Search Results - "Namba, Masumi"

Creation of X-linked Alport syndrome rat model with Col4a5 deficiency by Namba, Masumi, Kobayashi, Tomoe, Kohno, Mayumi, Koyano, Takayuki, Hirose, Takuo, Fukushima, Masaki, Matsuyama, Makoto

“…Alport syndrome is an inherited chronic human kidney disease, characterized by glomerular basement membrane abnormalities. This disease is caused by mutations…”
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The p21 dependent G2 arrest of the cell cycle in epithelial tubular cells links to the early stage of renal fibrosis by Koyano, Takayuki, Namba, Masumi, Kobayashi, Tomoe, Nakakuni, Kyomi, Nakano, Daisuke, Fukushima, Masaki, Nishiyama, Akira, Matsuyama, Makoto

“…Renal fibrosis is accompanied by the progression of chronic kidney disease. Despite a number of past and ongoing studies, our understanding of the underlying…”
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GONAD: A new method for germline genome editing in mice and rats by Namba, Masumi, Kobayashi, Tomoe, Koyano, Takayuki, Kohno, Mayumi, Ohtsuka, Masato, Matsuyama, Makoto

“…Recent advances in the CRISPR/Cas9 system have demonstrated it to be an efficient gene‐editing technology for various organisms. Laboratory mice and rats are…”
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Publisher Correction: Creation of X-linked Alport syndrome rat model with Col4a5 deficiency by Namba, Masumi, Kobayashi, Tomoe, Kohno, Mayumi, Koyano, Takayuki, Hirose, Takuo, Fukushima, Masaki, Matsuyama, Makoto

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Successful production of genome-edited rats by the rGONAD method by Kobayashi, Tomoe, Namba, Masumi, Koyano, Takayuki, Fukushima, Masaki, Sato, Masahiro, Ohtsuka, Masato, Matsuyama, Makoto

“…Recent progress in development of the CRISPR/Cas9 system has been shown to be an efficient gene-editing technology in various organisms. We recently developed…”
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An improved iliac lymph node method for production of monoclonal antibodies by Kobayashi, Tomoe, Namba, Masumi, Kohno, Mayumi, Koyano, Takayuki, Sado, Yoshikazu, Matsuyama, Makoto

“…Monoclonal antibodies have been applied in a wide range of biological and medical studies since the advent of cell fusion technology. Although cell fusion…”
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Neuromedin U-deficient rats do not lose body weight or food intake by Yokogi, Kyoka, Goto, Yuki, Otsuka, Mai, Ojima, Fumiya, Kobayashi, Tomoe, Tsuchiba, Yukina, Takeuchi, Yu, Namba, Masumi, Kohno, Mayumi, Tetsuka, Minami, Takeuchi, Sakae, Matsuyama, Makoto, Aizawa, Sayaka

“…Studies in genetically modified mice establish that essential roles of endogenous neuromedin U (NMU) are anorexigenic function and metabolic regulation,…”
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WCN24-1083 Functional Analyses of X-linked Alport syndrome rats utilizing the rGONAD technology by Matsuyama, Makoto, Namba, Masumi, Kohno, Mayumi, Kobayashi, Tomoe, Koyano, Takayuki, Fukushima, Masaki

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