Search Results - "Mullins, R.J."

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  1. 1

    Further Evidence For Tick Bites As A Cause Of The IgE Responses To Alpha-gal That Underlie A Major Increased In Delayed Anaphylaxis To Meat by James, H.R, Commins, S.P, Kelly, L.A, Pochan, S.L, Workman, L.J, Mullins, R.J, Platts-Mills, T.A.E

    Published in Journal of allergy and clinical immunology (01-02-2011)
    “…The IgE responses following tick bites have distinctive features: a) they can increase to high level (i.e., greater than 50 IU/ml within a month), b) IgE to…”
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    Journal Article
  2. 2

    Season of Birth is Associated with Risk of Food Allergy in Children by Vassallo, M.F, Banerji, A, Rudders, S.A, Clark, S, Mullins, R.J, Camargo, C.A

    Published in Journal of allergy and clinical immunology (01-02-2010)
    “…Season of birth among food allergy patients was compared to three control groups: children visiting ED for other reasons, and all Boston and Massachusetts…”
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    Journal Article
  3. 3

    Cold urticaria: a 20-year follow-up study by Jain, S.V., Mullins, R.J.

    “…Background Chronic cold urticaria results in significant morbidity, yet information on its natural history is limited. Objective We examined the natural…”
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    Journal Article
  4. 4

    Ketone Ester Effects on Biomarkers of Brain Metabolism and Cognitive Performance in Cognitively Intact Adults ≥ 55 Years Old. A Study Protocol for a Double-Blinded Randomized Controlled Clinical Trial by Avgerinos, K. I., Mullins, R. J., Egan, J. M., Kapogiannis, Dimitrios

    “…Background Ketone bodies have been proposed as an “energy rescue” for the Alzheimer’s disease (AD) brain, which underutilizes glucose. Prior research has shown…”
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    Isolation and structural characteristics of a monoclonal antibody-defined cross-reactive phospholipid antigen from Mycobacterium tuberculosis and Mycobacterium leprae by Fournié, J J, Mullins, R J, Basten, A

    Published in The Journal of biological chemistry (15-01-1991)
    “…A low molecular weight antigen of Mycobacterium leprae and other mycobacteria was previously defined in our laboratory by means of IgG2a monoclonal antibody…”
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    Journal Article