Search Results - "Magill, MJ"

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  1. 1

    Adenosine activates A2 receptors to inhibit neutrophil adhesion and injury to isolated cardiac myocytes by Bullough, DA, Magill, MJ, Firestein, GS, Mullane, KM

    Published in The Journal of immunology (1950) (01-09-1995)
    “…Inhibition of neutrophil-myocyte adhesion and adhesion-dependent myocyte injury by adenosine was evaluated using isolated TNF-alpha-activated canine cells…”
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    Journal Article
  2. 2

    Triptycenes: a novel synthetic class of bifunctional anticancer drugs that inhibit nucleoside transport, induce DNA cleavage and decrease the viability of leukemic cells in the nanomolar range in vitro by Perchellet, E M, Magill, M J, Huang, X, Brantis, C E, Hua, D H, Perchellet, J P

    Published in Anti-cancer drugs (01-09-1999)
    “…In contrast to their inactive parent compound triptycene (code name TT0), several triptycene (TT) analogs (code names TT1 to TT13), most of them new compounds,…”
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    Journal Article
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    Adenosine activates A sub(2) receptors to inhibit neutrophil adhesion and injury to isolated cardiac myocytes by Bullough, DA, Magill, MJ, Firestein, G S, Mullane, K M

    Published in The Journal of immunology (1950) (01-01-1995)
    “…Inhibition of neutrophil-myocyte adhesion and adhesion-dependent myocyte injury by adenosine was evaluated using isolated TNF- alpha -activated canine cells…”
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    Journal Article
  6. 6

    Carbohydrate- and CD18-dependent neutrophil adhesion to cardiac myocytes: effects of adenosine by Bullough, David A., Magill, Madeleine J., Mullane, Kevin M., Firestein, Gary S.

    Published in Cardiovascular research (01-08-1996)
    “…Objective: Adenosine inhibits neutrophil adhesion and injury to isolated cardiac myocytes. In the present study, the contribution of selectin and CD 18…”
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    Journal Article