Search Results - "Martens, Leonie"

Interleukin 17 Promotes Expression of Alarmins S100A8 and S100A9 During the Inflammatory Response of Keratinocytes by Christmann, Carolin, Zenker, Stefanie, Martens, Leonie, Hübner, Janina, Loser, Karin, Vogl, Thomas, Roth, Johannes

“…Psoriasis is one of the most common immune-mediated inflammatory skin diseases. Expression and secretion of two pro-inflammatory molecules of the S100-alarmin…”
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Genomic instability in the naturally and prematurely aged myocardium by De Majo, Federica, Martens, Leonie, Hegenbarth, Jana-Charlotte, Rühle, Frank, Hamczyk, Magda R., Nevado, Rosa M., Andrés, Vicente, Hilbold, Erika, Bär, Christian, Thum, Thomas, de Boer, Martine, Duncker, Dirk J., Schroen, Blanche, Armand, Anne-Sophie, Stoll, Monika, De Windt, Leon J.

“…Genomic instability, the unresolved accumulation of DNA variants, is hypothesized as one of the contributors to the natural aging process. We assessed the…”
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Monocyte subpopulation profiling indicates CDK6-derived cell differentiation and identifies subpopulation-specific miRNA expression sets in acute and stable coronary artery disease by Witten, Anika, Martens, Leonie, Schäfer, Ann-Christin, Troidl, Christian, Pankuweit, Sabine, Vlacil, Ann-Kathrin, Oberoi, Raghav, Schieffer, Bernhard, Grote, Karsten, Stoll, Monika, Markus, Birgit

“…Coronary artery disease (CAD) is a long-lasting inflammatory disease characterized by monocyte migration into the vessel wall leading to clinical events like…”
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LncRNA secondary structure in the cardiovascular system by Martens, Leonie, Rühle, Frank, Stoll, Monika

“…Long non-coding RNAs (lncRNAs) have been increasingly studied during the past decade. This led to an immense number of annotated transcripts, out of which many…”
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C/EBPδ-induced epigenetic changes control the dynamic gene transcription of S100a8 and S100a9 by Jauch-Speer, Saskia-Larissa, Herrera-Rivero, Marisol, Ludwig, Nadine, Véras De Carvalho, Bruna Caroline, Martens, Leonie, Wolf, Jonas, Imam Chasan, Achmet, Witten, Anika, Markus, Birgit, Schieffer, Bernhard, Vogl, Thomas, Rossaint, Jan, Stoll, Monika, Roth, Johannes, Fehler, Olesja

“…The proinflammatory alarmins S100A8 and S100A9 are among the most abundant proteins in neutrophils and monocytes but are completely silenced after…”
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A genetic variant alters the secondary structure of the lncRNA H19 and is associated with dilated cardiomyopathy by Martens, Leonie, Rühle, Frank, Witten, Anika, Meder, Benjamin, Katus, Hugo A., Arbustini, Eloisa, Hasenfuß, Gerd, Sinner, Moritz F., Kääb, Stefan, Pankuweit, Sabine, Angermann, Christiane, Bornberg-Bauer, Erich, Stoll, Monika

“…lncRNAs are at the core of many regulatory processes and have also been recognized to be involved in various complex diseases. They affect gene regulation…”
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DNA repair in cardiomyocytes is critical for maintaining cardiac function in mice by Boer, Martine, Lintel Hekkert, Maaike, Chang, Jiang, Thiel, Bibi S., Martens, Leonie, Bos, Maxime M., Kleijnen, Marion G. J., Ridwan, Yanto, Octavia, Yanti, Deel, Elza D., Blonden, Lau A., Brandt, Renata M. C., Barnhoorn, Sander, Bautista‐Niño, Paula K., Krabbendam‐Peters, Ilona, Wolswinkel, Rianne, Arshi, Banafsheh, Ghanbari, Mohsen, Kupatt, Christian, Windt, Leon J., Danser, A. H. Jan, Pluijm, Ingrid, Remme, Carol Ann, Stoll, Monika, Pothof, Joris, Roks, Anton J. M., Kavousi, Maryam, Essers, Jeroen, Velden, Jolanda, Hoeijmakers, Jan H. J., Duncker, Dirk J.

“…Heart failure has reached epidemic proportions in a progressively ageing population. The molecular mechanisms underlying heart failure remain elusive, but…”
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C/EBP[delta]-induced epigenetic changes control the dynamic gene transcription of S100a8 and S100a9 by Jauch-Speer, Saskia-Larissa, Herrera-Rivero, Marisol, Ludwig, Nadine, Véras De Carvalho, Bruna Caroline, Martens, Leonie, Wolf, Jonas, Imam Chasan, Achmet, Witten, Anika, Markus, Birgit, Schieffer, Bernhard, Vogl, Thomas, Rossaint, Jan, Stoll, Monika, Roth, Johannes, Fehler, Olesja

“…The proinflammatory alarmins S100A8 and S100A9 are among the most abundant proteins in neutrophils and monocytes but are completely silenced after…”
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