Search Results - "Manoj, M. Jeru"

CDKN2A exon-wise deletion status and novel somatic mutations in Indian glioma patients by Sibin, M. K., Bhat, Dhananjaya I., Lavanya, Ch, Jeru Manoj, M., Aakershita, S., Chetan, G. K.

“…Over the years, deletions of CDKN2A (p16) tumor suppressor gene has been studied using FISH and multiplex PCR, with major focus on exon 2 in various cancers,…”
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Environmental pollutants leading to carcinogenesis: process of natural selection of human cells due to chronic inflammation and sustained stress environment by Venkatesh, H. N, Jeru Manoj, M, Ghosh, D, Chetan, G. K

“…Epidemiological studies have shown that 70-90 % of all cancers are caused due to the chemicals present in environment. Exogenous exposure to carcinogens and…”
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RNA interference mediated downregulation of human telomerase reverse transcriptase (hTERT) in LN18 cells by Lavanya, Ch, Sibin, M. K., Srinivas Bharath, M. M., Manoj, M. Jeru, Venkataswamy, Manjunatha M., Bhat, Dhananjaya I., Narasinga Rao, K. V. L., Chetan, G. K.

“…Human telomerase reverse transcriptase (hTERT) gene is a biomarker for the targeted therapy in various cancers. Presence of increased telomerase activity is a…”
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Correlation o P16 and Bmi1 gene expression in human high grade glioma by Sibin, M K, Chetan, GK, Lavanya, Ch, Jeru, Manoj M, Bhat, Dhananjaya I

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Promoter methylation of PTEN gene in high grade gliomas by Lavanya, C, Chetan, GK, Sibin, MK, Jeru, Manoj M, Srinivas, Bharath MM

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Role of p16 deletion and Bmi1 copy number variation in glioma by Chetan, GK, Sibin, M K, Bhat, Dhananjaya I, Lavanya, Ch, Jeru, Manoj M, Geethashree, N

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The role of direct DNA repair gene O6-methylguanine-DNA methyltransferase (MGMT) in high grade malignant glioma by Manoj, M Jeru, Chetan, G K, Rao, KVL Narasinga, Venkatesh, HN, Sibin, MK, Lavanya, Ch

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Two gene polymorphisms (rs4977756 and rs11515) in CDKN2A/B and glioma risk in South Indian population by Sibin, M.K., Dhananjaya, I. Bhat, Narasingarao, K.V.L., Harshitha, S.M., Jeru-Manoj, M., Chetan, G.K.

“…Gliomas are most common neoplasms in the CNS with unknown aetiology. Gene polymorphisms have been studied in glioma to check its risk in different population…”
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