Search Results - "Munday, Hannah"

Investigation of gene‐environment interactions between 47 newly identified breast cancer susceptibility loci and environmental risk factors by Rudolph, Anja, Milne, Roger L., Truong, Thérèse, Knight, Julia A., Seibold, Petra, Flesch‐Janys, Dieter, Behrens, Sabine, Eilber, Ursula, Bolla, Manjeet K., Wang, Qin, Dennis, Joe, Dunning, Alison M., Shah, Mitul, Munday, Hannah R., Darabi, Hatef, Eriksson, Mikael, Brand, Judith S., Olson, Janet, Vachon, Celine M., Hallberg, Emily, Castelao, J. Esteban, Carracedo, Angel, Torres, Maria, Li, Jingmei, Humphreys, Keith, Cordina‐Duverger, Emilie, Menegaux, Florence, Flyger, Henrik, Nordestgaard, Børge G., Nielsen, Sune F., Yesilyurt, Betul T., Floris, Giuseppe, Leunen, Karin, Engelhardt, Ellen G., Broeks, Annegien, Rutgers, Emiel J., Glendon, Gord, Mulligan, Anna Marie, Cross, Simon, Reed, Malcolm, Gonzalez‐Neira, Anna, Arias Perez, José Ignacio, Provenzano, Elena, Apicella, Carmel, Southey, Melissa C., Spurdle, Amanda, Häberle, Lothar, Beckmann, Matthias W., Ekici, Arif B., Dieffenbach, Aida Karina, Arndt, Volker, Stegmaier, Christa, McLean, Catriona, Baglietto, Laura, Chanock, Stephen J., Lissowska, Jolanta, Sherman, Mark E., Brüning, Thomas, Hamann, Ute, Ko, Yon‐Dschun, Orr, Nick, Schoemaker, Minouk, Ashworth, Alan, Kosma, Veli‐Matti, Kataja, Vesa, Hartikainen, Jaana M., Mannermaa, Arto, Swerdlow, Anthony, Giles, Graham G., Brenner, Hermann, Fasching, Peter A., Chenevix‐Trench, Georgia, Hopper, John, Benítez, Javier, Cox, Angela, Andrulis, Irene L., Lambrechts, Diether, Gago‐Dominguez, Manuela, Couch, Fergus, Czene, Kamila, Bojesen, Stig E., Easton, Doug F., Schmidt, Marjanka K., Guénel, Pascal, Hall, Per, Pharoah, Paul D. P., Garcia‐Closas, Montserrat, Chang‐Claude, Jenny

“…A large genotyping project within the Breast Cancer Association Consortium (BCAC) recently identified 41 associations between single nucleotide polymorphisms…”
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Association between common variation in 120 candidate genes and breast cancer risk by Pharoah, Paul D P, Tyrer, Jonathan, Dunning, Alison M, Easton, Douglas F, Ponder, Bruce A J

“…Association studies in candidate genes have been widely used to search for common low penetrance susceptibility alleles, but few definite associations have…”
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IGF1 and IGFBP3 tagging polymorphisms are associated with circulating levels of IGF1, IGFBP3 and risk of breast cancer by Al-Zahrani, Ali, Sandhu, Manjinder S., Luben, Robert N., Thompson, Deborah, Baynes, Caroline, Pooley, Karen A., Luccarini, Craig, Munday, Hannah, Perkins, Barbara, Smith, Paula, Pharoah, Paul D.P., Wareham, Nicholas J., Easton, Douglas F., Ponder, Bruce A.J., Dunning, Alison M.

“…Experimental and observational studies in humans and animals suggest that insulin-like growth factor 1 (IGF1) and its principal binding protein, IGFBP3, may…”
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Association of single-nucleotide polymorphisms in the cell cycle genes with breast cancer in the British population by Driver, Kristy E., Song, Honglin, Lesueur, Fabienne, Ahmed, Shahana, Barbosa-Morais, Nuno L., Tyrer, Jonathan P., Ponder, Bruce A.J., Easton, Douglas F., Pharoah, Paul D.P., Dunning, Alison M.

“…Using a large-scale case–control study, we examined whether common single-nucleotide polymorphisms (SNPs) within 13 genes involved in the cell cycle pathway…”
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Common variants in the ATM, BRCA1, BRCA2, CHEK2 and TP53 cancer susceptibility genes are unlikely to increase breast cancer risk by Baynes, Caroline, Healey, Catherine S, Pooley, Karen A, Scollen, Serena, Luben, Robert N, Thompson, Deborah J, Pharoah, Paul D P, Easton, Douglas F, Ponder, Bruce A J, Dunning, Alison M

“…Certain rare, familial mutations in the ATM, BRCA1, BRCA2, CHEK2 or TP53 genes increase susceptibility to breast cancer but it has not, until now, been clear…”
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Tagging single-nucleotide polymorphisms in antioxidant defense enzymes and susceptibility to breast cancer by CEBRIAN, Arancha, PHAROAH, Paul D, PONDER, Bruce A. J, AHMED, Shahana, SMITH, Paula L, LUCCARINI, Craig, LUBEN, Robert, REDMAN, Karen, MUNDAY, Hannah, EASTON, Douglas F, DUNNING, Alison M

“…It is generally believed that the initiation of breast cancer is a consequence of cumulative genetic damage leading to genetic alterations and provoking…”
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