Search Results - "Linton, M.F"

Purine-rich foods, protein intake, and the prevalence of hyperuricemia: The Shanghai Men’s Health Study by Villegas, R, Xiang, Y.-B, Elasy, T, Xu, W.H, Cai, H, Cai, Q, Linton, M.F, Fazio, S, Zheng, W, Shu, X.-O

“…Abstract Background and aims Diet may play an important role in the development of hyperuricemia and gout. However, the association between dietary factors and…”
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Lifestyle determinants of c-reactive protein in middle-aged, urban Chinese men by Villegas, R, Xiang, Y.B, Cai, H, Elasy, T, Cai, Q, Zhang, X, Fazio, S, Linton, M.F, Li, H, Xu, W.H, Yang, G, Zheng, W, Shu, X.-O

“…Abstract Background and Aim: Increased levels of C-reactive protein (CRP), common in aging populations, are associated with higher risk for chronic diseases,…”
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Angiotensin receptor blocker vs ACE inhibitor effects on HDL functionality in patients on maintenance hemodialysis by Kaseda, R., Tsuchida, Y., Gamboa, J.L., Zhong, J., Zhang, L., Yang, H., Dikalova, A., Bian, A., Davies, S., Fogo, A.F., Linton, M.F., Brown, N.J., Ikizler, T.A., Kon, V.

“…Angiotensin receptor blockers (ARB) and angiotensin converting enzyme inhibitors (ACEI) reduce cardiovascular events in the general population. Maintenance…”
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Evaluation of macrophage-specific promoters using lentiviral delivery in mice by Levin, M C, Lidberg, U, Jirholt, P, Adiels, M, Wramstedt, A, Gustafsson, K, Greaves, D R, Li, S, Fazio, S, Linton, M F, Olofsson, S-O, Borén, J, Gjertsson, I

“…In gene therapy, tissue-specific promoters are useful tools to direct transgene expression and improve efficiency and safety. Macrophage-specific promoters…”
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Increased atherosclerosis in mice reconstituted with apolipoprotein E null macrophages by Fazio, S. (Vanderbilt University School of Medicine, Nashville, TN.), Babaev, V.R, Murray, A.B, Hasty, A.H, Carter, K.J, Gleaves, L.A, Atkinson, J.B, Linton, M.F

“…Macrophage-derived foam cells express apolipoprotein E (apoE) abundantly in atherosclerotic lesions. To examine the physiologic role of apoE secretion by the…”
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Expression of human apolipoprotein B90 in transgenic mice. Demonstration that apolipoprotein B90 lacks the structural requirements to form lipoprotein by McCormick, S P, Linton, M F, Hobbs, H H, Taylor, S, Curtiss, L K, Young, S G

“…Lipoprotein(a) (Lp(a)) is a lipoprotein formed by the disulfide linkage of apolipoprotein(a) (apo(a)) to the apoB100 of a low density lipoprotein particle…”
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