Search Results - "Lobitz, Nicole"
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1
Phthalic acid diamides activate ryanodine-sensitive Ca2+ release channels in insects
Published in Cell calcium (Edinburgh) (01-01-2006)“…Flubendiamide represents a novel chemical family of substituted phthalic acid diamides with potent insecticidal activity. So far, the molecular target and the…”
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2
Molecular cloning, functional expression, and pharmacological characterization of 5-hydroxytryptamine3 receptor cDNA and its splice variants from guinea pig
Published in Molecular pharmacology (01-02-1998)“…Polymerase chain reaction and rapid amplification of cDNA ends were used to isolate cDNAs encoding a 5-hydroxytryptamine3 (5-HT3) receptor subunit and its…”
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3
Evaluation of thoracic impedance trends for implant-based remote monitoring in heart failure patients - Results from the (J-)HomeCARE-II Study
Published in Journal of electrocardiology (01-03-2019)“…Remote monitoring by implantable devices substantially improves management of heart failure (HF) patients by providing diagnostic day-to-day data. The use of…”
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4
Phthalic acid diamides activate ryanodine-sensitive Ca super(2+) release channels in insects
Published in Cell calcium (Edinburgh) (01-01-2006)“…Flubendiamide represents a novel chemical family of substituted phthalic acid diamides with potent insecticidal activity. So far, the molecular target and the…”
Get full text
Journal Article -
5
Phthalic acid diamides activate ryanodine-sensitive Ca 2+ release channels in insects
Published in Cell calcium (Edinburgh) (2006)“…Flubendiamide represents a novel chemical family of substituted phthalic acid diamides with potent insecticidal activity. So far, the molecular target and the…”
Get full text
Journal Article -
6
A single amino-acid in the TM1 domain is an important determinant of the desensitization kinetics of recombinant human and guinea pig alpha-homomeric 5-hydroxytryptamine type 3 receptors
Published in Molecular pharmacology (01-04-2001)“…Desensitization of ligand-gated ion channels shapes synaptic responses and provides critical neuroprotection at central synapses, yet the molecular mechanisms…”
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