Search Results - "Lobie, P.E"

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    Trefoil factor 1 suppression of E-CADHERIN enhances prostate carcinoma cell invasiveness and metastasis by Bougen, N.M, Amiry, N, Yuan, Y, Kong, X.J, Pandey, V, Vidal, L.J.P, Perry, J.K, Zhu, T, Lobie, P.E

    Published in Cancer letters (10-05-2013)
    “…Highlights ► TFF1 enhances prostate carcinoma cell migration and invasion. ► TFF1 reduces expression of E-CADHERIN in prostate carcinoma cells and E-CADHERIN…”
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    Growth hormone (GH) induction of tyrosine phosphorylation and activation of mitogen-activated protein kinases in cells transfected with rat GH receptor cDNA by Moller, C. (Center for Biotechnology, Novum, Huddinge, Sweden), Hansson, A, Enberg, B, Lobie, P.E, Norstedt, G

    Published in The Journal of biological chemistry (15-11-1992)
    “…The mechanism of growth hormone (GH) action was studied in Chinese hamster ovary (CHO) cells transfected with GH receptor cDNA. Cytosolic extracts from GH- or…”
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    Receptor-mediated nuclear translocation of growth hormone by LOBIE, P. E, MERTANI, H, MOREL, G, MORALES-BUSTOS, O, NORSTEDT, G, WATERS, M. J

    Published in The Journal of biological chemistry (19-08-1994)
    “…We have previously shown that the growth hormone (GH) receptor-binding protein is associated with the nucleus. We show here both by electron microscopy and…”
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    Artemin is oncogenic for human mammary carcinoma cells by Kang, J, Perry, J K, Pandey, V, Fielder, G C, Mei, B, Qian, P X, Wu, Z S, Zhu, T, Liu, D X, Lobie, P E

    Published in Oncogene (14-05-2009)
    “…We report that artemin, a member of the glial cell line-derived neurotrophic factor family of ligands, is oncogenic for human mammary carcinoma. Artemin is…”
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    Phenotypic Conversion of Human Mammary Carcinoma Cells by Autocrine Human Growth Hormone by Mukhina, Svetlana, Mertani, Hichem C., Guo, Ke, Lee, Kok-Onn, Gluckman, Peter D., Lobie, Peter E., Grumbach, Melvin M.

    “…We report here that autocrine production of human growth hormone (hGH) results in a phenotypic conversion of mammary carcinoma cells such that they exhibit the…”
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    Artemin is estrogen regulated and mediates antiestrogen resistance in mammary carcinoma by Kang, J, Qian, P X, Pandey, V, Perry, J K, Miller, L D, Liu, E T, Zhu, T, Liu, D X, Lobie, P E

    Published in Oncogene (03-06-2010)
    “…We have previously identified an oncogenic role of artemin (ARTN), a member of glial cell derived neurotrophic factor family of ligands, in mammary carcinoma…”
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    Oncogenic transformation of human mammary epithelial cells by autocrine human growth hormone by TAO ZHU, EMERALD, B. Starling, XIN ZHANG, LEE, Kok-Onn, GLUCKMAN, Peter D, MERTANI, Hichem C, LOBIE, Peter E

    Published in Cancer research (Chicago, Ill.) (2005)
    “…The human growth hormone (hGH) gene is expressed in the normal human mammary epithelial cell and its expression increases concomitant with the acquisition of…”
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    DNMT3A and DNMT3B mediate autocrine hGH repression of plakoglobin gene transcription and consequent phenotypic conversion of mammary carcinoma cells by Shafiei, F, Rahnama, F, Pawella, L, Mitchell, M D, Gluckman, P D, Lobie, P E

    Published in Oncogene (17-04-2008)
    “…Directed by microarray analyses, we report that autocrine human growth hormone (hGH) increased the mRNA and protein expression of DNA methyltransferase 1…”
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    PO-482 Efficacy of inhibition of BAD Ser99 phosphorylation by a novel small molecule in cisplatin resistant ovarian cancer by Wang, Y, Chong, QY, Pandey, V, Basappa, S, Choolani, M, Huang, RYJ, Lobie, PE

    Published in ESMO open (01-07-2018)
    “…IntroductionHuman BAD (hBAD) is a pro-apoptotic Bcl-2 family member, whose apoptotic functions can be inactivated through phosphorylation of specific residues…”
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    PO-001 Therapeutic inhibition of trefoil factor 3(TFF3) decrease oncogenic behaviour in non-small cell lung cancer (NSCLC) by Zhang, M, Chong, QY, Pandey, V, Chen, RM, Basappa, S, Goh, BC, Lobie, PE

    Published in ESMO open (01-07-2018)
    “…IntroductionIncreasing evidence has shown that trefoil factor 3 (TFF3) is increased in expression in various cancers and promotes cancer progression. Elevated…”
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