Search Results - "Maschmeyer, P."

T-bet expression by Th cells promotes type 1 inflammation but is dispensable for colitis by Zimmermann, J, Kühl, A A, Weber, M, Grün, J R, Löffler, J, Haftmann, C, Riedel, R, Maschmeyer, P, Lehmann, K, Westendorf, K, Mashreghi, M-F, Löhning, M, Mack, M, Radbruch, A, Chang, H D

“…The transcription factor T-bet is highly expressed by Th cells isolated from the inflamed intestine of Crohn’s disease patients, and has been regarded a…”
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Depletion of pro-inflammatory T helper type 1 cells by antagomir-mediated inhibition of the microRNA-148a by Maschmeyer, P, Mashreghi, M-F

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CTLA-4 (CD152) blockade does not cause a pro-inflammatory cytokine profile in regulatory T cells by KOLAR, P, HOFF, H, MASCHMEYER, P, BURMESTER, G.-R, BRUNNER-WEINZIERL, M. C

“…The activation of T cells is closely regulated. One cell intrinsic mechanism is based on the expression of inhibitory molecules; another is mediated by…”
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Effects of celecoxib on the expression of osteoprotegerin, energy metabolism and cell viability in cultured human osteoblastic cells by KOLAR, P, LACH, S, BUTTGEREIT, F, GABER, T, MASCHMEYER, P, DZIURLA, R, TRIPMACHER, R, KROCKER, D, MATZIOLIS, G, PERKA, C, BURMESTER, G.-R

“…The selective COX-2 inhibitor celecoxib is widely used to treat pain and inflammation in rheumatoid arthritis, osteoarthritis, and ankylosing spondylitis. The…”
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Selektive Depletion proinflammatorischer Typ-1-T-Helfer-Zellen mit mikroRNA-148a-spezifischen Antagomirs by Maschmeyer, P., Mashreghi, M.-F.

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Human immune cells＇ behavior and survival under bioenergetically restricted conditions in an in vitro fracture hematoma model by Hoff, Paula, Maschmeyer, Patrick, Gaber, Timo, Schütze, Tabea, Raue, Tobias, Schmidt-Bleek, Katharina, Dziurla, René, Schellmann, Saskia, Lohanatha, Ferenz Leonard, Röhner, Eric, Ode, Andrea, Burmester, Gerd-Rüdiger, Duda, Georg N, Perka, Carsten, Buttgereit, Frank

“…The initial inflammatory phase of bone fracture healing represents a critical step for the outcome of the healing process. However, both the mechanisms…”
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