Search Results - "Murch, O."

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  1. 1

    Inhibition of endogenous hydrogen sulfide formation reduces the organ injury caused by endotoxemia by Collin, Marika, Anuar, Farhana B M, Murch, Oliver, Bhatia, Madhav, Moore, Philip K, Thiemermann, Christoph

    Published in British journal of pharmacology (01-10-2005)
    “…Hydrogen sulfide (H2S) is a naturally occurring gaseous transmitter, which may play important roles in normal physiology and disease. Here, we investigated the…”
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    Journal Article
  2. 2

    Peroxisome proliferator-activated receptor-γ antagonists GW9662 and T0070907 reduce the protective effects of lipopolysaccharide preconditioning against organ failure caused by endotoxemia by Collin, Marika, Murch, Oliver, Thiemermann, Christoph

    Published in Critical care medicine (01-04-2006)
    “…OBJECTIVE:There is evidence that a) ligands of the nuclear receptor peroxisome proliferator-activated receptor (PPAR)-γ and b) lipopolysaccharide…”
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    Serum concentrations of trimethoprim and sulfadiazine following oral paste administration to the horse by Sigel, C W, Byars, T D, Divers, T J, Murch, O, DeAngelis, D

    Published in American journal of veterinary research (01-01-1981)
    “…Two fasted and 2 fed horses were dosed orally with a combined trimethoprim and sulfadiazine paste formulation at a dose of 35 mg (1:5 combined active…”
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  6. 6

    Experimental induction of Proteus mirabilis cystitis in the pony and evaluation of therapy with trimethoprim-sulfadiazine by Divers, T J, Byars, T D, Murch, O, Sigel, C W

    Published in American journal of veterinary research (01-01-1981)
    “…Proteus mirabilis cystitis was induced in 9 ponies by chemically eroding the bladder mucosa before the organism was inoculated. Comparisons were made in the…”
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    Thin layer chromatographic screening method for sulfadiazine residues in calf tissues, plasma, and urine by Woolley, Jr, J L, Murch, O, Sigel, C S

    “…Because of the lack of specificity of the Bratton-Marshall procedure for assaying sulfonamides, a sensitive, specific tissue residue assay for sulfadiazine…”
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