Search Results - "Machkova, Martina"

An unusually high frequency of SCAD deficiency caused by two pathogenic variants in the ACADS gene and its relationship to the ethnic structure in Slovakia by Lisyová, Jana, Chandoga, Ján, Jungová, Petra, Repiský, Marcel, Knapková, Mária, Machková, Martina, Dluholucký, Svetozár, Behúlová, Darina, Šaligová, Jana, Potočňáková, Ľudmila, Lysinová, Miroslava, Böhmer, Daniel

“…Short-chain acyl-CoA dehydrogenase deficiency (SCADD) represents a rare autosomal recessive inborn metabolic disorder of mitochondrial β-oxidation of…”
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Phase 2b randomized trial of OX40 inhibitor telazorlimab for moderate-to-severe atopic dermatitis by Rewerska, Barbara, Sher, Lawrence D., Alpizar, Sady, Pauser, Sylvia, Pulka, Grazyna, Mozaffarian, Neelufar, Salhi, Yacine, Martinet, Camille, Jabert, Wafaa, Gudi, Girish, CA, Vinu, GN, Sunitha, Macoin, Julie, Anstett, Victor, Turrini, Riccardo, Doucey, Marie-Agnès, Blein, Stanislas, Konto, Cyril, Machkova, Martina

“…Telazorlimab is a humanized anti-OX40 monoclonal antibody being studied for treatment of T-cell–mediated diseases. This randomized, placebo-controlled, phase…”
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Early Onset of Efficacy With Fremanezumab in Patients With Medication Overuse and Documented Inadequate Response to 2–4 Classes of Migraine Preventive Treatments: Subgroup Analysis of the Randomized, Double-blind FOCUS Study (4400) by Katsarava, Zaza, Machkova, Martina, Ramirez-Campos, Verena, Cohen, Joshua M., Janka, Lindsay, Spierings, Egilius

“…Abstract only…”
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Cendakimab in Patients With Moderate to Severe Atopic Dermatitis: A Randomized Clinical Trial by Blauvelt, Andrew, Guttman-Yassky, Emma, Lynde, Charles, Khattri, Saakshi, Schlessinger, Joel, Imafuku, Shinichi, Tada, Yayoi, Morita, Akimichi, Wiseman, Marni, Kwiek, Bartlomiej, Machkova, Martina, Zhang, Peijin, Linaberry, Misti, Li, Jie, Zhang, Sandra, Franchin, Giovanni, Charles, Edgar D, De Oliveira, Claudia H M C, Silverberg, Jonathan I

“…Cendakimab selectively targets interleukin (IL)-13, a type 2 cytokine implicated in atopic dermatitis (AD) pathogenesis, by inhibiting binding to its receptors…”
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