Search Results - "Lian, Lubing"

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    A map of WW domain family interactions by Hu, Hai, Columbus, John, Zhang, Yi, Wu, Dongying, Lian, Lubing, Yang, Song, Goodwin, Jennifer, Luczak, Christine, Carter, Mark, Chen, Lin, James, Michael, Davis, Roger, Sudol, Marius, Rodwell, John, Herrero, Juan J.

    Published in Proteomics (Weinheim) (01-03-2004)
    “…WW domains are protein modules that bind proline‐rich ligands. WW domain‐ligand complexes are of importance as they have been implicated in several human…”
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    Journal Article
  3. 3

    Characterization of a carboxy-terminal BRCA1 interacting protein by WONG, A. K. C, ORMONDE, P. A, TAVTIGIAN, S. V, TENG, D. H.-F, BARTEL, P. L, PERO, R, YUAN CHEN, LUBING LIAN, SALADA, G, BERRY, S, LAWRENCE, Q, DAYANANTH, P, PHUONG HA

    Published in Oncogene (05-11-1998)
    “…There are several lines of evidence indicating that the carboxy-terminal region of the tumor suppressor protein BRCA1 is a functionally significant domain…”
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  4. 4

    cDNA cloning and gene mapping of human homologs for Schizosaccharomyces pombe rad17, rad1, and hus1 and cloning of homologs from mouse, caenorhabditis elegans, and Drosophila melanogaster by DEAN, F. B, LUBING LIAN, O'DONNEL, M

    Published in Genomics (San Diego, Calif.) (15-12-1998)
    “…Mutations in DNA repair/cell cycle checkpoint genes can lead to the development of cancer. The cloning of human homologs of yeast DNA repair/cell cycle…”
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    Journal Article
  5. 5

    Common Mechanism of Ligand Recognition by Group II/III WW Domains by Kato, Yusuke, Nagata, Koji, Takahashi, Mihoko, Lian, Lubing, Herrero, Juan J., Sudol, Marius, Tanokura, Masaru

    Published in The Journal of biological chemistry (01-07-2004)
    “…WW domain is a well known protein module that mediates protein to protein interactions by binding to proline-containing ligands. Based on the ligand…”
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    Journal Article
  6. 6

    Mutation Analyses of 268 Candidate Genes in Human Tumor Cell Lines by Teng, David H.-F., Chen, Yuan, Lian, Lubing, Ha, Phoung C., Tavtigian, Sean V., Wong, Alexander K.C.

    Published in Genomics (San Diego, Calif.) (15-06-2001)
    “…We have performed a homozygous deletion screen on 268 candidate genes in 90 human tumor cell lines derived from multiple types of cancers. Most of the…”
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  7. 7

    cDNA Cloning and Gene Mapping of Human Homologs forSchizosaccharomyces pombe rad17, rad1,andhus1and Cloning of Homologs from Mouse,Caenorhabditis elegans,andDrosophila melanogaster by Dean, Frank B., Lian, Lubing, O'Donnell, Mike

    Published in Genomics (San Diego, Calif.) (01-12-1998)
    “…Mutations in DNA repair/cell cycle checkpoint genes can lead to the development of cancer. The cloning of human homologs of yeast DNA repair/cell cycle…”
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    Journal Article
  8. 8

    Genomic Structure, Chromosomal Location, and Mutation Analysis of the Human CDC14A Gene by Wong, Alexander K.C., Chen, Yuan, Lian, Lubing, Ha, Phuong C., Petersen, Kelly, Laity, Kirsten, Carillo, Arlene, Emerson, Marion, Heichman, Karen, Gupte, Jamila, Tavtigian, Sean V., Teng, David H.-F.

    Published in Genomics (San Diego, Calif.) (15-07-1999)
    “…Human CDC14A is a dual-specificity phosphatase that shares sequence similarity with the recently identified tumor suppressor, MMAC1/PTEN/TEP1. By radiation…”
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    Journal Article
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    Common mechanism of ligand recognition by group II/III WW domains: redefining their functional classification by Kato, Yusuke, Nagata, Koji, Takahashi, Mihoko, Lian, Lubing, Herrero, Juan J, Sudol, Marius, Tanokura, Masaru

    Published in The Journal of biological chemistry (23-07-2004)
    “…WW domain is a well known protein module that mediates protein to protein interactions by binding to proline-containing ligands. Based on the ligand…”
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    Journal Article
  10. 10

    Quantitative Trait Locus Analysis for Chronic Pancreatitis and Diabetes Mellitus in the WBN/Kob Rat by Tsuji, Atsushi, Nishikawa, Tetsu, Mori, Masayuki, Suda, Koichi, Nishimori, Isao, Nishimura, Masahiko

    Published in Genomics (San Diego, Calif.) (15-06-2001)
    “…A causative gene mutation is still undefined in approximately half of patients with hereditary pancreatitis, and no genetic factor has been identified in most…”
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    Journal Article