Search Results - "Naumann, Steffen C"

Polo-like kinase 3 regulates CtIP during DNA double-strand break repair in G1 by Barton, Olivia, Naumann, Steffen C, Diemer-Biehs, Ronja, Künzel, Julia, Steinlage, Monika, Conrad, Sandro, Makharashvili, Nodar, Wang, Jiadong, Feng, Lin, Lopez, Bernard S, Paull, Tanya T, Chen, Junjie, Jeggo, Penny A, Löbrich, Markus

“…DNA double-strand breaks (DSBs) are repaired by nonhomologous end joining (NHEJ) or homologous recombination (HR). The C terminal binding protein-interacting…”
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Inefficient Double-Strand Break Repair in Murine Rod Photoreceptors with Inverted Heterochromatin Organization by Frohns, Antonia, Frohns, Florian, Naumann, Steffen C., Layer, Paul G., Löbrich, Markus

“…DNA double-strand break (DSB) repair is crucial for the maintenance of genomic stability, and chromatin organization represents one important factor…”
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Translesion polymerase η is upregulated by cancer therapeutics and confers anticancer drug resistance by Tomicic, Maja T, Aasland, Dorthe, Naumann, Steffen C, Meise, Ruth, Barckhausen, Christina, Kaina, Bernd, Christmann, Markus

“…DNA repair processes are a key determinant of the sensitivity of cancer cells to DNA-damaging chemotherapeutics, which may induce certain repair genes as a…”
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Brca2/Xrcc2 dependent HR, but not NHEJ, is required for protection against O(6)-methylguanine triggered apoptosis, DSBs and chromosomal aberrations by a process leading to SCEs by Roos, Wynand P, Nikolova, Teodora, Quiros, Steve, Naumann, Steffen C, Kiedron, Olivia, Zdzienicka, Małgorzata Z, Kaina, Bernd

“…O(6)-methylguanine (O(6)MeG) is a highly critical DNA adduct induced by methylating carcinogens and anticancer drugs such as temozolomide, streptozotocine,…”
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Near-Infrared Exposure Changes Cellular Responses to Ionizing Radiation by Heselich, Anja, Frohns, Florian, Frohns, Antonia, Naumann, Steffen C., Layer, Paul G.

“…Near infrared (NIR) and X‐rays are radiations from different sides of the wavelength spectrum but both are used during medical treatments, as they have severe…”
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Brca2/Xrcc2 dependent HR, but not NHEJ, is required for protection against O6-methylguanine triggered apoptosis, DSBs and chromosomal aberrations by a process leading to SCEs by ROOS, Wynand P, NIKOLOVA, Teodora, QUIROS, Steve, NAUMANN, Steffen C, KIEDRON, Olivia, ZDZIENICKA, Małgorzata Z, KAINA, Bernd

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Brca2/Xrcc2 dependent HR, but not NHEJ, is required for protection against O 6-methylguanine triggered apoptosis, DSBs and chromosomal aberrations by a process leading to SCEs by Roos, Wynand P., Nikolova, Teodora, Quiros, Steve, Naumann, Steffen C., Kiedron, Olivia, Zdzienicka, Małgorzata Z., Kaina, Bernd

“…O 6-methylguanine (O 6MeG) is a highly critical DNA adduct induced by methylating carcinogens and anticancer drugs such as temozolomide, streptozotocine,…”
Get full text