Search Results - "MEDJANE, Samir"
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1
Involvement of Toll-like Receptor 5 in the Recognition of Flagellated Bacteria
Published in Proceedings of the National Academy of Sciences - PNAS (15-08-2006)“…Toll-like receptors (TLRs) are key components of the immune system that detect microbial infection and trigger antimicrobial host defense responses. TLR5 is a…”
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2
Familial partial lipodystrophy type 2 and obesity, two adipose tissue pathologies with different inflammatory profiles
Published in Diabetology and metabolic syndrome (21-04-2023)“…The transition to metabolically unhealthy obesity (MUO) is driven by the limited expandability of adipose tissue (AT). Familial Partial Lipodystrophy type 2…”
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3
Response of Human Pulmonary Epithelial Cells to Lipopolysaccharide Involves Toll-like Receptor 4 (TLR4)-dependent Signaling Pathways
Published in The Journal of biological chemistry (01-01-2004)“…Pulmonary epithelial cells are continuously exposed to microbial challenges as a result of breathing. It is recognized that immune myeloid cells express…”
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4
A recurrent familial partial lipodystrophy due to a monoallelic or biallelic LMNA founder variant highlights the multifaceted cardiac manifestations of metabolic laminopathies
Published in European journal of endocrinology (01-10-2021)“…Aims LMNA-linked familial partial lipodystrophy type 2 (FPLD2) leads to insulin resistance-associated metabolic complications and cardiovascular diseases. We…”
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5
Response of human pulmonary epithelial cells to lipopolysaccharide involves Toll-like receptor 4 (TLR4)-dependent signaling pathways: evidence for an intracellular compartmentalization of TLR4
Published in The Journal of biological chemistry (23-01-2004)“…Pulmonary epithelial cells are continuously exposed to microbial challenges as a result of breathing. It is recognized that immune myeloid cells express…”
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Journal Article -
6
Involvement of Toll-like receptor S in the recognition of flagellated bacteria
Published in Proceedings of the National Academy of Sciences - PNAS (15-08-2006)“…Toll-like receptors (TLRs) are key components of the immune system that detect microbial infection and trigger antimicrobial host defense responses. TLRs is a…”
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Journal Article -
7
Surfactant protein-A and phosphatidylglycerol suppress type IIA phospholipase A2 synthesis via nuclear factor-κB
Published in American journal of respiratory and critical care medicine (15-09-2003)“…We previously showed that surfactant inhibits the synthesis of type IIA secretory phospholipase A2 (sPLA2-IIA) by alveolar macrophages. These cells have been…”
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8
Impact of CFTR DeltaF508 mutation on prostaglandin E2 production and type IIA phospholipase A2 expression by pulmonary epithelial cells
Published in American journal of physiology. Lung cellular and molecular physiology (01-11-2005)“…Cystic fibrosis (CF) is characterized by an exacerbated inflammatory pulmonary response with excessive production of inflammatory mediators. We investigated…”
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9
Transcription Factor 3′,5′-Cyclic Adenosine 5′-Monophosphate-Responsive Element-Binding Protein (CREB) Is Decreased during Human Adrenal Cortex Tumorigenesis and Fetal Development
Published in The journal of clinical endocrinology and metabolism (01-08-2003)“…Various cellular and molecular alterations of the cAMP pathway have been observed in adrenal Cushing syndrome. We recently reported the loss of cAMP-responsive…”
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10
Surfactant protein-A and phosphatidylglycerol suppress type IIA phospholipase A2 synthesis via nuclear factor-kappaB
Published in American journal of respiratory and critical care medicine (15-09-2003)“…We previously showed that surfactant inhibits the synthesis of type IIA secretory phospholipase A2 (sPLA2-IIA) by alveolar macrophages. These cells have been…”
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Journal Article -
11
Surfactant Protein-A and Phosphatidylglycerol Suppress Type IIA Phospholipase A2 Synthesis via Nuclear Factor-{kappa}B
Published in American journal of respiratory and critical care medicine (15-09-2003)“…We previously showed that surfactant inhibits the synthesis of type IIA secretory phospholipase A2 (sPLA2-IIA) by alveolar macrophages. These cells have been…”
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