N, Ramos, D.F, Rey, M.V, de Juan, V.G, Tussy, P.F, Franco, I.Z, Alvarez, M.I.H, Andrade, R, Medina, I, Monte, M.J, Marín, J.J.G, Crespo, J, Zorzano, A, Mato, J.M, Anguita, J, Rincon, M, Chantar, M.L.M

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14 EFFICAY AND SAFETY OF PEGYLATED INTERFERON AND RIBAVIRIN IN HCV-INFECTED PATIENTS AWAITING LIVER TRANSPLANTATION: A CASE-CONTROL STUDY by Carrion, J.A, Martinez-Bauer, E, Crespo, G, Garcia-Valdecasas, J.C, Navasa, N, Forns, X

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The mitochondrial negative regulator MCJ is a therapeutic target for acetaminophen-induced liver injury by Barbier-Torres, Lucía, Iruzubieta, Paula, Fernández-Ramos, David, Delgado, Teresa C., Taibo, Daniel, Guitiérrez-de-Juan, Virginia, Varela-Rey, Marta, Azkargorta, Mikel, Navasa, Nicolas, Fernández-Tussy, Pablo, Zubiete-Franco, Imanol, Simon, Jorge, Lopitz-Otsoa, Fernando, Lachiondo-Ortega, Sofia, Crespo, Javier, Masson, Steven, McCain, Misti Vanette, Villa, Erica, Reeves, Helen, Elortza, Felix, Lucena, Maria Isabel, Hernández-Alvarez, Maria Isabel, Zorzano, Antonio, Andrade, Raúl J., Lu, Shelly C., Mato, José M., Anguita, Juan, Rincón, Mercedes, Martínez-Chantar, María Luz

“…Acetaminophen (APAP) is the active component of many medications used to treat pain and fever worldwide. Its overuse provokes liver injury and it is the second…”
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THU-301 - MCJ/DnaJC15, the Mitochondrial Foe in Liver Injury by Torres, L.B., Iruzubieta, P., Taibo, D., Cardoso, T., Navasa, N., Ramos, D.F., Rey, M.V., de Juan, V.G., Tussy, P.F., Franco, I.Z., Alvarez, M.I.H., Andrade, R., Medina, I., Monte, M.J., Marín, J.J.G., Crespo, J., Zorzano, A., Mato, J.M., Anguita, J., Rincon, M., Chantar, M.L.M.

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Purification and characterization of GlcNAc-6-P 2-epimerase from Escherichia coli K92 by Ferrero, Miguel Angel, Martínez-Blanco, Honorina, Lopez-Velasco, Federico Felino, Ezquerro-Sáenz, Carlos, Navasa, Nicolas, Lozano, Sofia, Rodríguez-Aparicio, Leandro B

“…N-Acetylmannosamine (ManNAc) is the first committed intermediate in sialic acid metabolism. Thus, the mechanisms that control intracellular ManNAc levels are…”
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