Search Results - "Musameh, Muntaser D"

Large-Scale Analysis of Determinants, Stability, and Heritability of High-Density Lipoprotein Cholesterol Efflux Capacity by Koekemoer, Andrea L, Codd, Veryan, Masca, Nicholas G D, Nelson, Christopher P, Musameh, Muntaser D, Kaess, Bernhard M, Hengstenberg, Christian, Rader, Daniel J, Samani, Nilesh J

“…Cholesterol efflux capacity (CEC) has emerged as a biomarker of coronary artery disease risk beyond plasma high-density lipoprotein (HDL) cholesterol (HDL-C)…”
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Surgical or percutaneous repair of sinus of valsalva rupture: Case series and literature review by Paymard, Mohammad, Higgins, Mark, Sinhal, Ajay, Musameh, Muntaser

“…The rupture of the sinus of the Valsalva aneurysm is a rare but very serious condition. Rapid and accurate diagnosis and prompt treatment are critical for…”
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Analysis of gene-gene interactions among common variants in candidate cardiovascular genes in coronary artery disease by Musameh, Muntaser D, Wang, William Y S, Nelson, Christopher P, Lluís-Ganella, Carla, Debiec, Radoslaw, Subirana, Isaac, Elosua, Roberto, Balmforth, Anthony J, Ball, Stephen G, Hall, Alistair S, Kathiresan, Sekar, Thompson, John R, Lucas, Gavin, Samani, Nilesh J, Tomaszewski, Maciej

“…Only a small fraction of coronary artery disease (CAD) heritability has been explained by common variants identified to date. Interactions between genes of…”
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Improved Myocardial Function After Cold Storage With Preservation Solution Supplemented With a Carbon Monoxide–Releasing Molecule (CORM-3) by Musameh, Muntaser D., MD, Green, Colin J., PhD, DSc, Mann, Brian E., PhD, Fuller, Barry J., PhD, DSc, Motterlini, Roberto, PhD

“…Background Carbon monoxide–releasing molecules (CO-RMs) are pharmacologically active as they protect against cardiac graft rejection and cold ischemia–mediated…”
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Mackay Heart Failure Study: Examining the Root Causes, Compliance With Guideline-Based Therapy and Prognosis by Naing, Pyi, Zhang, Michael, Khine, Aye Myat Thandar, Aung, Hnin Sabai, Chean, Li Ning, Liaw, Jacqueline, Bazley, Margaret, Vaidya, Shantisagar, Musameh, Muntaser D., Khan, Ahmed

“…Heart failure patients have poor outcomes comparable to some malignancies; however, the modern guideline directed medical therapy (GDMT) has improved its…”
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Resuscitated cardiac arrest and prognosis following myocardial infarction by Alahmar, Albert E, Nelson, Christopher P, Snell, Kym I E, Yuyun, Matthew F, Musameh, Muntaser D, Timmis, Adam, Birkhead, John S, Chugh, Sumeet S, Thompson, John R, Squire, Iain B, Samani, Nilesh J

“…To determine whether resuscitated cardiac arrest (CA) complicating ST elevation myocardial infarction (STEMI) impacts outcome, particularly in patients…”
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Hypertension – a clinical update for physicians by Musameh, Muntaser D, Tomaszewski, Maciej, Williams, Bryan

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Genetic associations with lipoprotein subfractions provide information on their biological nature by PETERSEN, Ann-Kristin, STARK, Klaus, DANIEL, Hannelore, HAUNER, Hans, ADAMSKI, Jerzy, TOMASZEWSKI, Maciej, DÖRING, Angela, PETERS, Annette, WICHMANN, H.-Erich, KAESS, Bernhard M, KALBITZER, Hans Robert, HUBER, Fritz, MUSAMEH, Muntaser D, PFAHLERT, Volker, SAMANI, Nilesh J, KRONENBERG, Florian, DIEPLINGER, Hans, IIIIG, Thomas, HENGSTENBERG, Christian, SUHRE, Karsten, GIEGER, Christian, KASTENMÜLLER, Gabi, NELSON, Christopher P, RÖMISCH-MARGL, Werner, KREMER, Werner, RAFFLER, Johannes, KRUG, Susanne, SKURK, Thomas, RIST, Manuela J

“…Adverse levels of lipoproteins are highly heritable and constitute risk factors for cardiovascular outcomes. Hitherto, genome-wide association studies revealed…”
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Hypothesis-based analysis of gene-gene interactions and risk of myocardial infarction by Lucas, Gavin, Lluís-Ganella, Carla, Subirana, Isaac, Musameh, Muntaser D, Gonzalez, Juan Ramon, Nelson, Christopher P, Sentí, Mariano, Schwartz, Stephen M, Siscovick, David, O'Donnell, Christopher J, Melander, Olle, Salomaa, Veikko, Purcell, Shaun, Altshuler, David, Samani, Nilesh J, Kathiresan, Sekar, Elosua, Roberto

“…The genetic loci that have been found by genome-wide association studies to modulate risk of coronary heart disease explain only a fraction of its total…”
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CO Liberated From a Carbon Monoxide-Releasing Molecule Exerts a Positive Inotropic Effect in Doxorubicin-Induced Cardiomyopathy by Musameh, Muntaser D, Green, Colin J, Mann, Brian E, Motterlini, Roberto, Fuller, Barry J

“…Carbon monoxide (CO) liberated by a water-soluble carbon monoxide-releasing molecule (CORM-3) induces a positive inotropic effect with a negative chronotropic…”
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Post-Genomic Update on a Classical Candidate Gene for Coronary Artery Disease: ESR1 by LUCAS, Gavin, LLUIS-GANELLA, Carla, ELOSUA, Roberto, SUBIRANA, Isaac, SENTI, Mariano, WILLENBORG, Christina, MUSAMEH, Muntaser D, SCHWARTZ, Stephen M, O'DONNELL, Christopher J, MELANDER, Olle, SALOMAA, Veikko

“…After age, sex is the most important risk factor for coronary artery disease (CAD). The mechanism through which women are protected from CAD is still largely…”
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Large-scale gene-centric analysis identifies novel variants for coronary artery disease by Butterworth, AS, Farrall, M, Hardwick, RJ, Saleheen, D, Peden, JF, Soranzo, N, Chambers, JC, Sivapalaratnam, S, Kleber, ME, Klopp, N, Erdmann, J, Assimes, TL, Ball, SG, Balmforth, AJ, Barnes, TA, Basart, H, Baumert, J, Bezzina, CR, Boerwinkle, E, Boehm, BO, Brocheton, J, Bugert, P, Cambien, F, Clarke, R, Codd, V, Collins, R, Couper, D, Cupples, LA, de Jong, JS, Diemert, P, Ejebe, K, Elbers, CC, Elliott, P, Fornage, M, Franzosi, MG, Frossard, P, Garner, S, Goel, A, Goodall, AH, Hengstenberg, C, Hunt, SE, Kastelein, JJP, Klungel, OH, Kluter, H, Koch, K, Konig, IR, Kooner, AS, Laaksonen, R, Lathrop, M, Li, MY, Liu, K, McPherson, R, Musameh, MD, Musani, S, Nelson, CP, O'Donnell, CJ, Ongen, H, Papanicolaou, G, Peters, A, Peters, BJM, Potter, S, Psaty, BM, Qu, LM, Rader, DJ, Rasheed, A, Rice, C, Scott, J, Seedorf, U, Sehmi, JS, Sotoodehnia, N, Stark, K, Stephens, J, van der Schoot, CE, van der Schouw, YT, Tomaszewski, M, van der Harst, P, Vasan, RS, Wilde, AAM, Willenborg, C, Winkelmann, BR, Zaidi, M, Zhang, WH, Ziegler, A, de Bakker, PIW, Koenig, W, Marz, W, Trip, MD, Reilly, MP, Kathiresan, S, Schunkert, H, Hamsten, A, Hall, AS, Kooner, JS, Thompson, SG, Thompson, JR, Deloukas, P, Ouwehand, WH, Watkins, H, Danesh, J, Samani, NJ

“…Coronary artery disease (CAD) has a significant genetic contribution that is incompletely characterized. To complement genome-wide association (GWA) studies,…”
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Effects of the coronary artery disease associated LPA and 9p21 loci on risk of aortic valve stenosis by Trenkwalder, Teresa, Nelson, Christopher P., Musameh, Muntaser D., Mordi, Ify R., Kessler, Thorsten, Pellegrini, Costanza, Debiec, Radoslaw, Rheude, Tobias, Lazovic, Viktor, Zeng, Lingyao, Martinsson, Andreas, Gustav Smith, J., Gådin, Jesper R., Franco-Cereceda, Anders, Eriksson, Per, Nielsen, Jonas B., Graham, Sarah E., Willer, Cristen J., Hveem, Kristian, Kastrati, Adnan, Braund, Peter S., Palmer, Colin N.A., Aracil, Amparo, Husser, Oliver, Koenig, Wolfgang, Schunkert, Heribert, Lang, Chim C., Hengstenberg, Christian, Samani, Nilesh J.

“…Aortic valve stenosis (AVS) and coronary artery disease (CAD) have a significant genetic contribution and commonly co-exist. To compare and contrast genetic…”
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Abstract 17297: 9p21 CAD Risk Allele is Protective Against Calcific Aortic Stenosis by Musameh, Muntaser D, Braund, Peter S, Nelson, Christopher P, Gracey, Jay, Denniff, Matthew, Thompson, John, McCann, Gerry P, Bown, Matthew J, Sprigings, David, Samani, Nilesh J

“…Abstract only The 9p21 locus has emerged as one of the strongest risk variants associated with coronary artery disease (CAD). The risk allele for CAD is also…”
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69 A FUNCTIONAL GENETIC VARIANT OF FGF1 IS ASSOCIATED WITH BLOOD PRESSURE IN THE GENERAL POPULATION by Tomaszewski, Maciej, Denniff, Matthew, Myers, Stephen, Debiec, Radek, Bloomer, Lisa D.S., Barnes, Timothy, Bogdanski, Pawel, Büsst, Cara, Christofidou, Paraskevi, Nelson, Christopher P., Musameh, Muntaser D., Musialik, Katarzyna, Dominiczak, Anna F., Tobin, Martin, Navis, Gerjan, Zukowska-Szczechowska, Ewa, Harrap, Stephen, van der Harst, Pim, Samani, Nilesh J, Charchar, Fadi J.

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Analysis of Gene-Gene Interactions among Common Variants in Candidate Cardiovascular Genes in Coronary Artery Disease: e0117684 by Musameh, Muntaser D, Wang, William YS, Nelson, Christopher P, Lluis-Ganella, Carla, Debiec, Radoslaw, Subirana, Isaac, Elosua, Roberto, Balmforth, Anthony J, Ball, Stephen G, Hall, Alistair S

“…Objective Only a small fraction of coronary artery disease (CAD) heritability has been explained by common variants identified to date. Interactions between…”
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Post-Genomic Update on a Classical Candidate Gene for Coronary Artery Disease by Lucas, Gavin, Lluís-Ganella, Carla, Subirana, Isaac, Sentí, Mariano, Willenborg, Christina, Musameh, Muntaser D, Schwartz, Stephen M, OʼDonnell, Christopher J, Melander, Olle, Salomaa, Veikko, Elosua, Roberto

“…BACKGROUND—After age, sex is the most important risk factor for coronary artery disease (CAD). The mechanism through which women are protected from CAD is…”
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