Search Results - "Mansur, Daniel E."

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    KATP channels modulate cerebral blood flow and oxygen delivery during isocapnic hypoxia in humans by Rocha, Marcos P., Campos, Monique O., Mattos, João D., Mansur, Daniel E., Rocha, Helena N. M., Secher, Niels H., Nóbrega, Antonio C. L., Fernandes, Igor A.

    Published in The Journal of physiology (01-08-2020)
    “…Key points ATP‐sensitive K+ (KATP) channels mediate hypoxia‐induced cerebral vasodilatation and hyperperfusion in animals. We tested whether KATP channels…”
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    Journal Article
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    Reactive oxygen species play a modulatory role in the hyperventilatory response to poikilocapnic hyperoxia in humans by Fernandes, Igor A., Mattos, João D., Campos, Monique O., Rocha, Marcos P., Mansur, Daniel E., Rocha, Helena M., Garcia, Vinicius P., Alvares, Thiago, Secher, Niels H., Nóbrega, Antonio C.L.

    Published in The Journal of physiology (01-08-2021)
    “…Key points The proposed mechanism for the increased ventilation in response to hyperoxia includes a reduced brain CO2–[H+] washout‐induced central…”
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    Journal Article
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    Acid-sensing ion channels blockade attenuates pressor and sympathetic responses to skeletal muscle metaboreflex activation in humans by Campos, Monique O, Mansur, Daniel E, Mattos, João D, Paiva, Adrielle C S, Videira, Rogerio L R, Macefield, Vaughan G, da Nóbrega, Antonio C L, Fernandes, Igor A

    Published in Journal of applied physiology (1985) (01-11-2019)
    “…In animals, the blockade of acid-sensing ion channels (ASICs), cation pore-forming membrane proteins located in the free nerve endings of group IV afferent…”
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    Journal Article
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    Hypertension impairs hypoxia-induced angiogenesis in men by Garcia, Vinicius P, Rocha, Helena N M, Rocha, Marcos P, Mattos, João D, Campos, Monique O, Mansur, Daniel E, Secher, Niels H, Nóbrega, Antonio C L, Fernandes, Igor A, Rocha, Natália G

    Published in Journal of hypertension (01-06-2020)
    “…The inability of the organism to appropriately respond to hypoxia results in abnormal cell metabolism and function. Hypoxia-induced angiogenesis seems to be…”
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    Journal Article
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    K ATP channels modulate cerebral blood flow and oxygen delivery during isocapnic hypoxia in humans by Rocha, Marcos P, Campos, Monique O, Mattos, João D, Mansur, Daniel E, Rocha, Helena N M, Secher, Niels H, Nóbrega, Antonio C L, Fernandes, Igor A

    Published in The Journal of physiology (01-08-2020)
    “…ATP-sensitive K (K ) channels mediate hypoxia-induced cerebral vasodilatation and hyperperfusion in animals. We tested whether K channels blockade affects the…”
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    Journal Article
  9. 9

    Hypertension impairs hypoxia-induced angiogenesis in men by Garcia, Vinicius P., Rocha, Helena N.M., Rocha, Marcos P., Mattos, João D., Campos, Monique O., Mansur, Daniel E., Secher, Niels H., Nóbrega, Antonio C.L., Fernandes, Igor A., Rocha, Natália G.

    Published in Journal of hypertension (01-06-2020)
    “…OBJECTIVE:The inability of the organism to appropriately respond to hypoxia results in abnormal cell metabolism and function. Hypoxia-induced angiogenesis…”
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    Journal Article
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    Effects of face cooling on pulse waveform and sympathetic activity in hypertensive subjects by Prodel, Eliza, Barbosa, Thales C., Mansur, Daniel E., Nóbrega, Antonio Claudio L., Vianna, Lauro C.

    Published in Clinical autonomic research (01-02-2017)
    “…Adverse cardiovascular events occur more frequently during cold weather. To test the hypothesis that cold exposure would lead to increased sympathetic activity…”
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    Journal Article
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    Acid‐Sensing Ion Channels Blockade Attenuates Pressor and Sympathetic Responses to Skeletal Muscle Metaboreflex Activation in Humans by Fernandes, Igor Alexandre, Campos, Monique O, Mansur, Daniel E, Mattos, João, Paiva, Adrielle, Videira, Rogerio, Macefield, Vaughan, Nóbrega, Antonio CL

    Published in The FASEB journal (01-04-2019)
    “…In animals, the blockade of acid‐sensing ion channels (ASICS), cation pore‐forming membrane proteins located in the free nerve endings of group IV afferent…”
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    Journal Article
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