Search Results - "Lim, Hyoungsub"
-
1
Toll-like Receptor 2 Mediates Peripheral Nerve Injury-induced NADPH Oxidase 2 Expression in Spinal Cord Microglia
Published in The Journal of biological chemistry (15-03-2013)“…We have previously reported that NADPH oxidase 2 (Nox2) is up-regulated in spinal cord microglia after spinal nerve injury, demonstrating that it is critical…”
Get full text
Journal Article -
2
Toll-Like Receptor 2 Contributes to Chemokine Gene Expression and Macrophage Infiltration in the Dorsal Root Ganglia after Peripheral Nerve Injury
Published in Molecular pain (28-09-2011)“…Background: We have previously reported that nerve injury-induced neuropathic pain is attenuated in toll-like receptor 2 (TLR2) knock-out mice. In these mice,…”
Get full text
Journal Article -
3
Sexual dimorphism in cognitive disorders in a murine model of neuropathic pain
Published in Behavioral and brain functions (04-01-2020)“…A sex-difference in susceptibility to chronic pain is well-known. Although recent studies have begun to reveal the sex-dependent mechanisms of nerve…”
Get full text
Journal Article -
4
Toxoplasma gondii infection in the brain inhibits neuronal degeneration and learning and memory impairments in a murine model of Alzheimer's disease
Published in PloS one (21-03-2012)“…Immunosuppression is a characteristic feature of Toxoplasma gondii-infected murine hosts. The present study aimed to determine the effect of the…”
Get full text
Journal Article -
5
Polyamidoamine dendrimer-conjugated triamcinolone acetonide attenuates nerve injury-induced spinal cord microglia activation and mechanical allodynia
Published in Molecular pain (2017)“…Background Accumulating evidence on the causal role of spinal cord microglia activation in the development of neuropathic pain after peripheral nerve injury…”
Get full text
Journal Article -
6
IKK/NF-κB-dependent satellite glia activation induces spinal cord microglia activation and neuropathic pain after nerve injury
Published in Pain (Amsterdam) (01-09-2017)“…Increasing evidence indicates that both microglia and satellite glial cell (SGC) activation play causal roles in neuropathic pain development after peripheral…”
Get full text
Journal Article -
7
Inflammation‐Targeting Mesenchymal Stem Cells Combined with Photothermal Treatment Attenuate Severe Joint Inflammation (Adv. Mater. 11/2024)
Published in Advanced materials (Weinheim) (01-03-2024)“…Thermo‐Responsive Stem Cells In article number 2304333, Youn Joo Kang, Dongwoo Khang, and co‐workers demonstrate enhanced anti‐inflammatory therapeutic…”
Get full text
Journal Article -
8
Inflammation‐Targeting Mesenchymal Stem Cells Combined with Photothermal Treatment Attenuate Severe Joint Inflammation
Published in Advanced materials (Weinheim) (01-03-2024)“…Current clinical therapeutic efficacy for the treatment of osteo‐ and rheumatoid‐arthritis is obviously limited. Although mesenchymal stem cells (MSCs) are…”
Get full text
Journal Article -
9
GT1b functions as a novel endogenous agonist of toll‐like receptor 2 inducing neuropathic pain
Published in The EMBO journal (16-03-2020)“…Spinal cord microglia contribute to nerve injury‐induced neuropathic pain. We have previously demonstrated that toll‐like receptor 2 (TLR2) signaling is…”
Get full text
Journal Article -
10
GT 1b functions as a novel endogenous agonist of toll‐like receptor 2 inducing neuropathic pain
Published in The EMBO journal (16-03-2020)Get full text
Journal Article -
11
Identification and characterization of triamcinolone acetonide, a microglial-activation inhibitor
Published in Immunopharmacology and immunotoxicology (01-12-2012)“…Recent studies show that necrotic neuronal cells (NNC) activate microglia, thereby leading to neuronal cell death. This suggests that chemicals that inhibit…”
Get full text
Journal Article -
12
TLR4 enhances histamine-mediated pruritus by potentiating TRPV1 activity
Published in Molecular brain (21-08-2014)“…Recent studies have indicated that Toll-like receptor 4 (TLR4), a pathogen-recognition receptor that triggers inflammatory signals in innate immune cells, is…”
Get full text
Journal Article