Search Results - "Militello, Anna Maria"

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  1. 1

    Mechanism of Action and Clinical Efficacy of CDK4/6 Inhibitors in BRCA-Mutated, Estrogen Receptor-Positive Breast Cancers: Case Report and Literature Review by Militello, Anna Maria, Zielli, Teresa, Boggiani, Daniela, Michiara, Maria, Naldi, Nadia, Bortesi, Beatrice, Zanelli, Paola, Uliana, Vera, Giuliotti, Sara, Musolino, Antonino

    Published in Frontiers in oncology (13-08-2019)
    “…Sensitivity to endocrine therapy of patients with estrogen receptor (ER)-positive metastatic breast cancer and germline mutations is not yet fully elucidated…”
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    Journal Article
  2. 2

    Metronomic oral cyclophosphamide as maintenance therapy in metastatic pancreatic ductal adenocarcinoma by Reni, Michele, Orsi, Giulia, Peretti, Umberto, Macchini, Marina, Militello, Anna Maria, Falconi, Massimo, Cascinu, Stefano

    Published in Journal of clinical oncology (01-02-2023)
    “…716 Background: Metastatic Pancreatic Ductal Adenocarcinoma (mPDAC) has a grim prognosis, with limited therapeutic options. In this context, treating patients…”
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    Journal Article
  3. 3

    Treatment opportunities and future perspectives for pancreatic cancer patients with germline BRCA1-2 pathogenic variants by Macchini, Marina, Centonze, Federico, Peretti, Umberto, Orsi, Giulia, Militello, Anna Maria, Valente, Maria Maddalena, Cascinu, Stefano, Reni, Michele

    Published in Cancer treatment reviews (01-11-2021)
    “…•Personalized treatments and predictive biomarkers of pancreatic cancer (PDAC) are still lacking.•BRCA1-2 alterations increase sensitivity to platinum salts…”
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    Journal Article
  4. 4

    Epidemiology and geographic distribution of BRCA1-2 and DNA Damage response genes pathogenic variants in pancreatic ductal adenocarcinoma patients by Macchini, Marina, Centonze, Federico, Peretti, Umberto, Orsi, Giulia, Militello, Anna Maria, Valente, Maria Maddalena, Cascinu, Stefano, Reni, Michele

    Published in Cancer treatment reviews (01-03-2022)
    “…•BRCA1-2 mutations (gBRCA1-2) are responsible for PDAC in 15–20% of familiar cases.•gBRCA1-2 and DDR genes mutations (gDDR) emerged as therapeutic targets for…”
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    Journal Article
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