Search Results - "Lentink, Viola"

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    Regulation of Cardiac Gene Expression by KLF15, a Repressor of Myocardin Activity by Leenders, Joost J., Wijnen, Wino J., Hiller, Monika, van der Made, Ingeborg, Lentink, Viola, van Leeuwen, Rick E.W., Herias, Veronica, Pokharel, Saraswati, Heymans, Stephane, de Windt, Leon J., Høydal, Morten A., Pinto, Yigal M., Creemers, Esther E.

    Published in The Journal of biological chemistry (27-08-2010)
    “…Pathological forms of left ventricular hypertrophy (LVH) often progress to heart failure. Specific transcription factors have been identified that activate the…”
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    Journal Article
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    Dominant-Negative Control of cAMP-Dependent I Ks Upregulation in Human Long-QT Syndrome Type 1 by Heijman, Jordi, Spätjens, Roel L.H.M.G., Seyen, Sandrine R.M., Lentink, Viola, Kuijpers, Helma J.H., Boulet, Inge R., de Windt, Leon J., David, Miren, Volders, Paul G.A.

    Published in Circulation research (20-01-2012)
    “…Rationale: The mutation A341V in the S6 transmembrane segment of KCNQ1 , the α-subunit of the slowly activating delayed-rectifier K + (I Ks ) channel,…”
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    Journal Article
  5. 5

    Dominant-Negative Control of cAMP-Dependent IKs Upregulation in Human Long-QT Syndrome Type 1 by Heijman, Jordi, Spätjens, Roel L.H.M.G, Seyen, Sandrine R.M, Lentink, Viola, Kuijpers, Helma J.H, Boulet, Inge R, de Windt, Leon J, David, Miren, Volders, Paul G.A

    Published in Circulation research (20-01-2012)
    “…RATIONALE:The mutation A341V in the S6 transmembrane segment of KCNQ1, the α-subunit of the slowly activating delayed-rectifier K (IKs) channel, predisposes to…”
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    Journal Article
  6. 6

    Long-QT mutation p.K557E-Kv7.1: dominant-negative suppression of IKs, but preserved cAMP-dependent up-regulation by Spätjens, Roel L H M G, Bébarová, Markéta, Seyen, Sandrine R M, Lentink, Viola, Jongbloed, Roselie J, Arens, Yvonne H J M, Heijman, Jordi, Volders, Paul G A

    Published in Cardiovascular research (01-10-2014)
    “…Mutations in KCNQ1, encoding for Kv7.1, the α-subunit of the IKs channel, cause long-QT syndrome type 1, potentially predisposing patients to ventricular…”
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    Journal Article